In order to address the contradictory reports on the rat trace element status during a vitamin 8-6 deficiency, Zn, Cu and Fe balance was assessed over 8 weeks in 3 groups of young male rats. Group 1 was the control group fed on a diet supplemented with 3,5 mg/kg of vitamin B-6. Group 2 was the marginally deficient group, fed a diet supplemented with 1,75 mg/kg of vitamin B-6 and Group 3 was the totally deficient group without any vitamin 8-6 in the diet. Diet, urine and fecal samples were analysed to determine the mean daily Zn, Cu and Fe balance for each group during each week of the study. Urinary xanthurenic acid (XA), plasma pyridoxal (PL), and plasma pyridoxal 5' -phosphate (PLP) were also analysed in order to determine the vitamin 8-6 nutritional status of each animal group. The totally deficient Group 3 showed decreased growth and food consumption when compared to the control and marginally deficient groups. There was a significant difference in XA excretion and plasma PLP and PL levels between all 3 groups with a progressive increase in XA excretion and a progressive decrease in PLP and PL levels from Group to Group 3. Although all groups were found to be in a positive balance for Zn, Cu and Fe, the totally deficient group was in a less positive balance compared to Groups 1 and 2. Furthermore, the percentage excretion of Zn and Cu, when compared to the intake, was greater in this group. This increased excretion, coupled with a decreased food intake, accounted for the lowered Zn and Cu balances in the totally deficient group. Fe excretion did not differ significantly between all 3 groups. The marginally deficient Group 2 showed no significant difference in growth, food consumption, trace element balance and excretion when compared to the control group, indicating the beneficial effects of even subminimal levels of vitamin B-6. This study confirms that the nutritional status of t~ace elements, especially that of Zn and CU, is related to the vitamin B-6 status of the animal. / Thesis (M.Sc.)-University of Durban-Westville, 1988.

No abstract available. / Thesis (M.Sc.)-University of Durban-Westville, 1989.

Thesis (M.S.P.H.)--University of Michigan, 1939.

Thesis (M.S.P.H.)--University of Michigan, 1939.

No description available.

Approved for public release, distribution unlimited. / This thesis proposes a linear model based on human physiology for the explanation of the Motion Sickness Incidence (MSI) data found in previously reported experiments. The major human sensory systems taken into account are vestibular, visual, and the interaction between these two. The model is validated against the previous descriptive model and the corresponding experimental data. The proposed model predicts MSI with adequate precision (less than Å 5%) in the frequency range between 0.07 Hz and 0.25 Hz. The difference between the proposed model and the previous descriptive model is increased at the outer frequency regions of the data.

No abstract available. / Thesis (M.Sc.)-University of Durban-Westville, 1992.

No abstract available. / Thesis (M.Sc.)-University of KwaZulu-Natal, Westville, 1992.

No description available.

Thesis (M.S. in Operations Research and M.S. in Modeling, Virtual Environments and Simulation)--Naval Postgraduate School, Sept. 2004. / Thesis Advisor(s): Michael McCauley, Nita Miller. Includes bibliographical references (p. 149-162). Also available online.