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  • About
  • The Global ETD Search service is a free service for researchers to find electronic theses and dissertations. This service is provided by the Networked Digital Library of Theses and Dissertations.
    Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.

Global kidney disease

Herrera Añazco, Percy, Mezones Holguín, Edward, Hernández, Adrian V. 03 July 2014 (has links)
We read with interest the Lancet Series on Global Kidney Disease. Valerie Luyckx and colleagues describe the economics and medical management of chronic kidney disease in sub-Saharan Africa.1 We note clear similarities with patients in Peru. Indeed, in Peru, the Ministry of Health (MINSA)—which covers 70% of the population—does not have a comprehensive programme for the management of patients with chronic kidney disease, including renal replacement therapies. However, the Social Security System (Essalud)—which covers 20% of the population—has a chronic kidney disease programme. / Revisión por pares

Novel cancer therapeutics, the generation of ROS, and cell survival /

Mitchell, Clint, January 2006 (has links)
Thesis (Ph. D.)--Virginia Commonwealth University, 2006. / Prepared for: Dept. of Biomedical Engineering. Bibliography: leaves 202-220. Also available online.

Continuous Percutaneous Cyst Drainage for Multicystic Kidney

ITO, TAKAHIRO, ANDO, HISAMI, KANEKO, KENITIRO 25 November 1993 (has links)
No description available.

Glomerulosclerosis in the Col1a2-deficient mouse model homotrimer pathogenesis and MMP expression /

Roberts-Pilgrim, Anna M., Phillips, Charlotte L. January 2009 (has links)
Title from PDF of title page (University of Missouri--Columbia, viewed on Feb. 20, 2010). The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Dissertation advisor: Charlotte L. Phillips. Vita. Includes bibliographical references.


Barfuss, Delon Willis, 1942- January 1975 (has links)
No description available.

Adsorption of urea from the liquid phase by activated carbon

Shintre, Sudhir Gopal, 1943- January 1971 (has links)
No description available.

Isoenzymatic characterization of cell cultures of bush bean (Phaseolus vulgaris cv. Contender)

Arnison, Paul Grenville. January 1975 (has links)
No description available.

Antigenic components of glomerular basement membrane

Huang, Flora. January 1967 (has links)
No description available.

Role of hyperhomocysteinemia in the regulation of oxidative stress and inflammatory responses in the kidney: protective effect of folic acid supplementation

Hwang, Sun-Young January 2011 (has links)
Hyperhomocysteinemia, a condition of elevated blood homocysteine (Hcy) level, is an independent risk factor for cardiovascular disease. Folic acid supplementation can effectively reduce blood Hcy levels. Recent studies have demonstrated that hyperhomocysteinemia is also associated with kidney disease. However, the underlying mechanisms remain unclear. The overall objective of the study was to investigate the biochemical and molecular mechanisms of Hcy-induced kidney injury and the effect of folic acid supplementation on Hcy-induced kidney injury. Hyperhomocysteinemia was induced in Sprague-Dawley rats by feeding a high-methionine diet for 12 weeks. An elevation of serum total Hcy level was observed in hyperhomocysteinemic rats. Hyperhomocysteinemia-induced superoxide anion production via nicotinamide adenine dinucleotide phosphate (NADPH) oxidase activation resulted in oxidative stress in the kidney. Reduction of oxidative stress by inhibiting superoxide anion production effectively ameliorated hyperhomocysteinemia-induced kidney injury. Inflammatory responses such as increased chemokine expression have been implicated as one of the mechanisms of kidney disease. Monocyte chemoattractant protein-1 (MCP-1) is a potent chemokine that is involved in the inflammatory response in kidney disease. Nuclear factor-kappa B (NF-kappaB) plays an important role in upregulation of MCP-1 expression. We investigated the effect of hyperhomocysteinemia on MCP-1 expression and the molecular mechanism responsible for such an effect in rat kidneys as well as in human kidney proximal tubular cells.

Morphological chemical and functional correlates in the renal glomerulus in experimental nephritis

Gang, Nicholas Frank. January 1969 (has links)
No description available.

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