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Studies on the pathological mechanism of alopecia areata in C3H/HeJ mouse model / C3H/HeJモデルマウスを用いた円形脱毛症の病態メカニズムに関する研究

京都大学 / 新制・論文博士 / 博士(農学) / 乙第13497号 / 論農博第2901号 / 新制||農||1093(附属図書館) / 学位論文||R4||N5403(農学部図書室) / (主査)教授 谷 史人, 教授 佐々木 努, 教授 保川 清 / 学位規則第4条第2項該当 / Doctor of Agricultural Science / Kyoto University / DFAM

Identiferoai:union.ndltd.org:kyoto-u.ac.jp/oai:repository.kulib.kyoto-u.ac.jp:2433/276149
Date25 July 2022
CreatorsHashimoto, Kei
Contributors橋本, 惠以, ハシモト, ケイ
PublisherKyoto University, 京都大学
Source SetsKyoto University
LanguageEnglish
Detected LanguageEnglish
Typedoctoral thesis, Thesis or Dissertation
RightsThe contents described in Chapter 1 to Chapter 3 were originally published as follows: Hashimoto K, et al. Altered T cell subpopulations and serum anti-TYRP2 and tyrosinase antibodies in the acute and chronic phase of alopecia areata in the C3H/HeJ mouse model. J. Dermatol. Sci. 2021;104(1):21-29. DOI: 10.1016/j.jdermsci.2021.09.001 Hashimoto K, et al. Induction of alopecia areata in C3H/HeJ mice using cryopreserved lymphocytes. J. Dermatol. Sci. 2021;102(3):177-183. DOI: 10.1016/j.jdermsci.2021.04.009 Hashimoto K, et al. NLRP3 inflammasome activation contributes to development of alopecia areata in C3H/HeJ mice. Exp. Dermatol. 2022;31(2):133-142. DOI: 10.1111/exd.14432 Fig. G-1. and Fig. G-3. were originally published as follows: Darwin E, et al. Alopecia areata: Review of epidemiology, clinical features, pathogenesis, and new treatment options. Int. J. Trichol. 2018;10:51-60. DOI: 10.4103/ijt.ijt_99_17

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