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Statistical Analysis Of Censored Groundwater Monitoring DataJanuary 2014 (has links)
acase@tulane.edu
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Utilization of Ferrate as a Disinfection Technology for Wastewater ReuseJanuary 2013 (has links)
acase@tulane.edu
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Effectiveness of lead dust control and genetic susceptibility to lead absorptionHaynes, Erin N. January 2002 (has links)
Dissertation (D.P.H.)--University of Michigan.
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Effectiveness of lead dust control and genetic susceptibility to lead absorptionHaynes, Erin N. January 2002 (has links)
Dissertation (D.P.H.)--University of Michigan.
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ID3, Estrogenic Chemicals, and the Pathogenesis of Tumor-Like Proliferative Vascular LesionsAvecilla, Vincent E 27 October 2017 (has links)
Tumor-like proliferative vascular lesions manifest in several diseases such as peripheral arterial disease (PAD) and atherosclerosis (AS) after arterial injury. The cause of the vascular cell dysfunction in PAD patients is not known. Our recent novel discovery shows that inhibitor of differentiation 3 (ID3) is highly expressed in intimal lesions of clinical vascular disease samples. The central hypothesis of our study is: estrogenic chemical induced dysregulation of ID3 target genes is involved in the development of vascular disease. NHANES data analysis demonstrated higher geometric levels of all 6 PCB congeners in both PAD diagnosed participants and participants at risk of AS when compared to the rest of the population. Adjusted models showed association between higher exposure of PCBs, phthalates, BPA, and increased risk of PAD. Furthermore PCB153 was shown to have the highest geometric mean amongst all PCB congeners in both participants diagnosed with PAD and at risk of AS. Gene expression of ID3 & ID3 candidate targets in blood & tissue studies identified ID3 & ID3 candidate target genes as a driver of vascular disease. Overlapping ID3 & ID3 candidate target genes included: ABCB6, ACP1, BYSL, CAD, CDH15, DCBLD2, DHRS3, DNMT1, ID3, MCM4, and NDUFA7. The ID3 target genes involved in the: focal adhesion pathway were ACTN1, COL1A2, COL3A1, COL6A1, CTNNB1, IBSP, ID3, ITGA8, and MYL2; ECM-receptor interaction were COL1A2, COL3A1, COL6A1, IBSP, ID3, and ITGA8; oxidative phosphorylation pathway ATP5D, ATP5H, ATP6V0B, ATP6V0D1, ATP6V1B2, COX5A, COX7C, COX8A, CYC1, ID3, NDUFA1, NDUFA7, NDUFS4, NDUFV1, NDUFV2; and cell cycle pathway ANAPC10, ATM, CDKN2B, E2F5, MCM3, and MCM4. In summary our results showed an association between exposure to PCBs, phthalates, BPA, and increased risk of PAD and AS, and possible molecular mechanisms of interaction of ID3 target genes and estrogenic chemicals involved in PAD and AS.
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Continuous Monitoring As A Solution To The Large Sample Size Problem In Occupational Exposure AssessmentJanuary 2014 (has links)
acase@tulane.edu
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Analysis and Interpretation of Occupational Exposure Monitoring Data from the Occupational Safety and Health Administration’s Integrated Management Information System (IMIS) and OSHA Information System (OIS), 1979 – 2015Shockey, Taylor Morgan 30 September 2019 (has links)
No description available.
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