In the process of deflagration-to-detonation transition (DDT) in reactive gases, the flame typically accelerates first to the choked flame condition (known as a Chapman-Jouguet deflagration), where it propagates at the sound speed with respect to the product gases. Subsequently, the choked flame may transit to a detonation. In the present study, the transition length from choked flames to detonations was measured experimentally in laboratory-scale experiments in methane, ethane, ethylene, acetylene, and propane with oxygen as oxidizer. The choked flames were first generated following the quenching of an incident detonation after its interaction with cylindrical obstacles with two different blockage ratios, 75\% and 90\%. Comparison with a recently proposed model confirms that these are Chapman-Jouguet deflagrations. The subsequent acceleration was monitored via large-scale time-resolved shadowgraphy. The mechanism of transition was found to be through the amplification of transverse waves and hot spot ignition from local Mach reflections. The transition length was found to correlate very well with the mixture's sensitivity to temperature and pressure fluctuations. These fluctuations could be connected to a unique parameter (X), introduced by Radulescu. The parameter is the product of the non-dimensional activation energy (Ea/RT) and the ratio of chemical induction to reaction time (ti/tr). Mixtures with a higher X were found to be more prompt to hot spot ignition and amplification of the fast flame into detonations. The run-up distance for unstable mixtures was found to be much shorter than anticipated from a model neglecting the fluctuations in a 1-D framework. The run-up distance was also correlated to the detonation cell size, yielding LDDT ~ 7 - 50 cells, with the proportionality coefficient depending on X and the obstacle blockage ratio. Finally, a unique correlation for the run-up distance is proposed, yielding LDDT ~ 3000 c tr, where c is the sound speed in the shocked non-reacted gas, valid for large X.

Proefschrift--Utrecht. / Summaries in English and Dutch. "Stellingen": [2] p. inserted. Bibliography: p. 109-114.

Thesis (Ph. D.)--University of Wisconsin--Madison, 1975. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.

Thesis (M.A.)--University of Wisconsin--Madison, 1973. / eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.

No description available.

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Thesis (M.S.P.)--University of Michigan, 1943.

Thesis (M.S.P.)--University of Michigan, 1943.

In the first experiment, sexually immature male and female coho salmon were exposed for 21 days to dietary DDT at a level of 10 or 100 parts per million (ppm), or to methoxychlor at a level of 100 ppm. Exposure to 100 ppm DDT was found to increase the level of liver microsomal Cytochrome P-450 from a control level of 1.32 ± .04 nmoles/1000 mg liver to a level of 1.98 ± .04 nmoles/1000 mg liver, a statistically significant difference (P<.001). None of the treatments were found to affect the hepato-somatic index. In the second experiment, liver slices from sexually maturing male and female coho salmon fed 100 ppm DDT for 21 days or a control diet were incubated with 4-C¹⁴-estradiol-17β in vitro. Metabolites produced were extracted with dichloromethane, separated by thin-layer chromatography and assayed using scintillation counting techniques. Produced in the incubation were estrone, estriol and one other unidentified polar metabolite. DDT treatment was found to significantly increase the amount of estriol and unidentified metabolite produced (P< .001). In the third experiment, sexually maturing male and female coho salmon fed 100 ppm DDT for 21 days or for 7 days or a control diet were injected with 625,500 dpm 4-C¹⁴-estradiol-17β and permitted to metabolize the hormone in vivo. Serial blood samples were extracted, chromatographed and subjected to scintillation counting techniques to obtain values for Metabolic Clearance Rate, Half-life Time and Volumes of Distribution of the injected steroid. It was found that ingestion of DDT had no significant effect upon any of these parameters (P<.001). The pattern of metabolites produced in vivo closely resembled that produced in vitro. The evidence presented in this study suggests that enhancement of the activity of the Mixed Function Oxidase system in coho salmon may occur upon ingestion of an organochlorine insecticide, but that the phenomenon may have little significance in vivo. / Science, Faculty of / Zoology, Department of / Graduate