- About
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The Global ETD Search service is a free service for researchers
to find electronic theses and dissertations. This service is
provided by the
Networked Digital Library of Theses and Dissertations.
Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
Search results
Showing 1 to 10 of 100 (0.044 seconds)| 1 |
Lens-rupture; a new recessive gene in the mouse.Herer, Moe Lionel. January 1948 (has links)
Typewritten mss.
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The expression of rhino, hairless and naked genes in the house mouse.Wright, Annie Mary. January 1941 (has links)
No description available.
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Sorbitol dehydrogenase: gene structure, function and mutation李福基, Lee, Fuk-ki. January 1998 (has links)
published_or_final_version / Molecular Biology / Doctoral / Doctor of Philosophy
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Transport studies in primary cultures of mouse renal epithelial cellsBell, Cindy Lea. January 1986 (has links)
No description available.
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Structural organization of the mouse testin gene and characterization of its promoter sequence鄭智強, Cheng, Chi-keung. January 2000 (has links)
published_or_final_version / Zoology / Master / Master of Philosophy
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Molecular characterization of the insertional mouse mutant yellow submarine, Ysb李彥霆, Lee, Yin-ting. January 2002 (has links)
published_or_final_version / Biochemistry / Master / Master of Philosophy
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Chromosomal variations in Peromyscus boylei glasselli, P. boylei rowleyi, and P. StephaniSaxe, Debra Fay, 1952- January 1977 (has links)
No description available.
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CYTOGENETIC STUDIES ON GENUS MUS (RODENTIA, MURIDAE) WITH SPECIAL REFERENCES TO SPECIES FROM THAILANDMarkvong, Amara, 1945- January 1977 (has links)
No description available.
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Transport studies in primary cultures of mouse renal epithelial cellsBell, Cindy Lea. January 1986 (has links)
The Hyp (hypophosphatemic) mouse, a murine homologue of X-linked hypophosphatemia (XLH) in man, is a Mendelian disorder of phosphate (Pi) homeostasis. The mutant genotype is characterized by abnormal Pi transport at the brush border membrane (BBM) of the proximal tubule and a defect in renal metabolism of vitamin D$ sb3$. The exact nature of these defects has not been elucidated. / In order to determine if the defect is intrinsic to the renal cell or dependent upon an extrinsic humoral factor, I established primary cultures of renal epithelial cells from normal and Hyp mouse kidney. The cultures demonstrated several differentiated properties of epithelial cells of the renal proximal tubule, the site of the Pi transport defect in the Hyp mouse. / Primary cultures initiated from Hyp mice had decreased Pi transport (expressed as an uptake ratio, Pi/$ alpha$-MG), and increased production of 24,25 dihydroxyvitamin D$ sb3$. These results provide evidence for the intrinsic nature of the primary defect in the Hyp mouse. / This appears to be the first time that expression of a mutant transport gene has been demonstrated in cultured renal cells.
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Genetic control of the survival of trisomy 19 fetuses in miceTrasler, Tessa A. January 1987 (has links)
No description available.
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