- About
-
The Global ETD Search service is a free service for researchers
to find electronic theses and dissertations. This service is
provided by the
Networked Digital Library of Theses and Dissertations.
Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
Search results
Showing 291 to 300 of 13415 (0.03 seconds)| 291 |
Functional Characterization of the Electrophoretic Hemoglobin Polymorphism in the Atlantic Croaker (Micropogon undulatus)Shelly, Daniel Allen 01 January 1994 (has links) (PDF)
No description available.
|
| 292 |
Evaluating the Potential for Adaptive Response to Mercury in Captive-Dosed Zebra FinchesBuck, Kenton August 01 January 2013 (has links) (PDF)
No description available.
|
| 293 |
Salt and water balance in the oligohaline clam, Rangia cuneataHenry, Raymond Peter 01 January 1978 (has links) (PDF)
No description available.
|
| 294 |
Form and function: Mechanistic insights on the myelination and connections of axonsOprea, Laurentiu January 2023 (has links)
No description available.
|
| 295 |
Cell-specific CFTR expression, function and protein-protein interactions in human airwaysSato, Yukiko January 2022 (has links)
No description available.
|
| 296 |
The role of retinal feature detectors in visually guided prey captureTheriault, Catherine January 2023 (has links)
No description available.
|
| 297 |
The effect of high salt intake on RhoA-mDia1 signaling and cytoskeleton in vasopressin neuronsLi, Banruo January 2023 (has links)
No description available.
|
| 298 |
Characterizing and targeting fibrosis in a model of Lama2-related muscular dystrophyAccorsi, Anthony Andrew 06 June 2017 (has links)
Laminin-deficient congenital muscular dystrophy 1A (MDC1A) is the second
most prevalent congenital muscular dystrophy (CMD) and is due to a defect in the alpha
chain of the basement membrane protein laminin-211. This protein serves as the vital link
between the muscle cell membrane (sarcolemma) and the extracellular matrix (ECM) via
interactions with integrins and a-dystroglycan. Loss of laminin results in impaired
myofiber anchoring, structural instability, and a multitude of dysregulated signaling
pathways leading to many devastating secondary pathologies. Much of the work to date
has focused on studying pathology at the end-stages of disease in mouse models.
However, because this is a congenital disease that presents at/soon after birth, the most
relevant time periods of study are those that most closely reflect human disease, early
development. This thesis will characterize dysregulated pathways throughout
development and into end-stage pathology as well as elucidate amelioration of these
pathways due to intervention and applicable non-invasive biomeasures to track
therapeutic progress. Because this work focuses on secondary manifestations of
pathology, the work outlined in this thesis has the potential to be applied to an array of
neurodegenerative diseases that are currently without any form of treatment.
|
| 299 |
The novel functions of the SARS CoV-2 accessory protein ORF8McLean, Myles January 2022 (has links)
No description available.
|
| 300 |
Influence of the circadian rhythm on CD8+ T cell proliferation: A mathematical modeling perspectiveBalit, Nasri January 2022 (has links)
No description available.
|
Page generated in 0.0631 seconds