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The Global ETD Search service is a free service for researchers
to find electronic theses and dissertations. This service is
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Networked Digital Library of Theses and Dissertations.
Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
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Showing 11 to 20 of 26 (0.054 seconds)Spelling suggestions: "subject:"papillomavirus, human"" "subject:"papillomavirus, suman""
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Regulation of RNA processing in Human Papillomavirus Type 16 /Rush, Margaret, January 2005 (has links)
Diss. (sammanfattning) Uppsala : Uppsala universitet, 2005. / Härtill 3 uppsatser.
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| 12 |
Human papillomavirus in tonsillar cancer /Mellin, Hanna, January 2002 (has links)
Diss. (sammanfattning) Stockholm : Karolinska institutet, 2002. / Härtill 4 uppsatser.
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| 13 |
Investigation of the mechanism by which the human papillomavirus type-16 E6 oncoprotein induces telomerase in epithelial cells /Gewin, Lindy Carol, January 2004 (has links)
Thesis (Ph. D.)--University of Washington, 2004. / Vita. Includes bibliographical references (leaves 77-92).
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| 14 |
Longitudinal studies of human papillomavirus infection : with special reference to screening for cervical cancer and treatment of CIN /Elfgren, Kristina, January 1900 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2003. / Härtill 5 uppsatser.
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| 15 |
Cervical cancer : associations with HPV infection, barrier contraceptive use, and Pap smear screening /Mandelson, Margaret T. January 1994 (has links)
Thesis (Ph. D.)--University of Washington, 1994. / Vita. Includes bibliographical references (leaves [57]-62).
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| 16 |
Genomic variation of human papillomavirus type 16 in relation to risk for high grade cervical and anal intraepithelial neoplasia /Xi, Long Fu. January 1997 (has links)
Thesis (Ph. D.)--University of Washington, 1997. / Vita. Includes bibliographical references (leaves [79]-87).
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| 17 |
Multiple biological activities of the human papillomavirus type 16 E7 oncoprotein contribute to the abrogation of human epithelial cell cycle control /Helt, Anna-Marija. January 2002 (has links)
Thesis (Ph. D.)--University of Washington, 2002. / Vita. Includes bibliographical references (leaves 111-140).
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| 18 |
Three case-control studies : vulvar cancer, invasive cervical cancer, and in situ adenocarcinoma of the cervix /Madeleine, Margaret M. January 1996 (has links)
Thesis (Ph. D.)--University of Washington, 1996. / Vita. Includes bibliographical references (leaves [66]-84).
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| 19 |
Studies on the presence and influence of human papillomavirus (HPV) in head and neck tumors /Dahlgren, Liselotte, January 2005 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2005. / Härtill 4 uppsatser.
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| 20 |
Estudo in vitro sobre a interação celular e vias endocíticas de papilomavírus humano (HPV) em leucócitos do sangue periférico. / In vitro study on the interaction of human papillomavirus in cell from peripheral blood leukocytes.Szulczewski, Vívian 05 May 2009 (has links)
O papilomavírus humano (HPV) é o principal agente etiológico do câncer cervical e anogenital, sendo o HPV16 e o HPV18 os vírus de alto risco. Estudos recentes evidenciaram que além da transmissão sexual do HPV, há outras formas de contágio. Entretanto, a dificuldade na obtenção de quantidades viáveis do tipo selvagem ou mutante do HPV tem limitado em muito os estudos de diversos aspectos da biologia do papilomavírus. Este estudo investigou a possibilidade de o HPV infectar células leucocitárias do sangue periférico humano. Concluímos que as VLPs L1L2 do HPV16 podem utilizar a via endocítica do ferro mediada por clatrina, através do complexo VLPs-Transferrina-Receptor de Transferrina, permanecendo de forma latente em leucócitos. Esta porta de entrada oportunista poderia explicar a propagação crescente e alarmante deste agravo à saúde humana, motivo de preocupação nos sistemas mundiais de saúde pública. Este trabalho demonstrou pela primeira vez a internalização de VLPs L1L2 do HPV16 em leucócitos do sangue periférico humano. / Human papillomavirus (HPV) is the primary etiologic agent of anogenital and cervical cancer, caused mainly by the high-risk HPV16 and HPV18 viruses. Recent studies revealed that besides the sexual transmission of HPV, there are other forms of contagion. However, the difficulty in obtaining quantities of viable wild-type or mutant of HPV constitutes a limiting factor in the studies of various aspects of the biology of human papillomavirus. This study investigated the possibility of HPV infect the cells of human peripheral blood leukocytes. We conclude that the VLPs L1L2 of HPV16 may use the iron endocytic pathway clathrin-mediated through the complex VLPs-Transferrin-Transferrin Receptor and remained so latent in leukocytes. This port of entry opportunist could explain the growing and alarming spread of this disease to human health, cause for concern in the global public health. This study showed for the first time the internalization of VLPs L1L2 of HPV16 in human peripheral blood leukocytes.
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