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Brain derived neurotrophic factors (BDNF) and seprafilm?adhesion barrier on sciatic nerve regeneration in ratsLau, Chi-yan, Jane. January 2009 (has links)
Thesis (M. Med. Sc.)--University of Hong Kong, 2009. / Includes bibliographical references (p. 48-53).
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Enzyme studies in developing rat sciatic nerve and isolated Schwann cells /Knapp, Pamela Elise January 1983 (has links)
No description available.
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A chronological study of the degeneration phenomena of dorsal root ganglia cells following section of the sciatic nerve /Humbertson, Albert O. January 1962 (has links)
No description available.
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Brain derived neurotrophic factors (BDNF) and seprafilm® adhesion barrier on sciatic nerve regeneration in ratsLau, Chi-yan, Jane., 劉至欣. January 2009 (has links)
published_or_final_version / Orthopaedics and Traumatology / Master / Master of Medical Sciences
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Traumatic hindlimb neuropathies of dogs and cats : With special reference to the sciatic nerve and its originsSharp, N. J. H. January 1982 (has links)
No description available.
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Effects of chemical agents on the permeability of the nerve perineurium and spinal cord : a study using electrophysiological and electron microscopic techniquesTodd, Barbara Anne January 1995 (has links)
No description available.
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Myelin abnormalities in the optic and sciatic nerves of mice with GM1-gangliosidosisHeinecke, Karie A. January 2014 (has links)
Thesis advisor: Thomas N. Seyfried / GM1 gangliosidosis is a glycosphingolipid lysosomal storage disease caused by a genetic deficiency of acid b-galactosidase (β-gal), the enzyme that catabolyzes GM1 within lysosomes. Accumulation of GM1 and its asialo form (GA1) occurs primarily in the brain, leading to progressive neurodegeneration and brain dysfunction. Less information is available on the neurochemical pathology in optic nerve and sciatic nerve of GM1- gangliosidosis. Here we analyzed the lipid content and myelin structure in optic and sciatic nerve in 7 and 10 month old normal β-gal (+/?) and GM1-gangliosidosis β-gal (-/-) mice. Optic nerve weight was lower in the β-gal -/- mice than in unaffected β-gal +/? mice, but no difference was seen between the normal and the β-gal -/- mice for sciatic nerve weight. The concentrations of GM1 and GA1 were significantly higher in optic nerve and sciatic nerve in the β-gal -/- mice than in β-gal +/? mice. The content and composition of myelin-enriched cerebrosides, sulfatides, plasmalogen ethanolamines were significantly lower in optic nerve of β-gal -/- mice than in β-gal +/? mice, however cholesteryl esters were enriched in the β-gal -/- mice. No significant abnormalities in these myelin enriched lipids were detected in sciatic nerve of the β-gal -/- mice. The abnormalities in GM1 and myelin lipids in optic nerve of β-gal -/- mice were also associated with abnormalities in the X-ray diffraction pattern including myelin content in fresh nerves [M/(M +B)] and periodicity (d). With the exception of a slight reduction in myelin content, no abnormalities in the X-ray diffraction pattern were observed in sciatic nerve of β-gal -/- mice. The results indicate that neurochemical pathology is greater in optic nerve than in sciatic nerve of β-gal -/- mice. / Thesis (MS) — Boston College, 2014. / Submitted to: Boston College. Graduate School of Arts and Sciences. / Discipline: Biology.
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Molecular cloning and functional analysis of chondroitin 6-sulfotransferase (rat) in relation to post-traumatic nerve regenerationLiu, Jun, January 2004 (has links)
Thesis (Ph. D.)--University of Hong Kong, 2005. / Also available in print.
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Neurotoxicity of root canal medicaments a thesis submitted in partial fulfillment ... endodontics /Schippers, Douglas John. January 1986 (has links)
Thesis (M.S.)--University of Michigan, 1986.
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Neurotoxicity of root canal medicaments a thesis submitted in partial fulfillment ... endodontics /Schippers, Douglas John. January 1986 (has links)
Thesis (M.S.)--University of Michigan, 1986.
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