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Biochemical studies of peripheral nerve metabolism with particular reference to the role of thiamine.

Our clinical interest in the peripheral neuropathies has prompted the present study of metabolism of peripheral nerves. Our experiments were designed to investigate possible metabolic defects in various conditions of disturbed nerve function and to consider their relevance to clinical problems. As is well known, demyelinating lesions of nerves can be caused by a wide variety of etiological agents and conditions, resulting in fairly similar clinical disturbances. The mechanism by which interference with normal function occurs is not fully understood.

Identiferoai:union.ndltd.org:LACETR/oai:collectionscanada.gc.ca:QMM.113666
Date January 1962
CreatorsHeller, Irving. H.
ContributorsElliott, K. (Supervisor)
PublisherMcGill University
Source SetsLibrary and Archives Canada ETDs Repository / Centre d'archives des thèses électroniques de Bibliothèque et Archives Canada
LanguageEnglish
Detected LanguageEnglish
TypeElectronic Thesis or Dissertation
Formatapplication/pdf
CoverageDoctor of Philosophy. (Department of Health Sciences.)
RightsAll items in eScholarship@McGill are protected by copyright with all rights reserved unless otherwise indicated.
Relationalephsysno: NNNNNNNNN, Theses scanned by McGill Library.

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