25‐hydroxycholecalciferol (25‐OHD3) formed via hepatic hydroxylation from vitamin D,
cholecalciferol, represents the precursor of the biologically active vitamin D hormone,
1,25‐dihydroxyvitamin D. Due to a higher absorption rate and the omission of one
hydroxylation, dietary supplementation of 25‐OHD3 instead of vitamin D3 is considered
to be more efficient as plasma concentrations of 25‐OHD3 are increased more
pronounced. The present review summarises studies investigating potential beneficial
effects on mineral homeostasis, bone metabolism, health status and performance in sows,
piglets and fattening pigs. Results are inconsistent. While most studies could not
demonstrate any or only a slight impact of partial or total replacement of vitamin D3 by
25‐OHD3, some experiments indicated that 25‐OHD3 might alter physiological processes
when animals are challenged, for example, by a restricted mineral supply.
| Identifer | oai:union.ndltd.org:DRESDEN/oai:qucosa:de:qucosa:91668 |
| Date | 28 May 2024 |
| Creators | Lütke-Dörhoff, Michael, Schulz, Jochen, Westendarp, Heiner, Visscher, Christian, Wilkens, Mirja R. |
| Publisher | Wiley |
| Source Sets | Hochschulschriftenserver (HSSS) der SLUB Dresden |
| Language | English |
| Detected Language | English |
| Type | info:eu-repo/semantics/publishedVersion, doc-type:article, info:eu-repo/semantics/article, doc-type:Text |
| Rights | info:eu-repo/semantics/openAccess |
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