Conspiracy theory beliefs have become ubiquitous within our society. One cannot partake in any form of media without confronting different conspiracy theories, such as beliefs that the Earth is flat, that birds are not real, that the moon landing was fake, or that vaccines cause autism spectrum disorder. Conspiracy theories are beliefs that revolve around false explanations of public and political events concerning a secret organization with malicious intent. What makes people vulnerable to conspiracy theory beliefs? Prior research indicates that people often adopt conspiracy beliefs in an attempt to reduce feelings of anxiety, uncertainty, and threat. Unfortunately, prior research also suggests that conspiracy theory beliefs might not relieve these negative emotional states. As a result, one might conclude that conspiracy theory beliefs are misguided attempts to regulate one’s emotional state during times of anxiety and uncertainty. The goal for the current research was to gain a greater understanding of who may be more susceptible to conspiracy theory beliefs. As prior research has shown that adverse childhood experiences (ACEs) often predict poorer emotion regulation, we wanted to explore whether ACEs may predict conspiracy theory beliefs as explained through emotion dysregulation. In this exploratory research, we recruited 719 participants online through Sona at East Tennessee State University (Mage= 19.98) and asked participants to complete two scales to measure conspiracy theory beliefs, the Generic Conspiracist Beliefs Scale (GCB) and the Conspiracy Mentality Scale (CM), as well as self-report measures of ACEs and emotion dysregulation. Results showed that ACEs did not directly predict conspiracy theory beliefs; however, when emotional dysregulation was included as a mediator, ACEs predicted conspiracy theory beliefs on both GCB and CM scales. This suggests that people who indicated experiencing more ACEs within the first 18 years of life tended to score higher in emotion dysregulation and will have more difficulty regulating their own emotions when it comes to social and environmental problems. Because of this, individuals who experienced more ACEs may rely on external factors and maladaptive coping strategies, such as conspiracy theory beliefs, to regulate their negative emotional states. Although ACE scores were not a direct determining factor in conspiracy theory beliefs, they can be used to reveal and better understand other maladaptive traits and the possibility of developing psychological disorders in the future. As this research was exploratory, future research will need to confirm these findings, ideally with a more diverse sample in age, race and educational background. Despite these limitations, the current study aids in our understanding about who may be more susceptible to engaging in conspiracy theory beliefs and can inform about possible interventions in the future.

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According to adult attachment theory (Hazan and Shaver, 1987), people's levels of insecure attachment, both anxious and avoidant, are associated with their abilities to regulate emotions in a relational context. This study is the first to test emotion dysregulation as a mediator for the relationships between levels of insecure attachment and psychological aggression using dyadic data. Cross-sectional, self-report data were collected from 110 couples presenting for couple or family therapy at an outpatient clinic. Data were analyzed using path analysis informed by the Actor-Partner Interdependence Mediational Model (APIMeM; Ledermann, Macho and Kenny, 2011). While the findings did not support a mediating role of emotion dysregulation between levels of anxious or avoidant attachment and psychological aggression, results indicated direct partner effects between people's own levels of anxious attachment and their partners' psychological aggression. Higher levels of anxious attachment were associated with higher levels of emotion dysregulation in both males and females; higher levels of avoidant attachment were only associated with higher levels of emotions dysregulation in females. Limitations and clinical implications for couple therapists are discussed. / Master of Science

<p dir="ltr">Adolescence is a key developmental period characterized by increased maladaptive risky behaviors. Two related, but distinct constructs, urgency (the tendency to act rashly in response to strong negative or positive emotions) and emotion dysregulation are important risk factors for engaging in maladaptive risky behaviors. Thus far, research has largely agreed that these two risk factors are highly correlated; however, the causal direction between these constructs is less understood. The goal of the current study is to determine whether urgency predicts emotion dysregulation among adolescents. This project is a secondary data analysis of a larger study designed to test the effectiveness of a school-based intervention for youth at risk of maladaptive risk taking, as compared to a control group of youth enrolled in a health class. The current study utilized data from the control youth (n=544, 49.8% female, M_age=14.22, SD=0.52) to test whether urgency at baseline predicts change in emotion dysregulation over a nine-week period, and whether that relationship differs across boys and girls. Results found that negative, but not positive, urgency significantly predicted emotion dysregulation change (negative urgency: <i>b</i>=0.11, <i>p</i>=0.03; positive urgency: <i>b</i>=0.03, <i>p</i>=0.54). Gender did not moderate either relationship (<i>p</i>’s>0.30). This work provides initial evidence of a temporal relationship between negative urgency and changes in emotion dysregulation. The next step is to determine whether negative urgency imparts risk for malapative behaviors through its effect on emotion dysregulation. This program of research may lead to better identification of effective interventions to decrease negative urgency-based risk-taking and better identify those at risk of developing maladaptive risk-taking.</p>

The aim of the present study was to investigate the link between ruminative processes and dysregulated behaviors. Accordingly, the emotional cascade model (Selby et al., 2008 / 2009) was tested in a sample of Turkish university students using structural equation modeling. The emotional cascade model posits that the link between emotional and behavioral dysregulation may be through emotional cascades, which are repetitive cycles of rumination and negative affect that result in an increased attention paid to the emotional stimuli and intensification of emotional distress. Dysregulated behaviors, such as non-suicidal self-injury, are used in order to break this cycle and distract the person from ruminative processes. In the first part of the study, a common measure of cognitive emotion regulation, namely The Cognitive Emotion Regulation Questionnaire (CERQ / Garnefski et al., 2001) was adapted into Turkish and its psychometric properties were investigated. Results suggested that the Turkish version of the CERQ is a reliable and valid meausure of cognitive coping. Next, a structural equation model was tested to assess the relationship between emotional cascades (as indicated by rumination, thought suppression, catastrophizing) and behavioral dysregulation (as indicated by binge eating, non-suicidal self-injury, excessive reassurance-seeking, and drinking to cope). The results showed that the emotional cascades are associated to behavioral dysregulation. This relationship, however, did not remain significant when the effect of current psychological distress on behavioral dysregulation was controlled for. The importance and possible implications of the present study was discussed.

Emotion dysregulation and rumination are involved in the development, maintenance, and treatment of both posttraumatic stress disorder (PTSD) and sleep disturbances. We examined if and how these factors influenced the nature of the relationship between PTSD symptom severity and subjective sleep disturbances among trauma-exposed individuals. Using data gathered from a community sample of 199 trauma-exposed individuals (Mean age = 35.48; 59.80% female), we examined whether there were stronger significant associations between greater PTSD symptom severity and poorer sleep quality/lower sleep quantity at higher (vs. lower) levels of (1) negative emotion dysregulation and positive emotion dysregulation (both included in the same model) and (2) rumination. Participants recruited via Amazon's Mechanical Turk completed the PTSD Checklist for DSM-5, Brief Version of the Difficulties in Emotion Regulation Scale, Difficulties in Emotion Regulation Scale – Positive, Ruminative Thought Style Questionnaire, and sleep quality/quantity items from the Pittsburgh Sleep Quality Index. Results of simple and additive multiple moderation analyses showed that neither negative/positive emotion dysregulation nor rumination moderated the relationships between PTSD symptom severity and sleep quality/quantity. Exploratory analyses showed that negative emotion dysregulation (when examined independently) moderated the relationship between PTSD symptom severity and sleep quality. There were also significant associations between poorer sleep quality/lower sleep quantity and greater PTSD symptom severity at low to average levels of negative emotion dysregulation coupled with any level of positive emotion dysregulation. Findings inform theoretical perspectives on the PTSD-sleep relationship and clinical applications of targeting emotion dysregulation and rumination in the treatment of PTSD symptoms and sleep disturbances for trauma-exposed individuals.

Binge Eating Disorder (BED) is characterized by recurrent episodes of binge eating without the use of compensatory behaviors. Functional accounts of BED propose that negative affect is an antecedent to binge eating because binge eating serves to alleviate negative affect. However, previous studies investigating the association between negative affect and binge eating have yielded inconsistent findings, perhaps due to individual vulnerability factors that moderate the effects of negative affect on binge eating behavior. As one candidate, the current study investigated emotion regulation strategies that may be implicated in the maintenance of binge eating in BED, particularly under conditions of negative affect: brooding rumination, distress tolerance, and mood-related expectancies for eating. These emotion regulation strategies were: a) compared in 38 women with BED vs. 36 non-eating disordered female controls, b) examined in relation to markers of current binge eating severity among BED women, and c) used as predictors of caloric intake and urge to eat in response to a personally-relevant dysphoric mood induction upon presentation of snack foods in a "taste task." Results revealed that women with BED endorsed higher brooding rumination, more positive expectancies that eating serves to ameliorate negative affect, and lower distress tolerance than controls. Among women with BED, higher brooding rumination was associated with greater binge eating severity, and stronger expectancies that eating reduces negative affect were associated with more frequent binge eating episodes and greater urge to eat in response to depression. Surprisingly, better distress tolerance was associated with more frequent binge eating episodes. Women with BED consumed more calories and reported greater loss of control as well as a greater sense of guilt in response to the taste task relative to control participants. Contrary to hypothesis, there were no direct or indirect effects of any of the three emotion regulation strategies on change in urge to eat or calories consumed on the taste task following sad mood induction in BED women. In controls, better distress tolerance and stronger expectancies that eating alleviates negative affect were associated with decreased caloric intake on the taste task after mood induction. Overall, these findings highlight the importance of considering trans-diagnostic processes in BED as well as the need to identify other theoretically-relevant factors that contribute to the cognitive and behavioral features of BED. Limitations and directions for future studies are discussed.

Binge Eating Disorder (BED) is characterized by recurrent episodes of binge eating without the use of compensatory behaviors. Functional accounts of BED propose that negative affect is an antecedent to binge eating because binge eating serves to alleviate negative affect. However, previous studies investigating the association between negative affect and binge eating have yielded inconsistent findings, perhaps due to individual vulnerability factors that moderate the effects of negative affect on binge eating behavior. As one candidate, the current study investigated emotion regulation strategies that may be implicated in the maintenance of binge eating in BED, particularly under conditions of negative affect: brooding rumination, distress tolerance, and mood-related expectancies for eating. These emotion regulation strategies were: a) compared in 38 women with BED vs. 36 non-eating disordered female controls, b) examined in relation to markers of current binge eating severity among BED women, and c) used as predictors of caloric intake and urge to eat in response to a personally-relevant dysphoric mood induction upon presentation of snack foods in a "taste task." Results revealed that women with BED endorsed higher brooding rumination, more positive expectancies that eating serves to ameliorate negative affect, and lower distress tolerance than controls. Among women with BED, higher brooding rumination was associated with greater binge eating severity, and stronger expectancies that eating reduces negative affect were associated with more frequent binge eating episodes and greater urge to eat in response to depression. Surprisingly, better distress tolerance was associated with more frequent binge eating episodes. Women with BED consumed more calories and reported greater loss of control as well as a greater sense of guilt in response to the taste task relative to control participants. Contrary to hypothesis, there were no direct or indirect effects of any of the three emotion regulation strategies on change in urge to eat or calories consumed on the taste task following sad mood induction in BED women. In controls, better distress tolerance and stronger expectancies that eating alleviates negative affect were associated with decreased caloric intake on the taste task after mood induction. Overall, these findings highlight the importance of considering trans-diagnostic processes in BED as well as the need to identify other theoretically-relevant factors that contribute to the cognitive and behavioral features of BED. Limitations and directions for future studies are discussed.

Previous research has demonstrated an association between violence exposure and aggression; however, research exploring the association between violence exposure and the forms and functions of aggression is scarce. The aim of this study was to explore the associations between trauma exposure with both reactive and proactive functions of aggression by examining two potential mediators (e.g., psychopathic traits and emotional dysregulation). Participants included 132 male juvenile offenders mandated to treatment in a residential facility (M = 16.78 years old; SD = 1.25). Results indicate emotional dysregulation partially mediated the association between trauma exposure and reactive aggression while controlling for proactive aggression. However, no evidence was found to support the hypothesis that psychopathic traits mediated the association between trauma exposure and proactive aggression. Results suggest trauma exposure is important in the development of reactive aggression. Thus, treatment approaches for aggressive youth should address issues of trauma exposure.

Research shows that parental psychological control is associated with youth aggression in peer relationships. This includes various aggression roles (aggression and victimization), forms (overt and relational), and functions (proactive and reactive). The current study examined the role of two youth individual traits, Machiavellianism and dysregulation, in the association between psychological control and youth aggression. A sample of 142 participants (age M = 15.4, SD = 1.13, 93% male, 82% African-American) were recruited from several juvenile detention facilities in Louisiana. Participants completed a battery of questionnaires, including self-reports of Machiavellianism, dysregulation, aggression, victimization, and parental psychological control. Bootstrap analyses indicated youth Machiavellianism partially mediated the associations between psychological control and the aggression roles, forms, and functions. Youth dysregulation partially mediated the associations between psychological control and the aggression roles and forms. For the aggression functions, dysregulation partially mediated the association between psychological control and reactive aggression, and fully mediated the association between psychological control and proactive aggression. Regression analyses indicated psychological control and dysregulation were more strongly associated with reactive aggression than proactive aggression. Findings demonstrate the importance of the youth individual traits, Machiavellianism and dysregulation, in explaining the association between psychological control and youth aggression problems. These findings have implications for youth interventions, in that these individual traits may be useful targets to help decrease bullying and aggressive behaviors in peer relationships.