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The Global ETD Search service is a free service for researchers
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Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
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Showing 1 to 7 of 7 (0.034 seconds)Spelling suggestions: "subject:"biofluid mechanics"" "subject:"biofluid echanics""
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Role of Elasticity in Respiratory and Cardiovascular FlowSubramaniam, Dhananjay Radhakrishnan 23 July 2018 (has links)
No description available.
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CFD study on effect of branch sizes in human coronary arteryShrestha, Liza 01 December 2010 (has links)
Atherosclerosis is a term coined to describe a state in which arterial wall thickens due to the accumulation of fatty materials like cholesterol. Though not completely understood, it is believed to occur due to the accumulation of macrophage white blood cells and promoted by low density lipoprotein. Increase in accumulation of plaque leads to enlargement of arteries as arterial wall tries to remodel itself. But eventually the plaque ruptures, letting out its inner content to blood stream. The ruptured plaque clots and heals and shrinks down as well but leaves behind stenosis - narrowing of cross section. Depending on the degree of stenosis blood supply from the artery to its respective organ could decrease and even get blocked completely. Frequently, as the vulnerable plaques rupture, thrombus formed as such could flow through bloodstream towards smaller vessels and block them, leading to a sudden death of tissues fed by that vessel. If the plaques do not rupture and artery gets enlarged to a great extent then it results in an aneurysm. Such blockage of coronary arteries in heart can lead to myocardial infarction - heart attack, in carotid arteries in brain can lead to what is called a stroke, in peripheral arteries in legs can lead to ulcers, gangrene (death of tissue) and hence loss of leg, in renal arteries can lead to kidney malfunction. The most disturbing fact about atherosclerosis is the inability to detect the disease in preliminary stages. As stated by Miller (2001), most of the times coronary artery disease (CAD) gets diagnosed only after 50-75 percent occlusion of arteries.
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流体を伝えるつぶれやすい管の安定性解析 (剥離点の移動に伴う擾乱と下流流路の長さの影響について)青松, 達哉, AOMATSU, Tatsuya, 松崎, 雄嗣, MATSUZAKI, Yuji, 池田, 忠繁, IKEDA, Tadashige 04 1900 (has links)
No description available.
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Computational Flow Modeling of Human Upper Airway BreathingMylavarapu, Goutham 16 September 2013 (has links)
No description available.
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非対称分布声帯モデルによる疾患時の発声の数値解析 (第２報, 非対称な声帯振動の数値シミュレーション解析)青松, 達哉, AOMATSU, Tatsuya, 松崎, 雄嗣, MATSUZAKI, Yuji, 池田, 忠繁, IKEDA, Tadashige 03 1900 (has links)
No description available.
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非対称分布声帯モデルによる疾患時の発声の数値解析 (第１報, 発声開始肺圧の数値解析)青松, 達哉, AOMATSU, Tatsuya, 松崎, 雄嗣, MATSUZAKI, Yuji, 池田, 忠繁, IKEDA, Tadashige 03 1900 (has links)
No description available.
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Moderate Reynolds number flow. Application to the human upper airways.Van Hirtum, Annemie 30 June 2011 (has links) (PDF)
The study of fluid flow is an amasingly ordinary as well as fascinating subject. During the past few years I had the opportunity to work as a researcher in the field of fluid flow modelling applied to airflow through the human upper airways and related phenomena such as speech production, . . . The current document is a brief report on the research to which I participated aiming a small contribution to this rich and stimulating research area.
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