Spelling suggestions: "subject:"river disease"" "subject:"river adisease""
1 |
The effects of homologous cross circulation and in situ liver perfusion on fulminant hepatic failure rats /Mohsini, K. G. (Khawar G.) January 1982 (has links)
No description available.
|
2 |
Exploration of protective pathways in liver diseaseWahid, Talha 11 December 2021 (has links)
Obesity is increasing worldwide. The addition of excess calorie intake and unhealthy human behavior leads to also other diseases such as metabolic syndromes and liver disease such as non-alcoholic fatty liver disease. Furthermore, the accumulation of fat triggers specific mechanisms that, if prolonged, can cause tissue damage. For instance, the innate immune system becomes agitated in patients with NAFLD or obesity due to excessive fat accumulation. This leads to inflammation in specific tissues and infiltration of other immune cells. One of the main immune cells are neutrophils, which secrete a protease enzyme called protease neutrophil elastase. Interestingly, there have been studies conducted that have shown that when neutrophil elastase is knocked out in mouse models that mimic NAFLD, there seems to be a protective effect occurring in the body and lessen tissue scarring. A possible explanation, and the aim of this thesis, is to explore if autophagy is regulated and thus plays a role in protecting liver from inflammation and fibrosis. Western blotting approach was used to test this hypothesis. The protein samples that are used are extracted from neutrophil elastase knockout mice that have been fed a high-fat high-fructose diet and compare them to samples from wild-type control mice that have been fed a normal chow diet, high-fat diet, and high fat high fructose diet. The results indicated that potential upregulation of the autophagy pathway in the liver of neutrophil elastase knockout mice and more studies would need before accurately and reliably acknowledging the alternation of the autophagy pathway in the liver from mouse model of NAFLD and when neutrophil elastase is knocked out. / 2023-12-10T00:00:00Z
|
3 |
Disordered L+ lactate metabolism in manConnor, Henry January 1979 (has links)
No description available.
|
4 |
Regulation of secretion of #alpha#-2 macroglobulin by hepatic lipocytes in relation to hepatic fibrosisKawser, Choudhury Ali January 1993 (has links)
No description available.
|
5 |
Humoral responses to acetaldehyde-modified protein antigensRamsay, Lesley Anne January 1998 (has links)
No description available.
|
6 |
Lymphocyte subsets and immune mechanisms in primary sclerosing cholangitis (PSC)Martins, Eduardo B. G. January 1995 (has links)
No description available.
|
7 |
T-cell responses to the 2-oxo acid dehydrogenases in primary biliary cirrhosisJones, David E. J. January 1997 (has links)
No description available.
|
8 |
The genetic epidemiology of primary biliary cirrhosisGregory, Wendy L. January 1993 (has links)
No description available.
|
9 |
Glucose metabolism and insulin resistance in cirrhosisShmueli, Ehoud January 1992 (has links)
No description available.
|
10 |
Hepatitis C virus : studies of the molecular basis of virus/host interactionWatson, J. P. January 1996 (has links)
No description available.
|
Page generated in 0.0581 seconds