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Overexpression, purification, and characterization of mmgB and mmgC from Bacillus subtilis strain 168Russell, Spencer A. January 1900 (has links)
Thesis (M.S.)--The University of North Carolina at Greensboro, 2008. / Directed by Jason Reddick; submitted to the Dept. of Chemistry and Biochemistry. Title from PDF t.p. (viewed Aug. 27, 2009). Includes bibliographical references (p. 53-57).
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Isolation and characterization of Bacilus subtilis mutants resistant to ethidium bromide and aflatoxin B₁Bishop, Paul Edward 08 August 1973 (has links)
Graduation date: 1974
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Studies on Bacillus subtilis extracellular enzymesMay, Brian Kenneth January 1970 (has links)
v, 215 leaves : ill., xiii / Title page, contents and abstract only. The complete thesis in print form is available from the University Library. / Thesis (Ph.D.)--University of Adelaide, Dept. of Biochemistry, 1971
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Studies on Bacillus subtilis extracellular enzymes.May, Brian Kenneth. January 1970 (has links) (PDF)
Thesis (Ph.D.) -- University of Adelaide, Dept. of Biochemistry, 1971.
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Isolation and partial characterization of an oligo (dT)-bound RNA fraction of Bacillus subtillisGraef, Ellen. January 1978 (has links)
Thesis (M.S.)--Wisconsin. / Includes bibliographical references (leaves 56-60).
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Ribosomal RNA genes of Bacillus subtilisLoughney, Kate. January 1983 (has links)
Thesis (Ph. D.)--University of Wisconsin--Madison, 1983. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.
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The effect of penicillin on cell wall synthesis in Bacillus subtilisRoberts, Joseph. January 1962 (has links)
Thesis (M.S.)--University of Wisconsin--Madison, 1962. / Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 46-47)
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A streptomycin-resistant oligosporogenous mutant of Bacillus subtilisCampbell, Kristine M. January 1979 (has links)
Thesis--University of Wisconsin--Madison. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.
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The transcriptional control of spx in response to oxidative stress /Leelakriangsak, Montira. January 2007 (has links)
Thesis (Ph.D.) OGI School of Science & Engineering at OHSU, October 2007. / Includes bibliographical references (leaves 130-153).
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Consequences of bacillus subtilis in iron deficiencyPeters, Walter Joseph January 1968 (has links)
Cultures of Bacillus subtilis growing in an iron-deficient medium produced coproporphyrin III (coproporphyrin) and phenolic acids. (2,3-dihydroxybenzoylglycine (DHBG), 2,3-dihydroxybenzoic acid (DHB), or both). (DHB(G) refers to DHB or DHBG, or both compounds). Phenolic acid production was proportional to the amount of iron present, and occurred logarithmically, parallelling growth. In the presence of DHB, lower levels of iron inhibited phenolic acid production, so that the actual inhibition of synthesis may involve the Fe(+3): (DHB(G))₃ complex. Accumulatlon of DHB(G) was influenced by the levels of aromatic amino acids, anthranilic acid, and histidine in the medium. In vitro experiments demonstrated that DHB was formed from chorismic acid. In vivo and in vitro experiments with strain B-1471 showed that DHB was coupled to added glycine to form DHBG. Disappearance of DHB(G) was observed in all strains studied, but oxidation did not occur.
Phenolic acid production always preceded coproporphyrin production. Phenolic acids have very strong affinities for ferric iron. Their production may therefore allow the scavenging of the last traces of iron from the medium for hemin synthesis. The relationship between phenolic acid and coproporphyrin production was borne out by the following observations: (i) a higher level of iron was required to prevent coproporphyrin production than phenolic acid production (ii) the Fe(+3) (DHB(G))₃ complex was a more potent inhibitor of coproporphyrin production than iron alone (iii) a mutant blocked at σ-aminolevulinic acid synthetase did not produce phenolic acids during iron-deficient growth (iv) serine auxotrophs produced much lower levels of coproporphyrin and phenolic acids than the wild-type strain (v) some mutants defective in phenolic acid production produced low levels of coproporphyrin, whereas one strain of this type produced elevated levels of coproporphyrin.
Compounds known to inhibit normal functioning of the tricarboxylic acid cycle decreased coproporphyrin production in all strains studied. These inhibitors reduced DHBG excretion, but had no effect on DHB production. A number of analogs of DHB inhibited DHB(G) accumulation to varying degrees, depending upon their structure. The most potent inhibitors were m-substituted derivatives of benzoic acid. Two sideramines, ferrichrome and ferrioxamine, inhibited DHBG production in strain B-1471. The inhibitory action of ferrichrome was shown to be due to its ability to mediate cellular uptake of low levels of iron.
The capacity of B. subtilis for iron uptake was increased about 20-fold by growing the cells in an iron-deficient medium. Under these conditions, the addition of low levels of phenolic acids increased both the rate and extent of iron uptake. Mutants unable to synthesize normal levels of phenolic acid were shown to have a reduced capacity for iron uptake after growth in an iron-deficient medium. Mutants resistant to 8-hydroxyquinoline had an increased capacity for iron-uptake under these conditions. / Science, Faculty of / Microbiology and Immunology, Department of / Graduate
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