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Purpose: The impact of temperature on the coagulative response to exercise has not been well described. The purpose of this study was to assess the response of plasma thrombin-antithrombin (TAT) to exercise during exposure to both hot and cold temperatures, and to compare those responses to exercise under normal, temperate conditions. Methods: Fifteen healthy male subjects (25.3 + 4.3 years) volunteered to participate in this study. Subjects completed maximal cycle ergometer exercise tests in three different temperatures (20°C, 5° or 8° C, and 30°C) in an environmental chamber. All tests were conducted in random order and separated by at least seven days. Blood samples were obtained before and immediately after exercise and analyzed by Elisa to determine plasma concentrations of thrombin-antithrombin complex (TAT). Results: Subjects demonstrated significantly elevated plasma levels of TAT in all three temperatures immediately after exercise (normal =1.04 ± 0.44 ng/ml, cold =1.34 ± 0.79 ng/ml, hot =1.18 + 0.95 ng/ml) when compared to baseline measures (normal = 0.45 ± 0.26 ng/ml, cold = 0.88 + 0.57 ng/ml, hot = 0.64 + g/ml). Subjects also showed significant elevations in TAT concentrations both before and after exercise in the cold temperature when compared with the normal temperature. There was no significant difference between the hot and normal temperatures. Conclusion: An individual's coagulation potential is increased following maximal physical exertion and may be further increased by exposure to colder temperature. Key Words: coagulation, physical exertion, temperature, thrombosis. / School of Physical Education, Sport, and Exercise Science

The purpose of this study was to determine if mild heat stress induced by wearing a wet suit while swimming in relatively warm water (25.4 ± 0.1°C) increases the risk of heat injury during the subsequent cycling and running stages. Specificlly, during an Olympic distance triathlon in a hot and humid environment (32°C & 65% RH). Five male triathletes randomly completed two simulated triathlons (Swim=30 min; Bike=40 km; Run=10 km) in the laboratory using a swimming flume, cycle ergometer, and running treadmill. In both trials, all conditions were identical, except for the swimming portion in which a full length, sleeveless neoprene wet suit was worn during one trial (WS) and a competitive brief swimming suit during the other (SS). The swim portion consisted of a 30 min standardized swim in which oxygen consumption (V02) was replicated, regardless of WS or SS. During the cycling and running stages, however, the subjects were asked to complete the distances as fast as possible. Core Temperature (T) was not significantly different between the SS and WS trials at any time point during the triathlon. However, mean skin temperature (TSk) and mean body temperature (Tb) were higher (p<0.05) in the WS at 15 (TSk=+4.1°C, Tb=+1.5°C) and 30 min (TSk=+4°C, Tb=+1.6°C) of the swim. These TSk and Tb differences were eliminated by 15 min of the cycling stage and remained similar (p>0.05) through the end of the triathlon. Moreover, there were no differences (p>0.05) in V02, heart rate (HR), rating of perceived exertion (RPE), or thermal sensation (TS) between the WS and SS. Additionally, no significant differences were found in cycling (SS=1:14:46 ± 2:48 vs. WS=1:14:37 ± 2:54 min), running (SS=55:40 ± 1:49 vs. WS=57:20 ± 4:00 min) or total triathlon times (SS=2:40:26 ± 1:58 vs. WS=2:41:57 ± 1:37 min). Therefore, the primary finding was that wearing a wet suit during the swimming stage of an Olympic distance triathlon in 25.4°C water does not adversely affect the thermal responses or the triathlete's ability to perform on the subsequent cycling and running stages. / School of Physical Education

The purpose of this study was to establish an exercise protocol that demonstrated a relationship between rest-interval duration and the exercise induced human growth hormone (hGH) response. Ten recreationally trained male subjects (23 ± 0.9 yrs) performed three leg extension trials on an Eagle — Cybex leg extension machine that consisted of 4 sets of 10 repetitions. The workload and volume was kept constant, but each trial had different rest-interval durations. Rest-interval durations between sets were at: 30 sec intervals (T-30), 60 sec intervals (T-60), or 120 sec intervals (T-120). Blood samples were obtained pre- and 0 — 30 minutes post-exercise and analyzed for lactate and hGH. All blood lactates rose significantly above baseline after exercise, with no differences in time of occurrence between trials. Blood lactates were significantly greater after the T-30 trials, compared to that of the T-60 and T-120 trials. There was no significant difference in hGH concentrations between trials. However, the data did suggest a relationship between rest-interval duration and the variability of hGH responses. The T-30 trials yielded significantly greater variation in hGH concentrations than the T-120 trials, and the T-120 trials showed significantly less variation than both the T-30 and T-60 trials. Although significant differences were found in these variations between trials, they did not prevent any significant differences in concentrations between trials from being found. In summary, the results of this study demonstrated an exercise related increase in lactic acid that had an inverse relationship to the length of the rest-intervals. hGH data on the other hand, did not show a relationship between rest-interval duration and the hGH concentrations. The connection between rest-interval duration and variability of hGH responses could possibly suggest that hGH values may have been significantly different if an exercise protocol higher in volume were utilized. Additionally, the results indicate that there is no direct relationship between blood lactate and hGH concentrations, as others have suggested. / School of Physical Education, Sport, and Exercise Science

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This investigation examined the influence of two levels of dietary sodium (Na+) intake on intramuscular and extracellular potassium (K+) content. Nine unacclimatized college males exercised. (90 minutes of treadmill walking, 5.6km/hr, 6X grade) in an environmental chamber maintained at 40.1( + .05)°C and 23.5( + . 4) %RH, during two 8-day dietary-acclimation regimens. The first regimen employed a high Na+ diet (399mEq/day), the second a low Na+ diet (98mEq/day); both diets contained 80mEq K+/day. Total body K+ stores increased during the high Na+ diet (+138mEq, 4.1%) and the-low Na+ diet (+114mEq, 3.4%). By day 8 (D8) of both treatments, subjects exhibited a significantly lower (p<.O5) mean heart rate and rectal temperature. Oxygen consumption and sweat rate were unaltered but sweat responsiveness (ml/hr/°c) progressively increased during the acclimation trials. Plasma volume increased +16.3% (D4) and +10.7% (D8) under the high Na+ diet and only +3.0% (D4) and +7.0% (D8) under the low Na+ diet. The low sodium diet was associated with a significantly higher (p<.05) mean heart rate (D3-D5), higher rectal temperature (D3-D6), lower sweat responsiveness (D2) and a delayed plasma volume expansion (D4)--resulting in less effective heat removal. The total content of K+, Na+, and C1- in plasma changed isoosmotically with plasma volume. The diets were associated with equivalent urine K+ excretion, and. during exercise in the heat both diets resulted in significantly decreased urine K+ losses. Muscle K+ and sweat K+ concentrations were not altered by dietary intervention or acclimation trials. Total muscle water-was significantly increased, in accord with Na+ and Cl- increases, on D8 of the low Na+ diet only. It was concluded that dietary intake of Na+, in combination with 8 days. of exercise in the heat and heavy sweating, does not significantly diminish the intramuscular K+ or total body K+ content.