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Pharmacology of marine toxins and cholinergic mechanisms.Turner, Raymond Jeffry. January 1971 (has links) (PDF)
Thesis (M.Sc.) -- University of Adelaide, Department of Human Physiology and Pharmacology, 1971.
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Effects of cortical stimulants and cholinolytic agents on spontaneous and evoked potentialsA'Hearn, Maxine Clara. January 1966 (has links)
Thesis (Ph. D.)--University of Wisconsin, 1966. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.
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Synthesis of potential anticholinergic dioxolanes.Warner, Stephen Cooley January 1981 (has links)
No description available.
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Cholinergic modification of DRL performance in normal rats and rats with lesions of the septum /Thatcher, Karen Margret January 1975 (has links)
No description available.
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Pharmacological control of transient lower oesophageal sphincter relaxations /Lidums, Ilmars. January 1999 (has links) (PDF)
Thesis (Ph.D.) -- University of Adelaide, Dept. of Medicine, 1999. / Bibliography: leaves 181-233.
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Effects of some centrally acting cholinolytic drugs on choice and conflict behaviour in ratsPoel, Augustinus Maria van der, January 1973 (has links)
Thesis--Leyden. / Summary in Dutch. Includes bibliographical references.
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Effects of some centrally acting cholinolytic drugs on choice and conflict behaviour in ratsPoel, Augustinus Maria van der, January 1973 (has links)
Thesis--Leyden. / Summary in Dutch. Includes bibliographical references.
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Effect of cevimeline on oral health and quality of life in Sjögren's syndrome patientsLeung, Chiu-man, Katherine. January 2006 (has links)
Thesis (Ph. D.)--University of Hong Kong, 2007. / Title proper from title frame. Also available in printed format.
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Pharmacological control of transient lower oesophageal sphincter relaxations / Ilmars Lidums.Lidums, Ilmars January 1999 (has links)
Bibliography: leaves 181-233. / 233 p. : ill. ; 30 cm. / Title page, contents and abstract only. The complete thesis in print form is available from the University Library. / Investigates pharmacological control of transient lower oesophagal sphincter relaxations as a treatment of gastro-oesophageal reflux. Two major classes of pharmaceutical agents were explored; anticholinergic agents and the GABAb agonist, baclofen. / Thesis (Ph.D.)--University of Adelaide, Dept. of Medicine, 1999
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The effects of anticholinergic drugs on sea anemonesShortridge, Kenneth Robert 01 January 1959 (has links)
The first proof of the chemical mediation of nerve impulses by the peripheral release of specific chemicals was made by Loewi (1921). He demonstrated that during stimulation of the vagus nerve leading to an excised frog’s heart (Figure 1) a chemical mediator that he called Vagus-substance was released which slowed the action of the heart. This chemical mediator entered a perfusion fluid consisting of isotonic sodium chloride which was pumped out of the ventricle of the heart and diverted so as to drip onto a second frog's heart. The dripping perfusion medium slowed the pulsation of the second heart upon stimulation of the donor heart. When the stimulus was removed, the donor heart returned to normal pulsation and shortly after the recipient heart returned to normal. The Vagus-substance in the perfusion fluid was identified by Feldberg and Krayer (1933) as acetylcholine.
Cholineacetylase forms acetylcholine from acetic acid and choline at the neuromuscular junction (Figure 2) in the presence of energy. The energy is obtained as a result of the action potential produced by the nerve impulse traveling along the nerve to the neuromuscular junction.
The acetylcholine proceeds to cross the synapse at the neuromuscular junction and activates the muscle causing its contraction. The persistent presence of the acetylcholine at the junction would result in constant depolarization of the muscle resulting in fibrillation. The acetylcholine is destroyed by acetylcholinesterase causing repolarization so that the muscle is able to respond to further nerve impulses.
Acetylcholine possesses two actions in vertebrates: muscarinic and nicotinic. The muscarinic action is exhibited at the neuromuscular functions of smooth muscle and sweat glands and the nicotine action is exhibited at the neuromuscular junction of striated muscle and at the synapse within ganglia.
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