Previous research indicates an increase in the prevalence of depression around adolescence, especially for females. Research suggests depressogenic cognitions play an essential role in the development of depression and may mediate the relation between risk factors and depression. Research has also shown the family environment, negative life events, and maternal depression are all related to the development of depressogenic cognitions. Additionally, few studies have tested models of depression while measuring both anxiety and depressive symptoms despite the high rates of comorbidity between the two disorders. The current study used path analytic techniques to integrate correlates of depression while accounting for comorbid anxiety symptoms in comprehensive model of depression for early adolescent girls. Participants included 203 girls, aged 9-14, along with their mothers. Participants completed self-report measures of the family environment, cognitive triad, and negative life events. Mothers of participants completed a self-report measure of psychopathology. Participants also completed a semi-structured diagnostic interview, which served as the measure for symptoms of depression and anxiety. Results supported previous literature finding a more depressogenic cognitive triad was significantly associated with higher depressive severity. Family environments, characterized by more cohesive and less conflictual family relationships, more communication, and higher engagement in social/recreational activities, were significantly associated with a more positive cognitive triad. Additionally, more negative life events were significantly associated with a more depressogenic cognitive triad. Both family social/recreational activities and negative life events had significant indirect effects on depression. Results indicated a strong relation between anxiety and depression, with anxiety having a significant positive direct effect on depression. The pathways from maternal depression and anxiety to the cognitive triad, anxiety symptoms to the cognitive triad, as well as family environment variables, maternal depression and anxiety and negative life events to anxiety symptoms were not found to be significant. Results from an exploratory analysis suggest anxiety may moderate the relation between the cognitive triad and depression. Implications of these results, limitations, and recommendations for future research are provided. / text

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This document proposes a study to ascertain if a relationship exists between levels of child and parent anxiety symptoms during an intervention designed to decrease anxiety in youth. This document systemically describes family variables related to anxiety in youth at the individual, parent-child, marital, whole family, and extra familial subsystem levels, in addition to previous research analyzing parental anxiety and the direction of change between child and parent anxiety during youth-focused interventions. The study involves gathering anxiety symptom data from parent and child participants at 14 time points and analyzing it using dependent samples t-test, regression, and ANOVA repeated measure analyses. These analyses serve to answer the research questions of whether child and parent anxiety symptoms improve in a youth-focused CBT intervention for anxiety from pre- to post-treatment, whether there exists a significant relationship between the severity of anxiety symptoms in youth and parents surrounding a youth-focused anxiety intervention, and whether this relationship is consistent. / text

The overall goal of the current study was to determine whether experiences of relational and physical victimization were related to anxiety and depressive symptoms in a sample of emerging adults. This study also investigated whether these associations were moderated by gender, as well as by sympathetic nervous system (SNS) and parasympathetic nervous system (PNS) reactivity to peer stress. Although work in this area has focused on children (e.g., Cullerton-Sen & Crick, 2005; Rudolph et al., 2009), it appears the presence and function of victimization changes with age, and the negative effects of victimization can last through early adulthood (e.g., Gros et al., 2010; Kumpulainen et al., 1999; Roth et al., 2002). Despite the potential for victimization to influence outcomes in emerging adults, research on these associations is lacking in this age group (Heilbron & Prinstein, 2008). A goal of the current study was to examine these processes in an older sample. Additionally, as individuals may react to peer victimization differently, factors that may help explain these differences were investigated. Specifically, evidence suggests that the interaction of the SNS and the PNS may serve as a moderator in the relationship between stressors and adjustment outcomes (Cummings et al., 2007; El-Sheikh et al., 2009; ObradoviÄ? et al., 2010). Further, research suggests that different patterns of interaction of the SNS and the PNS provide important information in the prediction of adjustment outcomes (El-Sheikh et al., 2009) and that both systems must be examined in order to more fully understand the relationship between physiological reactivity and adjustment outcomes (Beauchaine, 2001). Thus, in the current study, the interaction between two physiological measures, SNS reactivity to stress (as measured by skin conductance reactivity [SCL-R]) and PNS reactivity to stress (as measured by respiratory sinus arrhythmia [RSA-R]), was examined as a moderator of the association between peer victimization and adjustment outcomes. The moderating role of gender was also examined. Two hundred and forty-six emerging adults participated in the current study (74% female; Mage = 18.77) and were recruited from introductory psychology courses at a northeastern public university. Participants' SCL-R and RSA-R were assessed using a stress protocol during which they discussed an experience of relational victimization (e.g., being left out). Levels of relational and physical victimization, anxiety and depressive symptoms, and gender were gathered using self-report. Findings suggested that both physical and relational victimization were related to both anxiety and depressive symptoms. Additionally, females were more likely to experience relational victimization than males, while males were more likely to experience physical victimization than females. Relational victimization was related to depressive symptoms only in individuals demonstrating coactivation (i.e., blunted RSA withdrawal and increased SCL-R) and coinhibition (i.e., RSA withdrawal and blunted SCL-R) patterns of stress reactivity, although the interaction for this effect only approached conventional levels of statistical significance. These patterns may have emerged as a result of the breakdown of regulation in the physiological response to stress, with either the SNS or the PNS failing to perform adequately (El- Sheikh & Erath, 2011; El-Sheikh et al., 2009). These findings suggest that experiences of victimization are related to negative adjustment outcomes in emerging adults, as well as highlight potential areas that may serve as mechanisms for future interventions.

abstract: Anxiety sensitivity (AS; the fear of anxiety-related bodily sensations) has been earmarked as a significant risk factor in the development and maintenance of pathological anxiety in adults and children. Given the potential implications of heightened AS, recent research has focused on investigating the etiology and developmental course of elevated AS; however, most of this work has been conducted with adults and is retrospective in nature. Data from college students show that early anxiety-related learning experiences may be a primary source of heightened AS levels, but it remains unclear whether AS in children is linked to their learning experiences (i.e., parental reinforcement, modeling, punishment, and/or transmission of information about anxiety-related behaviors). Based on AS theory and its iterations, an emerging theoretical model was developed to aid further exploration of the putative causes and consequences of heightened AS levels. Using a sample of 70 clinic-referred youth (ages 6 to 16 years old; 51.4% Hispanic/Latino), the present study sought to further explicate the role of learning in the development of AS and anxiety symptoms. Results suggest that childhood learning experiences may be an important precursor to heightened AS levels and, subsequently, increased experiences of anxiety symptoms. Findings also indicate that some youth may be more vulnerable to anxiety-related learning experiences and suggest that culture may play a role in the relations among learning, AS, and anxiety symptoms. / Dissertation/Thesis / M.A. Psychology 2012

abstract: This dissertation used the risk and resilience framework to examine the associations between perceived racial discrimination, family racial socialization, nativity status, and psychological distress. Regression analyses were conducted to test the links between perceived racial discrimination and psychological distress and the moderation on these associations by family racial socialization and nativity status. Results suggest, for U.S.-born adolescents, cultural socialization strengthened the relation between subtle racial discrimination and anxiety symptoms. In addition, promotion of mistrust buffered the relations of both subtle and blatant racial discrimination on depressive symptoms. For foreign-born adolescents, promotion of mistrust exacerbated the association between blatant racial discrimination and depressive symptoms. Overall, the findings revealed the detrimental effects of perceived racial discrimination on the mental health of Asian American adolescents, how some family racial socialization strategies strengthen or weaken the relation between perceived racial discrimination and psychological distress, and the different ways foreign-born and U.S-born adolescents may interpret racial discrimination and experience family racial socialization. / Dissertation/Thesis / Ph.D. Family and Human Development 2012

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Background: The lack of adherence to drug therapy is a major problem; it can contribute to significant deterioration of disease and increased health-care costs. Improving medication adherence is a big challenge; there is no simple solution to the problem. It is thus essential to improve our knowledge of non-adherence (NA) and its causes. Aims: The aims of the thesis were to study the influence of gender and psychological distress on self-reported, intentional and unintentional non-adherent behaviour, and to investigate the reasons for NA. Methods: A population-based study that included a postal questionnaire was carried out in a cross-section of the general Swedish population (n=7,985, aged 18-84 years). The response rate was 61.1% (n=4,875) and current prescription drug use was reported by 2,802 participants. The questionnaire covered use of prescription drugs, NA to the drug regimens, reasons for NA, economic status, attitudes to drugs, and the presence of somatic or mental problems, and also included the Hospital Anxiety and Depression Scale questionnaire. Results: The results showed differences in various self-reported non-adherent behaviour patterns and reasons for NA between the genders. In most cases, these remained after controlling for confounders such as socioeconomic factors and attitudes to drugs that are known to differ between women and men. Associations were also found between symptoms of anxiety and/or depression and the presence of intentional or unintentional non-adherent behaviour (with a stronger average association for intentional NA), and between anxiety/depression and some of the reasons given for NA, e.g. adverse drug reactions (ADRs). Conclusions: Although it was not possible to confirm causal relationships, this thesis emphasises the effects of gender and psychological distress on NA. In summary, both gender and anxiety and/or depression influenced non-adherent behaviour and the reasons given for NA. For instance, ADRs seemed to influence the decision not to take the drug as prescribed, especially among women and participants under psychological distress. It is suggested that a deep understanding of the causes of NA and of the impact of gender and psychological distress on the outcomes would help those aiming to improve adherence to prescribed medication.

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Fearful temperament represents one of the robust predictors of the development of child and adolescent anxiety. Not all children with fearful temperament unvaryingly develop anxiety, however. Diverse processes resulting from the interplay among attention, cognitive control, and motivational system drive the trajectories toward more adaptive or maladaptive directions. In this study, I examined various factors that underlie the association between fearful temperament at age 9 and adolescent anxiety symptoms including attention biases, different components of effortful control, and frontal EEG asymmetry. 78 children participated in this study. Results indicate that fearful temperament at age 9 significantly predicted adolescent anxiety symptoms. This association, however, was moderated by children's effortful control and frontal EEG asymmetry at age 9. Specifically, fear at 9 years predicted adolescent anxiety only when children had low attentional control, low inhibitory control, low activation control, and exhibited greater right activation from baseline to task. The associations between AB and fearful temperament as well as anxiety were not significant. The association between fear at 9 years and sustained AB during adolescence, however, was moderated by children's attentional control, inhibitory control, and frontal EEG asymmetry at age 9. Specifically, fear predicted attention biases away from threat when children had high attentional control, high inhibitory control, and showed greater left activation. The findings will be discussed in terms of the roles of attention biases in the development of anxiety and how different components of effortful control and frontal EEG asymmetry contribute to the resilience process. / Doctor of Philosophy / Anxiety disorders represent one of the most commonly occurring mental health problems in childhood and adolescence. Children who tend to show wariness and distress to negative stimuli are more likely to have anxiety. Not all children with fearful temperament develop anxiety, however. Certain individual characteristics can protect fearful children from having anxiety symptoms. In this study, I examined the roles of attentional biases toward threat (AB), different components of self-regulation (EC), and the asymmetrical frontal brain activation (FA) in changing the relation between fearful temperament and anxiety. 78 children participated in this study. Results indicated that adolescents were at higher risk for anxiety if they showed high fearful temperament at age 9. However, the risk could be attenuated if children were better able to control their attention and behaviors, and exhibited greater left activation from resting to a mildly stressful situation at age 9. In addition, fearful children were better able to direct attention away from threat during adolescence if they were better able to control their attention and behaviors, and exhibited greater left activation from resting to a mildly stressful situation at age 9. The findings provide suggestions for early identification and intervention of children who are more vulnerable to anxiety during adolescence.