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Pathogenicity of IgG-Fc desialylation and its association with Th17 cells in an animal model of systemic lupus erythematosus / 全身性エリテマトーデスの動物モデルにおけるIgG-Fc脱シアル化の病原性とTh17細胞との関連

京都大学 / 新制・課程博士 / 博士(医学) / 甲第24994号 / 医博第5028号 / 新制||医||1069(附属図書館) / 京都大学大学院医学研究科医学専攻 / (主査)教授 上野 英樹, 教授 椛島 健治, 教授 濵﨑 洋子 / 学位規則第4条第1項該当 / Doctor of Medical Science / Kyoto University / DFAM

Identiferoai:union.ndltd.org:kyoto-u.ac.jp/oai:repository.kulib.kyoto-u.ac.jp:2433/287181
Date23 January 2024
CreatorsNishida, Yuri
Contributors西田, 優理, ニシダ, ユリ
PublisherKyoto University, 京都大学
Source SetsKyoto University
LanguageEnglish
Detected LanguageEnglish
Typedoctoral thesis, Thesis or Dissertation
Rights許諾条件により本文は2024-06-10に公開, This is a pre-copyedited, author-produced version of an article accepted for publication in Modern Rheumatology following peer review. The version of record [Yuri Nishida and others, Pathogenicity of IgG-Fc desialylation and its association with Th17 cells in an animal model of systemic lupus erythematosus, Modern Rheumatology, 2023;, road054] is available online at: https://academic.oup.com/mr/advance-article-abstract/doi/10.1093/mr/road054/7193780?redirectedFrom=fulltext https://doi.org/10.1093/mr/road054
Relationhttps://doi.org/10.1093/mr/road054

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