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Cellular dysfunction associated with feline leukemia virus-infections in cats /Lafrado, Louis J. January 1986 (has links)
No description available.
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Pathogenesis of feline leukemia virus-induced erythroid aplasia : hematologic, immunologic, and therapeutic studies /Zack, Philip M. January 1987 (has links)
No description available.
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Determination of feline interleukin 2 characteristics in specific-pathogen-free and feline leukemia virus-infected cats and the effects of 1,1-dimethylhydrazine on interleukin 1 and 2 activities in the murine system /Bauer, Richard M. January 1987 (has links)
No description available.
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Inhibition of lymphocyte functions by inactivated feline leukemia virus and feline leukemia virus proteins : a putative model for immunosuppression /Mathes, Lawrence Eaton January 1977 (has links)
No description available.
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Understanding Receptor Adaptation And Co-receptor Use For Feline Leukemia VirusesHussain, Naveen 10 August 2009 (has links)
Feline leukemia viruses (FeLVs) are pathogenic retroviruses of the domestic cat. FeLV transmission and emergence of pathogenic variants show striking similarity to HIV pathogenesis. The emergence of pathogenic subgroup-C FeLV from the transmitted subgroup-A FeLV coincides with a switch in host receptor used for infection as a result of mutations in the viral envelope protein (Env). I have characterized a novel FeLV Env that may represent an evolutionary intermediate between FeLV-A and FeLV-C.
I have also reported evidence suggesting that FeLVs may use co-factors/co-receptors for infection. I have found that FeLVs inefficiently infect murine NIH3T3 cells overexpressing FeLV receptors (NIH3T3/Receptor). I have provided evidence that the low infection is caused by a block at a post-binding but pre-entry stage of FeLV infection. Furthermore, fusion of NIH3T3/Receptor cells with highly susceptible cells rescues inhibition to infection suggesting that FeLVs, like HIV, may also use co-receptors for infection.
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Understanding Receptor Adaptation And Co-receptor Use For Feline Leukemia VirusesHussain, Naveen 10 August 2009 (has links)
Feline leukemia viruses (FeLVs) are pathogenic retroviruses of the domestic cat. FeLV transmission and emergence of pathogenic variants show striking similarity to HIV pathogenesis. The emergence of pathogenic subgroup-C FeLV from the transmitted subgroup-A FeLV coincides with a switch in host receptor used for infection as a result of mutations in the viral envelope protein (Env). I have characterized a novel FeLV Env that may represent an evolutionary intermediate between FeLV-A and FeLV-C.
I have also reported evidence suggesting that FeLVs may use co-factors/co-receptors for infection. I have found that FeLVs inefficiently infect murine NIH3T3 cells overexpressing FeLV receptors (NIH3T3/Receptor). I have provided evidence that the low infection is caused by a block at a post-binding but pre-entry stage of FeLV infection. Furthermore, fusion of NIH3T3/Receptor cells with highly susceptible cells rescues inhibition to infection suggesting that FeLVs, like HIV, may also use co-receptors for infection.
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Feline cellular immune functions ADCC, NK, and in vitro generated cytolytic cells /Kooistra, Linda H. January 1984 (has links)
Thesis (Ph. D.)--University of Wisconsin--Madison, 1984. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographies.
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In vitro characterization of cell-mediated immune function in normal and feline leukemia virus-infected cats /Stiff, Mary Irene January 1982 (has links)
No description available.
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Erythroid aplasia and platelet abnormalities in cats induced by the Kawakami-Theilen strain of feline leukemia virus /Boyce, John Thomas January 1983 (has links)
No description available.
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Circulating immune complex studies during the preneoplastic stages of feline leukemia virus infection /Tuomari, Darrell L. January 1983 (has links)
No description available.
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