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Effects of cyclophosphamide exposure of male rats on progeny outcome and site of action on male germ cell nuclei in the testis and epididymisQiu, Jianping, 1953- January 1994 (has links)
No description available.
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Association between epithelial-cadherin and rat embryo malformations during organogenesis in vitroChen, Beiyun January 1994 (has links)
No description available.
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Foetal toxicity of methyl isocyanate metabolitesGuest, Ian January 1993 (has links)
No description available.
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Investigation into genetically programmed responses to cadmium and mercury in HeLa cells by differential display and two-dimensional gel electrophoresisCharoensri, Nicha January 2004 (has links)
No description available.
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Endocrine disruption in the female following «in utero» exposure to the plasticizer di(2-ethylhexyl) phthalateMeltzer, Deborah January 2014 (has links)
No description available.
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Understanding the effects of exposure to an environmentally relevant mixture of brominated flame retardant congeners on the function and development of the male gonadMa, Yiqian January 2013 (has links)
No description available.
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Immunotoxic effects of orally administered sodium tungstate in wild-type and pre-leukemic mouse modelsKelly, Alexander January 2013 (has links)
No description available.
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Influence of embryonic exposure to hexabromocyclododecane (HBCD) on the corticosterone response and "fight or flight" behaviours of captive American kestrelsKobiliris, Demetrios January 2010 (has links)
No description available.
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Enhanced cadmium-induced testicular necrosis and renal proximal tubular damage caused by gene-dose increase in a <em>Slc39a8</em>-transgenic mouse lineWANG, BIN 03 April 2007 (has links)
No description available.
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Interactions of pentavalent and trivalent arsenic with intracellular thiolsWinski, Shannon Lee, 1967- January 1991 (has links)
Trivalent arsenic (As(III)) exposure is linked to cancer, but exposure includes pentavalent arsenic (As(V)). These forms interconvert in biological systems, and the mechanism is not understood. Arsenic has a high affinity for sulfhydryls, and the interaction of As(III) and As(V) with biological sulfhydryls and cellular uptake was investigated in this study. Incubation of rat blood at 1 mM arsenic showed As(V) uptake, membrane and protein binding to be time dependant. These processes were almost immediate with As(III) incubation. Incubation at 25 mM As(V) resulted in 40% thiol depletion with no oxidized glutathione formation. Metabolite analysis showed half of the As(V) converted to As(III). In As(III) incubation, 27% thiol depletion occurred with 300% increase in GSSG. As(V) reduction combined with thiol depletion could indicate that arsenate used sulfhydryls as reducing equivalents. Because arsenate uptake was slow this reduction may not be a main contributor to the overall in vivo process.
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