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Effect of hyperbaric oxygen on venous PO2, transcutaneous PO2, and VO2max in a normobaric environmentHodges, Alastair N. H. January 2000 (has links)
No description available.
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The effect of cognition on heart rate and the rating of perceived exertion at varied exercise intensities /Bell, James W. January 1982 (has links)
No description available.
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Effect of exercise on early normal-fed and overfed miceTucker, Tammy Joan January 2011 (has links)
Typescript (photocopy). / Digitized by Kansas Correctional Industries
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Cardiopulmonary responses to exercise in the duckKiley, James Patrick. January 1978 (has links)
Call number: LD2668 .T4 1978 K536 / Master of Science
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THE EFFECTS OF ACUTE AEROBIC STRESS ON PREPREGNANT WOMEN OF CHILDBEARING AGE.Olbright, Lucinda Griffith. January 1985 (has links)
No description available.
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The comparison of recovery kinetics of oxygen consumption and heart rate between children and adults: a practicalsuggestion on maximizing the usage of recovery kinetics in clinicalsettingChan, Wing-wai, Stephen., 陳永偉. January 2003 (has links)
published_or_final_version / Sports Science / Master / Master of Science in Sports Science
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The influence of exercise on vitamin B-6 metabolismMunoz, Kathy Denise 29 January 1982 (has links)
The present study was designed to investigate the
effects of exercise on vitamin B-6 metabolism. Four groups
of subjects (intermittent, college, untrained, and high school)
consisting of nineteen male and two female individuals, participated
in the study. The subjects exercised either on a
bicycle ergometer, by running three 1500 meter intervals, or
both. Blood samples drawn prior to exercise (pre), after exercise
(post), and 30 minutes after exercise (30 minute post),
were analyzed for plasma pyridoxal 5'-phosphate (PLP), plasma
B-6 (PB6), glucose, hemoglobin and hematocrit. A 24-hr urine
collection the day before and the day of exercise was analyzed
for urinary B-6 (UB6), 4-pyridoxic acid (4PA), creatinine, and
urea nitrogen.
The dietary intake of the four groups was greater than the
RDA in vitamin B-6, riboflavin, thiamin, niacin, vitamins A and
C, calcium and iron. The B-6/protein ratios of the college and
untrained groups were adequate while the high school group's
ratios were considered inadequate.
The bicycle ergometer had a significant effect on the plasma
PLP levels of the college and untrained groups (P< 0.005) and PB6
levels of the college group following exercise (P< 0.005). The
30 minute post plasma PLP levels were significantly lower for
the college group (P< 0.005).
During the run, the college group had significantly higher
post exercise levels of plasma PLP (P< 0.005) and PBS (P< 0.005}
as compared to the pre exercise sample. The high school group
also had significantly higher levels of plasma PLP following,
exercise for all three runs (P< 0.005, P< 0.025, and P< 0.01,
respectively) as well as higher PB6 levels (P< 0.025, P< 0.01,
and P< 0.025, respectively).
The college athletes had a greater percent change in plasma
PLP (P < 0.01) from the pre to post sample during the run as
compared to the high school athletes.
Urinary B-6 and 4PA were not significantly altered during
either exercise suggesting a shift in PLP and the unphosphorylated
forms of vitamin B-6 from one compartment to another. The significantly
higher levels of plasma PLP and PB6 following exercise
were attributed to an increased utilization of glycogen phosphorylase
in the skeletal muscle with a subsequent release of PLP. / Graduation date: 1982
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Effect of vitamin B-6 supplementation before strenuous exercise on restoration of plasma urea and ammonia levelsCampuzano, Gloria 11 March 1988 (has links)
The objectives of this study were a) to determine
if pyridoxine (PN) supplementation increases the rate at
which plasma urea and ammonia return to basal levels,
following exercise, b) to determine, by open circuit
calorimetry, the utilization of carbohydrates, and c) to
further understand vitamin B-6 metabolism during and
following strenuous exercise. Six male athletes (age 26 ±
5 years and VO₂ max 66.4 ± 6.9 ml/kg/min) exercised for 1
hour on a cycle ergometer at 72% VO₂ max at two points
during a 17 day study. For the first 8 days subjects
received daily a placebo solution, while during the next
half they received a PN dose (20 mg). Subjects consumed
a constant diet the day before, day of, and day after the
exercise test. Blood samples were taken the day of the
exercise test at fasting (Fl), pre-exercise (PE), during exercise (DE), 1 min post exercise (I'P), 6 hour post
exercise (6hP), and the day after the exercise test at
fasting (F2). Plasma was analyzed for ammonia, urea, and
pyridoxal 5'-phosphate (PLP). ANOVA showed no
significant difference between treatments for either
plasma ammonia or urea. While there was a significant
increase (p<0.001) in plasma ammonia levels over time
with the placebo, with supplementation the increase over
time was not significant. With PN supplementation,
plasma PLP levels were significantly correlated (p<0.05)
with plasma ammonia levels at I'P. A slight decrease in
plasma urea concentration was observed with the PN
treatment at PE, DE, I'P, and 6hP. It was concluded
that PN may reduce adverse consequences of plasma
ammonia and urea seen with exercise. On the other hand,
pyridoxine supplementation may produced a shift in the
utilization of substrates of the subjects. Metabolic
rate results showed that the contribution of
carbohydrates as a energy source increased from 43.5 ±
13.7% with the placebo, to 52.0 ± 6.7% with the PN
treatment (not significantly different). This observation
lead to the conclusion that PN supplementation decreases
glycogen stores compared to the glycogen stores without
supplementation. Since the findings from this study
suggest slightly more rapid plasma ammonia and urea
restoration but decreased glycogen stores, they do not
provide evidence for or against an increased need for vitamin B-6 in persons that are involved in strenuous
exercises of medium duration. / Graduation date: 1988
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THE EFFECT OF AN ACUTE BOUT OF EXERCISE ON SELECTED PULMONARY FUNCTION MEASUREMENTS.BUONO, MICHAEL JOSEPH. January 1982 (has links)
A series of five studies were conducted to examine the effect of exercise on selected pulmonary function measurements. Studies I and II determined the effect of an acute bout of exercise on various lung volumes immediately post-exercise and over a 24-hour post-exercise period. There were significant mean increases of 210 ml (20.6%) and 260 ml (20.8%) in the 5-minute post-exercise residual volume (RV) measurement for studies I and II, respectively. There also were significant mean increases of 170 ml (3.4%) and 190 ml (2.7%) in the 5-minute post-exercise total lung capacity (TLC) for studies I and II, respectively, while vital capacity (VC) remained unchanged. RV and TLC remained significantly increased over the pre-exercise values through 30 and 15 minutes of recovery, respectively. Studies III through V were undertaken to determine the physiological mechanism underlying the responses reported in studies I and II. In study III, transthoracic electrical impedance (TEI) was significantly decreased below the pre-exercise value through 30-minutes of recovery, indicating that there was an increase in thoracic fluid volume following exercise. However, TEI measurements alone cannot separate between intra- and extravascular fluid shifts. Therefore, studies IV and V attempted to identify whether the decrease in TEI and increase in RV reported in study III were due to intra- or extravascular fluid shifts. Study IV examined the TEI, RV, and TLC responses before and following exercise, as central blood volume (CBV) was experimentally increased via G-suit inflation, and decreased via venous occlusion tourniquets. The results suggest that RV is relatively insensitive to intravascular volume shifts within the thorax. Study V determined and followed the effect of an acute bout of exercise on lung diffusion capacity (D(,Lco)). D(,Lco)/V(,A) did not increase significantly following exercise, suggesting that the decrease in TEI following exercise is the result of extravascular fluid accumulation. It was concluded that a sub-clinical pulmonary edema occurs following exercise. A logical sequence of events based on the results of studies I through V was proposed as a possible explanation for the responses of RV and TLC following exercise.
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SERUM FREE FATTY ACID CONCENTRATION DURING POST-EXERCISE RECOVERY (INSULIN, HUNGER).MAXWELL, BESS DEVERE. January 1985 (has links)
In order to achieve a better understanding of the impact of exercise on the concentration of serum free fatty acids (FFA) during post-exercise recovery, the purposes of this study were: (1) to determine the relationships between exercise intensity, total exercise energy expenditure, and the concentration of serum FFA during post-exercise recovery; (2) to examine the effects of exoge- nous glucose on post-exercise serum FFA and hormones controlling the FFA response; and (3) to examine the impact of acute exercise on hunger. Untrained, 12-h fasted, college-age males performed cycle ergometer exercise at exercise intensities ranging from 29 to 59% peak ‘VO₂ for total energy expenditures ranging from 162 to 320 kcal. Blood samples, hunger ratings, and metabolic indices were collected or measured before, during, and for 3 h post-exercise. In response to exercise of approximately 300 kcal, FFA was elevated for 3 h post-exercise. The FFA response was a function of total exercise energy expenditure, rather than exercise intensity, or combined effects of these factors. The response was associated with low insulin concentration but no changes were observed in blood glucose, glucagon, growth hormone, or cortisol. Glucose ingestion and infusion studies demonstrated that possible mechanisms con- tributing to the post-exercise FFA response included decreases in FFA re-esterification, increases in triglyceride hydrolysis, and decreases in sympathetic input to adipose tissue. Exercise caused a suppression of hunger for 2 h post-exercise which was a function of the combined effects of exercise intensity and total energy expenditure. An increase in core temperature may have contributed to the anorexigenic effect of exercise. In conclusion, exercise, performed in and followed by a period of fasting caused an elevation of FFA for 3 h during post-exercise recovery. The post-exercise recovery period should be considered an important phase in the physiological impact of exercise on the storage and utilization of fat.
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