No description available.

No description available.

No description available.

No description available.

Abstract available in Audiofonologia.

A chart review of Veterans with (1) Tinnitus Only, (2) Tinnitus + Anxiety/Depression, and (3) Tinnitus + post-traumatic stress disorder was conducted to determine the prevalence of comorbid health conditions. Patients with comorbid mental health conditions also were likely to suffer from additional comorbid health conditions (e.g., insomnia, etc).

No description available.

It has been suggested that the presence of a limited area of damaged outer hair cells (OHCs) with intact inner hair cells, which may not be detected on the conventional audiogram, results in unbalanced neural activity between Type I and Type II fibers leading to tinnitus. In normal-hearing tinnitus patients, DPOAEs provided ambiguous data of OHC function when compared to non-tinnitus controls. Hearing loss in the extended high-frequency (EHF) region may decrease DPOAEs evoked at lower frequencies. Results of EHF audiometry in tinnitus patients are limited. The aim of the study was to evaluate DPOAEs and EHF thresholds in normal-hearing patients reporting unilateral tinnitus in left ear. Thus, each subject acted as their own control. Data were obtained for 26 subjects with bilateral hearing thresholds10, 12.5, 14, and 16 kHz. The DP-grams were measured in the 0.5-8 kHz range using 65/55-dB SPL primaries and f2/f1=1.2. The data analyses included DPOAEs with S/N>3 dB. Median audiometric data showed that thresholds in the left ears were significantly higher than those in the right ears at all four EHFs. Mean DPOAE levels of the left ears were lower than those of the right ears in the frequency range above 1 kHz. Additionally, a paired-comparison test of DPOAE levels of each patient’s right and left ear revealed significant differences at 6 and 8 kHz. The results indicate that: 1. OHC impairment in the most basal region reduces contribution to more apically generated DPOAEs; 2. OHC impairment in a limited area, which may be revealed by DPOAEs but not by conventional audiometry, can contribute to tinnitus generation; and 3. patients with unilateral tinnitus and normal hearing on the conventional audiogram are likely to demonstrate hearing loss in the EHF region.

No description available.

Tinnitus is the perception of sound in the absence of an actual sound stimulus. Recent developments have shifted the focus to the central nervous system and the neural correlate of tinnitus. Broadly, tinnitus involves cortical map rearrangement, pathological neural synchrony, and increased spontaneous firing rates. Various cortical regions, such as Heschl’s gyrus in the auditory cortex, have been found to be associated with different aspects of tinnitus, such as perception and loudness. I propose a cortical stimulation mapping study of Heschl’s gyrus using a depth and subdural electrode montage to conduct electrocorticography. This study would provide high-resolution data on abnormal frequency band oscillations characteristic of tinnitus and pinpoint regions where they occur. The validity of the neural synchrony model would also be tested in this study.