141 |
Studies on human red cell cholinesterase in relation to muscle disease.Robinson, Joseph Desmond, January 1900 (has links)
Thesis (M. Phil.)--University of Hong Kong, 1978. / Xeorx copy of typescript.
|
142 |
Analysis of potential muscular determinants of the preferred walk-run transition speed in human gaitSasaki, Kotaro 28 August 2008 (has links)
Not available / text
|
143 |
REACTIVE HYPEREMIA IN RED AND WHITE MUSCLE OF THE CHICKEN IN RESPONSE TO BLOOD FLOW CESSATION OF VARYING DURATIONKlabunde, Richard Edwin, 1948- January 1975 (has links)
No description available.
|
144 |
NEUROTROPHIC CONTROL OF MAMMALIAN SKELETAL MUSCLE: THE ROLE OF SUBMECHANICAL THRESHOLD BIOELECTRIC ACTIVITYBaumbach, Neal James, 1949- January 1976 (has links)
No description available.
|
145 |
Chemical composition of bovine muscles as influenced by sexVavra, Martin January 1969 (has links)
No description available.
|
146 |
Effect of Naproxen on delayed onset muscle sorenessLecomte, Jacqueline January 1995 (has links)
The purpose was to determine the effect of Naproxen in attenuating the symptoms (muscle soreness level) and signs (plasma CK activity and muscular strength decrement) of delayed onset muscle soreness (DOMS). Twenty subjects were randomly assigned Naproxen (500 mg BID) or placebo in a double-blind, crossover design. Two testing phases, each 8 days in duration, were separated by a washout period of 7 days. Eccentric single-leg exercises were performed on Days 1, 3 and 4 to induce DOMS in the quadriceps muscles. Perception of muscle soreness, plasma CK, and knee extensor torque were evaluated throughout each phase. Following the eccentric exercise, plasma CK levels were similarly elevated in both Naproxen and placebo conditions. After DOMS had developed, Naproxen reduced the perception of soreness on Day 3 when muscle soreness was highest. Following treatments with Naproxen, peak quadriceps torque during leg extension at 60$ sp circ$/s was higher compared to placebo, however at higher velocity (180 and 300$ sp circ$/s) peak muscle torques were similar. The data indicate that therapeutic doses of Naproxen do not prevent CK release into the plasma but decreases the perception of muscle soreness and positively influences quadriceps peak torque.
|
147 |
The purification of substances which control muscle parasitism in experimental trichinellosis.Essien, Ebong Udofia. January 1975 (has links)
No description available.
|
148 |
Studies on water-soluble proteins of bovine skeletal muscle.Randall, C. J. January 1965 (has links)
Proteins comprise approximately 80 per cent of the solid matter of muscle tissue and in the living animal are called upon to perform many varied functions. Proteins are present in muscle tissue which are capable of rapid contraction and relaxation, together with a complex system of enzymes to supply these elements with energy, a surrounding network of connective tissue to bind them into place and to bear the stress of their movements, and an intricate blood supply and nervous system. [...]
|
149 |
An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegansViveiros, Ryan 05 1900 (has links)
During C. elegans embryogenesis, myoblasts initially form two rows along the left and right lateral midlines and at ~290 min of development migrate dorsally and ventrally to form the four muscle quadrants present upon hatching (Sulston et al, 1983). As the myoblasts migrate they are still dividing, as are many other cells in their immediate environment. This means the cell-cell contact of cells during migration is dynamic and can vary from animal to animal (Schnabel et al, 1997). This situation creates an environment where the extracellular matrix (ECM) and cell surface contacts are in constant flux, which begs the questions as to how these cells navigate unerringly to their final destination.
In an attempt to identify genes mediating these migrations, I performed an RNAi based screen targeting 776 genes predicted to be members of the extracellular matrix (ECM), or one of its receptors. Using both feeding and injection based RNAi, I was able to identify three genes of interest. Knockdowns of F56B3.2 resulted in paralyzed animals with detached muscle, making it a good candidate for a new component of the muscle attachment complex. F33G12.4 knockdowns resulted in an embryonic arrest phenotype with an abnormal muscle lineage, possibly stemming from polarity defects. The only knockdown that resulted in muscle migration defects was that for lam-2, which encodes for the laminin gamma subunit. Analysis of the lam-2 knockdown, as well as knockdowns for the other laminin subunits, revealed dorsal/ventral migration defects as well as a posterior displacement of the anterior-most ventral muscle cells. Investigation of this posterior displacement has led to the identification of a previously un-described anterior muscle migration event and its dependency upon the extension of muscle processes from the leading cells.
|
150 |
Mental influence on physiological cost for a restricted muscle group of the right forearmJohnson, Larry Ray 08 1900 (has links)
No description available.
|
Page generated in 0.0234 seconds