published_or_final_version / Community Medicine / Master / Master of Public Health

Diss. (sammanfattning) Stockholm : Karol. inst. / Härtill 5 uppsatser.

Thesis (M.F.A.)--Rochester Institute of Technology, 19xx. / Typescript. Includes bibliographical references.

Thesis (M.S.)--University of Wisconsin--Madison. / Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaf 10).

Thesis (M.P.H.)--University of Hong Kong, 2008. / Includes bibliographical references (p. 51-53).

Thesis--Dorpat. / Curriculum vitae. "Literatura": p. [178]-193.

Afhandling - Copenhagen. / "Translated from the Danish by Einar Christensen. Case histories translated by Elisabeth Aagesen." Summaries in English and Danish. Bibliography: p. [345]-358.

Chronic hypertension was induced in female albino rats. Amelioration by means of daily feeding of histidine and ascorbic acid, histidine and vitamin E, histidine and urease, vitamin E, urease and ammonium chloride was attempted. Blood pressures were determined under sodium pentathol anesthetic by the indirect method using the foot. Chronic hypertension was produced by two methods, (1) Injections of DCA. This method was found to give inconsistent results. Several modifications of this procedure were tried unsuccessfully. (2) The use of a choline deficient diet. This method proved to give consistent results in producing chronic hypertension. Vitamin E was the only substance found useful in ameliorating the experimental chronic hypertension. This amelioration resulted only in the presence of excess vitamin E. This fact was thought to give further evidence for the vasodilator properties of vitamin E. An hypothesis was advanced that DCA may inhibit the sympathetic nervous system, to sensitize the vascular responsiveness to pressor and depressor substances. / Science, Faculty of / Botany, Department of / Zoology, Department of / Graduate

The effects of dietary and hormonal factors upon the blood pressure of male Wistar rats were investigated. 1. Desoxycorticosterone acetate was found to increase the blood pressure in the presence of excess dietary salt. 2. Estrogen overdosage produced hypertension in rats. Castrate animals were more sensitive to the effects of estrogen. Estrogen overdosage aggravated experimental hypertension induced by nutritional choline deficiency. 3. A period of nutritional choline deficiency was followed by hypertension. Desoxycorticosterone accelerated the development of hypertension in choline deficient animals. 4. Hypertension developed in rats 2 months after a prolonged period of inanition. 5. Antihistamine was found to induce profound variations in the blood pressure of male rats. 6. Rats pre-treated with desoxycorticosterone acetate showed a greater pressor response to adrenaline than normal intact rats. 7. Desoxycorticosterone and antihistamine were found to facilitate the occurrence of the Trueta shunt. / Science, Faculty of / Botany, Department of / Zoology, Department of / Graduate

Thesis (M.S.)--University of Michigan, 1964.