Spelling suggestions: "subject:"myocardial infarction."" "subject:"nyocardial infarction.""
1 |
Psychophysiological recovery after acute myocardial infarction /Buchanan, Lynne M. January 1989 (has links)
Thesis (Ph. D.)--University of Washington, 1989. / Vita. Includes bibliographical references (leaves [94]-105.).
|
2 |
Biochemical and clinical characterization of urban black Africans with myocardial infarction.Reavis, Stephen C January 2015 (has links)
It is widely accepted that the occurrence of myocardial
infarction (MI) among Black South Africans is much lower
than that of South Africans of European or Asian
ancestry. Over the past decade, this disease among
Black Africans in urban areas appears tc be increasing.
The prevalence of MI among Black Africans admitted fo
Baragwanath Hospital (BGH) over a four year period
(1984-1987) was determined by computer identification of
all patients demonstrating elevated creatine kinase
isoenzymes. Diagnosis of MI was confirmed by reviewing
the hospital bedletters.
|
3 |
Myocardial survival and function under conditions of anoxiaWeil, Daniel B. January 1959 (has links)
Thesis (M.A.)--Boston University / Rat heart muscle was subjected to quantitated conditions of anoxia achieved by extracting oxygen from Ringer's solution under vacuum and heat. Special Van Slyke analyses measured the amount of oxygen remaining in solution after the extraction procedures had been carried out.
Myocardial response to anoxia was studied in terms of survival, or the ability of the heart tissue to remain alive during anoxia with resumption of function when placed in an oxygenated environment, and in terms of function, or the ability of heart muscle to contract during anoxia [TRUNCATED]
|
4 |
Expert opinions on best practice for the occupational therapy management of visual perceptual deficits in adults post strokeReed, Bronwyn Joy January 2013 (has links)
A research report submitted to the Faculty of Health Sciences, University of the Witwatersrand, Johannesburg, in partial fulfilment of the requirements for the degree of Master of Science in Occupational Therapy
Johannesburg / Visual perceptual impairment is a common consequence of stroke which may affect all areas of occupational performance. The evidence for the management of these deficits in occupational therapy is not clear. The purpose of this qualitative study was therefore to determine the expert opinion for best practice in the management of visual perceptual deficits in adults post stroke in the South African context.
Interviews with eight expert occupational therapists indicated that a lack of clarity exists in this area of clinical practice. Findings indicate this is a complex area of practice where management needs to be focussed on occupation based therapy for the individual patient in their context. The use of standardised and non-standardised assessments and compensation verses remediation in intervention must be individually determined, and the use of a “top down” approach was considered preferable. This study presents the first step in the development of clinical guidelines for management of this deficit in occupational therapy in South Africa.
|
5 |
Functional proteomic analysis of the myocardial PKC[epsilon] subproteome and PKC[epsilon]-AKT-eNOS signaling modules during cardioprotectionZhang, Jun. January 2003 (has links) (PDF)
Thesis (Ph. D.)--University of Louisville, 2003. / Department of Physiology and Biophysics. Vita. "December 2003." Includes bibliographical references (leaves 102-115).
|
6 |
Acute myocardial infarction with a non-diagnostic electrocardiogram : case presentation and overviewPrzybojewski, J. Z., Gilburt, S. G. M. 12 1900 (has links)
The original publication is available at http://www.samj.org.za / The clinical presentation of a young hypertensive White man with acute high lateral non-transmural myocardial infarction (MI) is documented. This diagnosis was established on the grounds of a history of chest pain, elevated serial serum enzyme levels, technetium-99m pyrophosphate ('hot-spot') scintigraphy, exercise thallium-201 ('cold-spot') scanning, left ventricular cine angiography and selective coronary arteriography. Daily resting 12-lead ECGs failed to demonstrate unequivocal features of acute non-transmural subendocardial MI. The diagnostic difficulties facing the clinician in a case of acute MI associated with a non-diagnostic ECG are stressed, and the ECG features of acute subendocardial MI are reviewed. / Publishers' version
|
7 |
Potential Role for the Sarcolemmal Membrane Associated Protein Isoform 3 (SLMAP3) in Cardiac Remodeling Post Myocardial InfarctionLefnaier, Wafa January 2017 (has links)
ABSTRACT
The Sarcolemmal Membrane Associated Protein 3 (SLMAP3) is a tail-anchored membrane protein, which is ubiquitously expressed in tissues including myocardium. It is a component of subcellular membranes and the centrosome, and it appears to serve distinct roles in cell growth and membrane biology. In addition, mutations in SLMAP have been linked to Brugada syndrome, which leads to cardiac dysfunction and death. Here, we have examined the effects of different levels of SLMAP3 on postnatal heart function, pre and post myocardial infarction (MI).Transgenic (TG) mice with a cardiac specific expression of SLMAP3 isoform were generated and assessed with echocardiography to measure function, immunohistochemistry for histology, TUNEL assay for apoptosis, Masson’s trichrome staining for fibrosis, and Western blots for protein expression.
Baseline echocardiography of 8 weeks old TG mice showed a normal cardiac function that was expressed in ejection fraction percent (%EF=66%±7.42), which was similar to those of wild type mice (%EF=67%±9.36), p<0.05, n=20-25 (in each group). MI was induced by permanent ligation of left anterior descending (LAD) artery in 9 week old WT & TG mice, while sham was the control. No death was recorded in SLMAP3 TG mice up to one year post MI, whereas 70% of WT mice had deceased, p<0.01, n=17-18 (in each group). Cardiac function was assessed by echocardiography (at 4 week post MI) showed a partially restored ejection fraction percent (%EF~49.2%±17.02) in SLMAP3 TG mice post MI compared to (%EF~36.4%±15.25) in WT mice post MI, p<0.05, n=15-16 (in each group). Furthermore, infarct size (IS) as well as collagen area (CA) post MI were significantly attenuated (IS~43%±8.82, CA~35%±5.15) in SLMAP3 TG myocardium in comparison to WT (IS~53%±9.30, CA~47%±7.36), p<0.05, n=20- 22 (each group). Moreover, expression of the heart failure biomarker galectin3 was markedly
ii
attenuated (1.8±0.20) in SLMAP3 TG hearts post MI compared to (3.2±0.35) in WT, p<0.01, n=4-5 (in each group). The apoptotic index in SLMAP3 TG myocardium assessed by TUNEL was markedly decreased (77±11.48) in comparison to WT (112±15.32), p<0.05, (n=20-22 in each group). Further, expression of proapoptotic proteins (Caspase3 and Bax) was significantly attenuated in SLMAP3 TG (p<0.05, n=4-5 in each group) while the expression of the prosurvival proteins (Bcl2 and caveolin3) was significantly upregulated (p<0.05, n=4-5 (in each group) in post MI. These data indicate that increased SLMAP3 levels serve to protect myocardium post MI through mechanisms which promote cell survival and limit cardiac fibrosis. Strategies to increase SLMAP3 level in myocardium may provide new therapeutic options in the treatment of heart failure.
|
8 |
The Epidemiology of Acute Myocardial Infarction and the Role of ThrombolysisBrophy, James January 1998 (has links)
No description available.
|
9 |
Egpare se belewing van hulle huweliksverhouding voor en na 'n miokardiale infarksieGoosen, Helletje. January 2001 (has links)
Thesis (M.A.(Sielk)--University of Pretoria, 2001. / Includes bibliographical references (p. 279-306).
|
10 |
Insulin resistance, genetic variation and cytokines : associations to myocardial infarction risk /Bennet, Anna, January 2003 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2003. / Härtill 4 uppsatser.
|
Page generated in 0.1074 seconds