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Role of lysosomal iron in oxidative stress-induced cell deathKurz, Tino January 2006 (has links)
No description available.
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22 |
Excitatory amino acid-induced oligodendrocyte cell death in vitro : mechanisms and protective strategiesRosin, Claudia January 2004 (has links)
No description available.
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23 |
Apoptosis in intestinal epithelium during regenerationO'Connor, M. G. January 2004 (has links)
No description available.
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Modes and mechanisms of cell death induced by reactive nitrogen and oxygen speciesin pancreatic β-cellsHosker, Bill Stephen January 2004 (has links)
No description available.
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Identification of KERES, a novel component of the apoptosis pathway in Drosophila melanogasterDeas, Emma Elizabeth January 2005 (has links)
No description available.
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Mitochondrial biochemistry and its role in apoptosis : focus on invasion/metastasisSimmons, Tracy Dawn January 2004 (has links)
No description available.
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Regulation of cell survival by caspase-mediated SRF cleavageGreaney, Peter January 2004 (has links)
No description available.
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28 |
A study of the structure and function of tumour necrosis factor receptor superfamily membersKimberley, Fiona January 2004 (has links)
No description available.
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29 |
Analysis of the HIF-p VHL interactionMole, David January 2002 (has links)
No description available.
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30 |
The molecular mechanisms of calicivirus-induced apoptosisBrennan, Benjamin January 2008 (has links)
The Caliciviridae family are a group of small, non-enveloped viruses that contain a single stranded positive sense RNA genome approximately 7.5 kb in size. Several members of this virus family are significant pathogens of both animals and man. Perhaps the most well known member is Norovirus, which is reported to be the leading cause of viral gastroenteritis worldwide (approx. 95% of cases). Despite this, the molecular effects of the virus on host cells and pathogenicity of the disease has not been well reported due to the inability to grow most of these viruses in cell culture. Two members of the Calciviridae,, however, have been shown to replicate efficiently in certain cell lines. For this reason, these viruses are being used as models to study virus/host interactions. The aim of this project was to (i) further characterise the molecular mechanisms of FCV-induced apoptosis by examining the events that occur upstream of bax translocation to the mitochondria; (ii) examine the molecular mechanisms of apoptosis occurring during infection of CRFK cells with virulent systemic disease causing isolates of FCV and (iii) perform preliminary investigations into the mechanism of cell death occurring during MNV-1 infection.
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