Spelling suggestions: "subject:"neuromuscular transmission"" "subject:"euromuscular transmission""
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Cellular and molecular analysis of motor neuron development in the zebrafish hindbrainBingham, Stephanie, January 2003 (has links)
Thesis (Ph. D.)--University of Missouri-Columbia, 2003. / Typescript. Vita. Includes bibliographical references (leaves 234-254). Also available on the Internet.
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Role of transmembrane protein strabismus in motor neuron migration in the zebrafish hindbrainSittaramane, Vinoth. Chandrasekhar, Anand, January 2008 (has links)
Title from PDF of title page (University of Missouri--Columbia, viewed on Feb 25, 2010). The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Dissertation advisor: Dr. Anand Chandrasekhar. Vita. Includes bibliographical references.
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A model of the neuronal encoding mechanism for studying neural systems.Varano, Vincent. January 1973 (has links)
No description available.
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Parallel processing of nociceptive information evidence for multiple reflex and ascending nociceptive pathways /Kalliomäki, Jarkko. January 1992 (has links)
Thesis (doctoral)--Lund University, 1992. / Added t.p. with thesis statement inserted.
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The influence of the gamma motor system on stance and volitional movement in normal manShambes, Georgia Marie, January 1968 (has links)
Thesis (Ph. D.)--University of Wisconsin--Madison, 1968. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.
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Parallel processing of nociceptive information evidence for multiple reflex and ascending nociceptive pathways /Kalliomäki, Jarkko. January 1992 (has links)
Thesis (doctoral)--Lund University, 1992. / Added t.p. with thesis statement inserted.
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Molecular changes following genetic and physical disruption of neuromuscular synapses in developing and adult mice /Caudron, Audrey. January 2006 (has links) (PDF)
Thesis (M.Phil.) - University of Queensland, 2006. / Includes bibliography.
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Effects of volume and exercise complexity on neural adaptations, strength gains and lean body mass in untrained adultsBlaak, John B. January 2002 (has links)
Thesis (M.S.)--University of Wisconsin--La Crosse, 2002. / Includes bibliographical references.
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Studies on the action of selected venom components on the frog neuromuscular systemSantana De Sa, Sonia January 1975 (has links)
No description available.
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The effects of anticholinergic drugs on sea anemonesShortridge, Kenneth Robert 01 January 1959 (has links)
The first proof of the chemical mediation of nerve impulses by the peripheral release of specific chemicals was made by Loewi (1921). He demonstrated that during stimulation of the vagus nerve leading to an excised frog’s heart (Figure 1) a chemical mediator that he called Vagus-substance was released which slowed the action of the heart. This chemical mediator entered a perfusion fluid consisting of isotonic sodium chloride which was pumped out of the ventricle of the heart and diverted so as to drip onto a second frog's heart. The dripping perfusion medium slowed the pulsation of the second heart upon stimulation of the donor heart. When the stimulus was removed, the donor heart returned to normal pulsation and shortly after the recipient heart returned to normal. The Vagus-substance in the perfusion fluid was identified by Feldberg and Krayer (1933) as acetylcholine.
Cholineacetylase forms acetylcholine from acetic acid and choline at the neuromuscular junction (Figure 2) in the presence of energy. The energy is obtained as a result of the action potential produced by the nerve impulse traveling along the nerve to the neuromuscular junction.
The acetylcholine proceeds to cross the synapse at the neuromuscular junction and activates the muscle causing its contraction. The persistent presence of the acetylcholine at the junction would result in constant depolarization of the muscle resulting in fibrillation. The acetylcholine is destroyed by acetylcholinesterase causing repolarization so that the muscle is able to respond to further nerve impulses.
Acetylcholine possesses two actions in vertebrates: muscarinic and nicotinic. The muscarinic action is exhibited at the neuromuscular functions of smooth muscle and sweat glands and the nicotine action is exhibited at the neuromuscular junction of striated muscle and at the synapse within ganglia.
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