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Cobalt(III) and nickel(II) complexes of tris(3-dimethylarsinopropyl)arsine and tris(3-dimenthylarsinopropyl) phosphine /Benner, Gereld Stokes January 1966 (has links)
No description available.
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Mechanism of reactions of nickel (II) complexes of mercaptoamines with alkyl halide /Blinn, Elliott Lawrence January 1967 (has links)
No description available.
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The ionic and electronic defect structure of pure and chromium-doped nickel oxide /Meier, Gerald Herbert January 1968 (has links)
No description available.
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An equilibrium and reaction study of the glycylglycinate - glyoxalate - nickel(II) and zinc(II) systems.Gosser, Leo Anthony January 1969 (has links)
No description available.
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Diffusion of cobalt, nickel, and iron in cobalt oxide and nickel oxide /Crow, Wesley Blanke January 1969 (has links)
No description available.
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The oxidation mechanism of the Ni-Cr-Al alloys /Kosak, Robert January 1969 (has links)
No description available.
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The nickel(II) and cobalt(III) complexes of linear tetradentate ligands /Hung, Ellen Hsia January 1970 (has links)
No description available.
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Some aspects of the physical metallurgy and weldability of 10 nickel modified steel /Snide, James Amos January 1975 (has links)
No description available.
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Development of an in vitro Method to Determine Nickel Toxicity and CarcinogenicityRenaud, Matthew 14 May 2012 (has links)
Nickel is a widely used metal in industrial and commercial applications, being both workable and highly resistant to corrosion. Certain nickel compounds are known human carcinogens, but not all nickel compounds are equally hazardous. A robust method of assessing the carcinogenic potential of various nickel compounds is needed in order to determine safe occupational exposure levels. This work attempts to develop an in vitro mammalian cell culture method for assessing the carcinogenic and toxic potential of nickel compounds by metabolic and cell cycle analysis. Two cell lines, C3H/10T1/2 and MRC-5, were used. Measurements of extracellular metabolites by enzymatic methods and HPLC were combined with cell counts and cell cycle and apoptosis analysis by flow cytometry. This work shows that nickel sulphate can elicit a metabolic response similar to that of organic carcinogens, though the underlying mechanisms are likely different. / Vale Canada Limited, NSERC
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Sample size effects related to nickel, titanium and nickel-titanium at the micron size scaleNorfleet, David Matthew, January 2007 (has links)
Thesis (Ph. D.)--Ohio State University, 2007. / Title from first page of PDF file. Includes bibliographical references (p. 162-169).
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