Thesis (M.D.)--University of Melbourne, 1986. / Spine title: Risk factors in traumatic spinal cord paralysis. "An analysis of 124 consecutive admissions to the Victorian Spinal Injuries Unit, Austin Hospital from 1st March, 1983 to 28th December, 1984." Typescript (photocopy). Includes bibliographical references (v. 4, leaves 745-754).

Thèse--Université de Paris. / "Bibliographie": p. [93]-96.

No description available.

Sleep Paralysis was written to be performed by a chamber orchestra of 17 performers total. The piece runs between 11 and 12 minutes and has a distinct programmatic approach. This piece attempts to convey the emotions and feelings one might experience when he or she is stricken with sleep paralysis—a condition where one wakes up during sleep but cannot move. Thus, this piece is set up as a dream gone badly. Harmonic and rhythmic tension increase throughout the piece until it reaches a moment of “paralysis” where the harmonies are static and melodies and themes appear in a dream-like fashion. Eventually, the ever-mounting tension resolves to free the listener of the paralysis; however, one should not assume that the paralysis is gone forever. / text

Call number: LD2668 .T4 1978 P38 / Master of Science

Abstract not available

Thesis (Ph.D.)--University of Delaware, 2006. / Principal faculty advisor: Stuart A. Binder-Macleod, Dept. of Physical Therapy. Includes bibliographical references.

Hyperkalemic periodic paralysis (HyperKPP) is an autosomal dominant human skeletal muscle channelopathy that causes periods of myotonic discharge and periodic paralysis due to defective Nav1.4 sodium channels. Patients are asymptomatic at birth, attacks become short and frequent during childhood, and more severe during adolescence. Since the Nav1.4 content in the cell membrane is relatively constant during childhood, it was hypothesized that some symptoms start with the defective Nav1.4 channels, while other symptoms start after some changes occur in gene expression affecting other membrane channel content and/or activity. To test the hypothesis, the contractile characteristics of EDL and soleus muscles from HyperKPP mice from the age of 0.5 to 12 months were tested in vitro. For both EDL and soleus, contractile defects, including low force generation, instability and large unstimulated force were observed by two weeks of age. With aging, the defects did not worsen, but muscles actually showed some improvement. Considering that Nav1.4 protein content reaches maximum at three weeks of age, the data suggests that HyperKPP symptoms are solely due to the defective Nav1.4 channels.

Hyperkalemic periodic paralysis (HyperKPP) is an autosomal dominant human skeletal muscle channelopathy that causes periods of myotonic discharge and periodic paralysis due to defective Nav1.4 sodium channels. Patients are asymptomatic at birth, attacks become short and frequent during childhood, and more severe during adolescence. Since the Nav1.4 content in the cell membrane is relatively constant during childhood, it was hypothesized that some symptoms start with the defective Nav1.4 channels, while other symptoms start after some changes occur in gene expression affecting other membrane channel content and/or activity. To test the hypothesis, the contractile characteristics of EDL and soleus muscles from HyperKPP mice from the age of 0.5 to 12 months were tested in vitro. For both EDL and soleus, contractile defects, including low force generation, instability and large unstimulated force were observed by two weeks of age. With aging, the defects did not worsen, but muscles actually showed some improvement. Considering that Nav1.4 protein content reaches maximum at three weeks of age, the data suggests that HyperKPP symptoms are solely due to the defective Nav1.4 channels.

The subject ot the research: children sick with cerebral paralysis. The problem of the research: hypotherapy is a new and hot theme, because this kind of treatment is rather obscure and rarely applied in treating children with cerebral paralysis in Lithuania. The aim of the research: to determine the influence of hypotherapy on children sick with cerebral paralysis. Tasks of the paper: 1. To evaluate the movements of sick children according to GMFM test before hypotherapy, during it and after it. 2. To determine the possibilities of the accomplishment of exercises both on a standing horse and walking at foot‘s pace of children sick with cerebral paralysis . 3. To evaluate the achievements of children sick with cerebral paralysis ( riders) before and after hypotherapy. 4. To make a survey among parents having children sick with cerebral paralysis. The hypothesis of the research: the application of hypotherapy should improve the motion functions and the children‘s emotional state. The methods of the research: the analysis of scientific literature, interview, testing, observation, statistical analysis of the research data. The results of the research: comparing the initial Gross Motor Function Measure – GMFM (Ulrich, 1993 ) testing results to the final ( after hypotherapy ) ones, the improvement in children‘s health is obvious. The achievements of most children sick with cerebral paralysis ( riders ) before hypotherapy were poor, and satisfactory or good after it. Summing up... [to full text]