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Altered gene expression in the human airways during oxidative stress /Dandrea, Tiziana, January 2003 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2003. / Härtill 4 uppsatser.
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Airway responses to NO₂ and allergen in asthmatics /Barck, Charlotte, January 2005 (has links)
Diss. (sammanfattning) Stockholm : Karolinska institutet, 2005. / Härtill 4 uppsatser.
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An experimental investigation of the conversion of NO to NO₂ in a simulated gas turbine environment /Hunderup, James W., January 1993 (has links)
Thesis (M.S.)--Virginia Polytechnic Institute and State University, 1993. / Vita. Abstract. Includes bibliographical references (leaves 121-123). Also available via the Internet.
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Quantum chaos of the NO2 molecule in high magnetic fieldsNygård, Jesper. January 1900 (has links) (PDF)
Thesis (M.S.)--Københavns universitet, 1996. / Title from title screen (viewed on July 9, 2008). Title from document title page. Includes bibliographical references. Available in PDF format via the World Wide Web.
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The kinetics of some reactions of nitroso-compoundsVoisey, M. A. January 1965 (has links)
No description available.
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An investigation of the influence of sound on flow stability, flame noise, and nitrogen oxide levels in natural gas flames /Knezek, Robert Alois January 1974 (has links)
No description available.
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Laminar head-on flame quenching in a spherical combustion bombSellnau, Mark Charles. January 1981 (has links)
Thesis: M.S., Massachusetts Institute of Technology, Department of Mechanical Engineering, 1981 / Includes bibliographical references. / by Mark Charles Sellnau. / M.S. / M.S. Massachusetts Institute of Technology, Department of Mechanical Engineering
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Colorimetric reagents for nitrogen dioxideTrump, Eric Laurence. January 1984 (has links)
Call number: LD2668 .T4 1984 T78 / Master of Science
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Assessment of air pollution in residential areas : a case study of Kinondoni Municipality, TanzaniaNdambuki, J.M., Rwanga, S. January 2008 (has links)
Published Article / Air pollution, just like any other type of pollution, produces harmful effects to man and his environment. In spite of knowing this, many less industrialised countries of the world have no air quality monitoring strategies in place. Consequently, documented evidence of air pollution studies in such countries is scarce. This is the case in Tanzania. This scenario is made worse by lack of scientific tools which could aid in identifying air pollution-prone areas with a view to aiding town planners in locating safe sites for schools, hospitals and residential areas as well as parks. In this paper, we present results of a study carried out in the city of Dar-es-Salaam, Tanzania on air pollution in residential areas. Results show significant air pollution in the studied areas. Moreover, both adults and children are at risk due to lead pollution in the air.
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Acute cardiovascular effects of exposure to air pollution : components, vascular mechanisms and protecting the publicLangrish, Jeremy Patrick January 2012 (has links)
Exposure to air pollution, particularly fine and ultrafine particulate matter derived from combustion sources, has been consistently associated with increased cardiovascular morbidity and mortality. Recent controlled exposure studies demonstrate that short-term exposure to diesel exhaust, which can contribute up to 40% of urban particulate air pollution, results in impaired vascular endothelial and fibrinolytic function in healthy volunteers, and increased exercise-induced myocardial ischaemia in patients with coronary heart disease. These observations may, in part, explain the observed increase in cardiovascular events following exposure to air pollution. Despite these observations there remain uncertainties regarding the key constituents of the air pollution mixture that mediate these adverse effects, and the underlying physiological and biological pathways involved. In these studies, using two controlled exposure facilities, I explored the vascular effects of the most prevalent gaseous component of the air pollution mixture – nitrogen dioxide – and the mechanisms responsible for impaired vasomotor function following exposure to diesel exhaust. Furthermore, I investigated the effect of acute exposure to “real-world” urban air pollution in both healthy volunteers and patients with coronary heart disease, and the effect of reducing that exposure using a simple facemask. In total, 10 healthy volunteers were exposed to nitrogen dioxide, and 29 healthy volunteers exposed to dilute diesel exhaust in a series of doubleblind randomised crossover studies. Exposure to nitrogen dioxide had no effect on either vasomotor function or endogenous fibrinolysis, providing indirect evidence that the adverse vascular effects are predominantly driven by particulate components. Following exposure to diesel exhaust there was no up regulation of endothelin-1 production, although there was increased vasoconstriction to intra-arterial infusion of endothelin-1. Following endothelin A receptor antagonism, there was attenuated vasodilatation following exposure to diesel exhaust as compared to air, an effect abrogated by endothelin B receptor antagonism. My findings suggest that the endothelin system does not play a central role in the adverse vascular effects of air pollution, but given the tonic interaction between the endothelin and nitric oxide systems, these observations could be explained by reduced nitric oxide bioavailability. Following diesel exhaust inhalation, plasma nitrite concentrations (as a marker for nitric oxide generation) are markedly increased without changes in haemodynamics or basal blood flow consistent with increased nitric oxide consumption. In the presence of a nitric oxide clamp, and without endogenous nitric oxide release, the vascular responses to vasodilators are similar. This perturbation of nitric oxide consumption and release appears to underlie the observed vascular endothelial effects. Fifteen healthy volunteers and 98 patients with coronary artery disease were recruited in Beijing, China. Subjects walked along a predefined city centre route for 2 hours in the presence and absence of a highly efficient facemask to reduce personal particulate air pollution exposure in an open label randomised crossover study. When wearing a facemask, there was an attenuation of exercise-induced increases in blood pressure, an improvement in heart rate variability, reduced myocardial ischaemia and subjects reported fewer symptoms. My findings have identified the biological mechanisms underlying the adverse vascular effects of exposure to diesel exhaust, and have helped to clarify the components responsible for these effects. Moreover, I have identified important benefits of reducing personal exposure to particulate matter using a simple facemask that have the potential to reduce cardiovascular events in patients living in urban or industrialised areas. Ongoing research in this area will provide further insight into the underlying vascular mechanisms, and the potential benefits of reducing particulate air pollution exposure, and may result in important targeted interventions to reduce the impact of air pollution on cardiovascular health.
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