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From language revalorisation to language revitalisation? : discourses of Maya language promotion in YucatánCru, Josep January 2014 (has links)
Against the background of worldwide processes of language abandonment that are taking place at an unprecedented and rapid pace, in the last two decades language revitalisation has become an ever more prominent area of academic research. This thesis looks at the ideological underpinnings of Yucatec Maya language promotion in the Yucatán Peninsula of Mexico, based on the discourses of both official institutions and grassroots actors. After introducing the historical processes that have led to the present sociolinguistic minorisation of speakers of Maya in the Yucatán Peninsula, I analyse salient themes for language policy and planning pointed out by activists and institutions. Both official and grassroots discourses gathered in the field overwhelmingly revolve around the key concepts of revalorisation and rescate. These notions undergird the strategies that most participants consider as necessary for Maya language promotion, namely, the drafting of specific language legislation; the use of Maya in the education system; and an emphasis on the development of literacy in Maya. While policies in these areas may have a positive impact on raising the status and public profile of Maya and may lead to its legitimation, I argue that they present considerable limitations for actual revitalisation, which I believe should be part of a wider sociopolitical movement coming from the grassroots. On the one hand, vertical language policies that emanate from official institutions, the school being a prominent one, have been central in the cultural and linguistic assimilation to Spanish of indigenous peoples in Mexico. On the other hand, institutional policies that replicate the essentialist tenets of hegemonic languages on minorised languages, such as standardisation, actually devalue plurilingual and mixed practices on the ground and raise the issue of purism, which in the case of Yucatán may be contributing to language shift to Spanish and hindering the revitalisation process. Seen as an alternative and complementary project that comes above all from the ground up, I maintain that grassroots language promotion beyond institutional settings and control is effectively working towards the revitalisation of Maya. Along these lines, the use of this language in social media and modern music genres by youths, as part of their expanding communicative repertoires and heteroglossic practices on the ground, is opening up promising spaces for its maintenance and reproduction.
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Ni-Ta系合金の研究 : (第1報) 機械的性質および耐変色性について / Studies on Ni-Ta Alloys : (Part 1) Mechanical Properties and Discoloration加我, 正行 24 March 1979 (has links)
日本歯科理工学会, 加我 正行 = KAGA, Masayuki, Ni-Ta系合金の研究 : (第1報) 機械的性質および耐変色性について = Studies on Ni-Ta Alloys : (Part 1) Mechanical Properties and Discoloration, 歯科理工学雑誌 19(48), 1978 OCT, pp.271-278 / Hokkaido University (北海道大学) / 博士 / 歯学
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Ni-Ta系合金の研究 : (第2報) 組織とかたさおよび陶材焼付について / Studies on Ni-Ta Alloys : (Part 2) Age-hardening and Metal-to-Ceramic Bonding加我, 正行 24 March 1979 (has links)
日本歯科理工学会, 加我 正行 = KAGA, Masayuki, Ni-Ta系合金の研究 : (第2報) 組織とかたさおよび陶材焼付について = Studies on Ni-Ta Alloys : (Part 2) Age-hardening and Metal-to-Ceramic Bonding, 歯科理工学雑誌 20(49), 1979 JAN, pp.44-51 / Hokkaido University (北海道大学) / 博士 / 歯学
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ポケット探針型および根分岐部用超音波スケーラーチップによる歯肉縁下スケーリング・ルートプレーニングの研究 / A study of subgingival scaling and root planing with pocket-probe type and furcation-probe type ultrasonic scaler菅谷, 勉 30 September 1992 (has links)
Hokkaido University (北海道大学) / 博士 / 歯学
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Long-term effects of prostaglandin E2 on the mineralization of a clonal osteoblastic cell line (MC3T3-E1) / 骨芽細胞様細胞株(MC3T3-E1)の石灰化に対するプロスタグランジンE2長期投与の効果Kajii, Takashi 25 March 1999 (has links)
共著者あり。共著者名: Kuniaki Suzuki, Masatake Yoshikawa, Tohru Imai, Akira Matsumotob and Shinji Nakamura. Elsevier Science Ltd., Takashi Kajii, Kuniaki Suzuki, Masatake Yoshikawa, Tohru Imai, Akira Matsumotob and Shinji Nakamura, Long-term effects of prostaglandin E2 on the mineralization of a clonal osteoblastic cell line (MC3T3-E1), Archives of Oral Biology, 44(3), 1999 MAR, pp.233-241. doi:10.1016/S0003-9969(98)00120-4. Journal Website: http://intl.elsevierhealth.com/journals/arob/ / Prostaglandin (PG) E2 is thought to be a mediator of the effect of mechanical stress on bone formation, but its effects on osteoblasts have not yet been fully described. Here, the effects of the continuous application of PGE2 and indomethacin, an inhibitor of prostaglandin G/H synthase (cyclo-oxygenase), on the proliferation, differentiation and mineralization of a clonal osteoblastic cell line, MC3T3-E1, were investigated. The cells were cultured in media with either a high (1 μg/ml) or a low (1 ng/ml) concentration of PGE2, with indomethacin (1 μg/ml) and, as a control, with neither agent. The effects of PGE2 and indomethacin were assessed quantitatively. Indomethacin and a high concentration of PGE2 increased the total protein compared to the control and low-PGE2 cultures. 7 days after confluence, alkaline phosphatase (ALP) activity within the cells and extracellular matrices increased. This increase was highest with indomethacin and lowest with a high concentration of PGE2. ALP activity also increased in the medium, but only 21 days after confluence; the effects of the agents were similar to those on the cells and matrices. The accumulation of calcium, inorganic phosphate and hydroxyproline was highest with indomethacin. PGE2 production was at its maximum when the cells were at confluence and was inhibited by indomethacin. Specific [3H]PGE2 binding to the microsomal fraction of the cell was also measured to examine the expression of the PGE2 receptor. The amount of [3H]PGE2 binding per mg of protein was highest at confluence, then decreased and again increased in the mineralizing stage. These results suggest that indomethacin increases ALP activity and the accumulation of mineralized tissue in MC3T3-E1 cells, presumably by inhibiting the production of PGE2. PGE2 could signal the suppression of mineralization as early as confluence. / Hokkaido University (北海道大学) / 博士 / 歯学
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放射線照射ラットの抜歯創治癒過程に関する形態学的研究 / Morphological studies on the healing process of extraction wound in irradiated rats飯塚, 正 24 March 1984 (has links)
歯科基礎医学会, 飯塚 正 = Tadashi Iizuka, 放射線照射ラットの抜歯創治癒過程に関する形態学的研究 = Morphological studies on the healing process of extraction wound in irradiated rats, 歯科基礎医学会雑誌, 26(3), SEP 1984, pp.745-785 / Hokkaido University (北海道大学) / 博士 / 歯学
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YAGレーザー照射によるラット顎下腺の損傷と修復に関する形態学的研究 / Morphological studies on the regeneration of rat submandibular gland following YAG laser irradiation高橋, 茂 25 March 1992 (has links)
歯科基礎医学会, 高橋茂 = Shigeru Takahashi, YAGレーザー照射によるラット顎下腺の損傷と修復に関する形態学的研究 = Morphological studies on the regeneration of rat submandibular gland following YAG laser irradiation, 歯科基礎医学会雑誌, APR 1993, 35(2), pp.115-146 / Hokkaido University (北海道大学) / 博士 / 歯学
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実験的歯の移動時における圧迫側歯槽骨に生じる背部骨吸収と血管分布 / Rear resorption at the pressure side incident in orthodontic tooth movement日下部, 豊寿 25 March 1998 (has links)
歯科基礎医学会, 日下部 豊寿 = Toyohisa Kusakabe, 実験的歯の移動時における圧迫側歯槽骨に生じる背部骨吸収と血管分布 = Rear resorption at the pressure side incident in orthodontic tooth movement, 歯科基礎医学会雑誌, 39(6), DEC 1997, pp.623-640 / Hokkaido University (北海道大学) / 博士 / 歯学
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The numerical synthesis and inversion of acoustic fields using the Hankel transform with application to the estimation of the plane wave reflection coefficient of the ocean bottomJanuary 1983 (has links)
Douglas R. Mook. / Originally published as thesis (Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, M.S., 1984). / Includes bibliographical references. / Advanced Research Projects Agency Contract no. N00014-81-K-0742 NR-049-506 National Science Foundation Grant ECS80-07102
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La ópera Electra de Strauss y Hofmannsthal: una recepción de la tragedia de Sófocles en la Viena finisecularGutiérrez Silva, Francisco January 2017 (has links)
Informe de Seminario para optar al grado de Licenciado en Lengua y Literatura Hispánica mención Literatura / Proyecto Fondecyt de iniciación N°11140911
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