No description available.

Thesis--University of Wisconsin--Madison. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 255-265).

Objective: To investigate the adverse effect of welding exposure on pulmonary function test and respiratory symptoms in welders. Design: Cross-sectional study Setting: Guangzhou, China Subjects: 350 male welders in an automobile assembly factory and 350 controls in MTR corporation who had no exposure to welding fumes and toxic gases. Methods: The data was collected from Guangzhou No.12 People’s Hospital. This inclusion and exclusion criteria for subjects and controls were set. The relationship between pulmonary function, respiratory symptoms and welding exposure was investigated by comparing the prevalence of abnormal pulmonary function and respiratory symptoms among welders and non-welders. Chi-square was used for comparison of categorical variables. Logistic regression was performed to estimate odds ratios after adjustment for age, working duration and smoking habits. Results: After accounting for age, working years and smoking habit, the adjusted OR (95% C.I.) of pulmonary function among welders and non-welders was 1.425 (0.916, 2.215), suggesting that welders was associated with a 1.43 times as high the odds of being tested abnormal pulmonary function as non-welders. However, there was no significant (P=0.116) difference in odds between welders and non-welders. The differences of respiratory symptoms in odds between welders and non-welders was not significant (P>0.268). But when restricted to smokers, significant difference (p<0.05) in abnormal pulmonary function was found between welders (17.5%) and non-welders (10.6%). Of the respiratory symptoms, only prevalence of cough was found to be statistically higher (p<0.05) among welders (16.1%) who smoked than non-welders (8.0%) who smoked. Conclusion: It is not evident from my study thatwelders who worked in a well-ventilation working environment and well protected by wearing personal protective equipment are subjected to increased prevalence of abnormal pulmonary function and respiratory symptoms. Studies with larger samples and longer period of observation are warranted. Smoking was observed to be the most important factor contributing to the presence of abnormal pulmonary function and respiratory symptoms. Smoking cessation would be needed to carry out in welders. / published_or_final_version / Public Health / Master / Master of Public Health

Thesis (Ph.D.)--University of Western Australia, 2006.

Thesis (Ph. D.)--West Virginia University, 2009. / Title from document title page. Document formatted into pages; contains ix, 66 p. : ill. (some col.). Includes abstract. Includes bibliographical references (p. 58-61).

No description available.

No description available.

No description available.

A series of five studies were conducted to examine the effect of exercise on selected pulmonary function measurements. Studies I and II determined the effect of an acute bout of exercise on various lung volumes immediately post-exercise and over a 24-hour post-exercise period. There were significant mean increases of 210 ml (20.6%) and 260 ml (20.8%) in the 5-minute post-exercise residual volume (RV) measurement for studies I and II, respectively. There also were significant mean increases of 170 ml (3.4%) and 190 ml (2.7%) in the 5-minute post-exercise total lung capacity (TLC) for studies I and II, respectively, while vital capacity (VC) remained unchanged. RV and TLC remained significantly increased over the pre-exercise values through 30 and 15 minutes of recovery, respectively. Studies III through V were undertaken to determine the physiological mechanism underlying the responses reported in studies I and II. In study III, transthoracic electrical impedance (TEI) was significantly decreased below the pre-exercise value through 30-minutes of recovery, indicating that there was an increase in thoracic fluid volume following exercise. However, TEI measurements alone cannot separate between intra- and extravascular fluid shifts. Therefore, studies IV and V attempted to identify whether the decrease in TEI and increase in RV reported in study III were due to intra- or extravascular fluid shifts. Study IV examined the TEI, RV, and TLC responses before and following exercise, as central blood volume (CBV) was experimentally increased via G-suit inflation, and decreased via venous occlusion tourniquets. The results suggest that RV is relatively insensitive to intravascular volume shifts within the thorax. Study V determined and followed the effect of an acute bout of exercise on lung diffusion capacity (D(,Lco)). D(,Lco)/V(,A) did not increase significantly following exercise, suggesting that the decrease in TEI following exercise is the result of extravascular fluid accumulation. It was concluded that a sub-clinical pulmonary edema occurs following exercise. A logical sequence of events based on the results of studies I through V was proposed as a possible explanation for the responses of RV and TLC following exercise.

Thesis--Wisconsin. / Vita. Includes bibliographical references.