Thesis (Ph. D.)--University of Wisconsin, 1964. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Bibliography: leaves 87-91.

Thesis (Ph.D.)--Hong Kong University of Science and Technology, 2006. / Includes bibliographical references (leaves 138-157). Also available in electronic version.

Previous measurements on CdS (I) have shown a temperature shift in the single electron spin resonance (ESR) impurity band line. At 34 GHz this shift was found to be: dg= - 2.5 x 10ˉ ⁴/°K dT in going from 4.2°K to 1.7°K. This thesis presents the construction and testing of a metal temperature controlled dewar for the purpose of measuring the shift over the temperature range from 40°K to 1.7°K using an X-band (9 GHz)spectrometer. The signal was observed between 1.7°K and 4.2°K but rapidly broadened and could not be seen at higher temperatures. Since the lowest attainable temperature of the metal dewar was around 5°K, it could not be used as planned. Further studies using glass dewars proved that the g-shift at 9 GHz was too small, to be measured. The theory for the g-shift is discussed and the performance of the dewar evaluated. / Science, Faculty of / Physics and Astronomy, Department of / Graduate

The electronic g-tensor in antimony doped cadmium sulphide has been measured at 1.1°K. A sample doped to a room temperature resistivity of 3.3 ohm-cm exhibited an anisotropic g-tensor with g// = 1.788 and g⊥ = 1.770. A single asymmetric line was observed. The asymmetry of this line was found to decrease with decreasing incident power. Further experiments to study this line shape are indicated. / Science, Faculty of / Physics and Astronomy, Department of / Graduate

In the present investigation, zinc was plated onto a cadmium crystal to determine the effect on the scattering parameter.

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Vision is a crucial aspect of life for humans and animals. Impaired vision can lead to reduced quality of life along with other complications. Cataracts are a leading cause of impaired vision and blindness worldwide. Cataracts develop as a process of aging, although several environmental and lifestyle factors increase the risk of this disease. The toxic metal cadmium (Cd) has been associated with cataract formation and other ocular diseases such as macular degeneration. Cadmium exposure experiments were conducted to investigate potential pathways or mechanisms by which Cd may contribute to cataract formation and ocular disease. Zebra fish larvae (72, 96, and 120 hours post fertilization), adult zebra sh (6-month male, 10-month male, and 10-month female) and the B3 human lens epithelial (HLE) cell line were acutely exposed to varying concentrations of Cd. Transcriptomic changes relative to control (0 μM Cd) were determined using microarray analysis for zebra sh larvae and RNA sequencing (RNA-Seq) for adult zebra sh and HLE cells. Gene Ontology (GO) enrichment analysis for the zebra sh larvae exposure (50 μM Cd for 4 or 8 hours) enriched the "retina development in camera-type eye" term, and genes involved in enrichment (dnmt1, ccna2, fen1, mcm3 and slbp) were down-regulated. Gene set enrichment analysis (GSEA) for the 10-month male zebra sh exposure (50 μM Cd for 4 hours) enriched the "embryonic eye morphogenesis" gene set and signi ficant genes involved in enrichment (tcf7l1a, pitx2, fzd8a, sfrp5, lmx1bb, mfap2, six3b, lum, phactr4b, and foxc1a) were down-regulated. GSEA for the 10-month female zebra sh (50 μM Cd for 4 hours) enriched the "photoreceptor cell differentiation" gene set and signi cant genes involved in enrichment (odc1, thrb, and ush2a) were up-regulated. GO enrichment analysis for up-regulated genes in the HLE cell exposure (10 μM Cd for 4 hours) enriched the terms "eye development" (22 genes) and "lens development in camera-type eye" (CITED2, SKIL, CRYAB, SLC7A11, TGFB2, EPHA2, BCAR3, WNT5B, and BMP4). These results show cadmium is capable of altering transcription of eye-related genes in both zebra sh and human models, which may contribute to the formation of ocular disease. Many of these genes are involved in lens and retina development, yet they are also associated with diseases in these eye structures. Future studies could assess the consequences of altered transcription of these genes which could help elucidate the mechanisms of these changes and the overall effect of cadmium exposure on ocular disease. Ultimately, our study characterized the regulation of eye-related genes in response to Cd exposure and provided valuable knowledge laying the foundation for identi fication of the molecular mechanisms contributing to ocular diseases. / Thesis / Master of Science (MSc) / The eye is a sphere-like organ which is important for visualizing your surroundings. It is composed of many different structures such as the cornea, lens and retina. Many eye diseases have been characterized by abnormalities in eye structures; for example, a cataract occurs when the lens becomes cloudy and unable to focus light while macular degeneration is defined by progressive deterioration of the retinal macula region. While these diseases can occur through the natural aging process, certain environmental factors can increase risk. Exposure to cadmium, a toxic heavy metal which causes negative effects in animals, has shown to be associated with eye disease like cataracts and macular degeneration. In order to expand on this knowledge, we exposed both zebrafish and human lens cells to cadmium. By utilizing different experimental methods such as microarray analysis and RNA sequencing, eye-related genes which were affected by cadmium were revealed. Identifying the relationship between eye diseases, cadmium and gene expression will help identify the mechanism by which cadmium contributes to eye disease formation.