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The mechanisms of platelet activating factor induced alterations in lung functions /Göggel, Rolf. January 2002 (has links)
Lübeck, University, Thesis (doctoral), 2001.
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Human antibody responses to a chlamydia-secreted protease factor : a thesis /Bosnic, Anthony Martin. January 2005 (has links)
Thesis (M.S.) --University of Texas Graduate School of Biomedical Sciences at San Antonio, 2005. / Vita. Includes bibliographical references.
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Modulation of the response to cutaneous injury /Muller, Michael John. January 2000 (has links) (PDF)
Thesis (M. Med. Sc.)--University of Queensland, 2001. / Includes bibliographical references.
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Potentiating effects of platelet activating factor on endothelin-1 induced rat arota vasoconstriction管漢偉, Koon, Hon-wai, Michael. January 1998 (has links)
published_or_final_version / Pharmacology / Master / Master of Philosophy
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Platelet activity and arachidonic acid metabolism: modulation by factors in plasma and cerebrospinal fluidand by dietUsman, Rukhsana. January 1994 (has links)
published_or_final_version / Biochemistry / Doctoral / Doctor of Philosophy
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Potentiating effects of platelet activating factor on endothelin-1 induced rat arota vasoconstriction /Koon, Hon-wai, Michael. January 1998 (has links)
Thesis (M. Phil.)--University of Hong Kong, 1999. / Includes bibliographical references (leaves 103-119).
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Platelet activity and arachidonic acid metabolism : modulation by factors in plasma and cerebrospinal fluid and by diet /Usman, Rukhsana. January 1994 (has links)
Thesis (Ph. D.)--University of Hong Kong, 1994. / Includes bibliographical references (leaves 155-179).
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Studies on formation and stabilization of pathological thrombi in vivoPožgajová, Miroslava. Unknown Date (has links) (PDF)
University, Diss., 2006--Würzburg. / Erscheinungsjahr an der Haupttitelstelle: 2005.
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Platelet Activating Factor Enhances in Vitro Fertilization of Rabbit OocytesRoudebush, William E., Minhas, Brijinder S., Ricker, Deborah D., Palmer, Thomas V., Dodson, Melvin G. 01 January 1990 (has links)
Capacitation of spermatozoa is essential for fertilization. Rabbit spermatozoa are particularly difficult to capacitate in vitro and require treatment with high-ionic-strength Brackett's defined medium. Spermatozoa treated with platelet activating factor had significantly higher fertilization rates when compared with nontreated (fresh, twice washed) spermatozoa (63% vs 34%). Fertilization rates of spermatozoa treated with platelet activating factor, although higher than those of high-ionic-strength capacitated spermatozoa, were not significantly different (63% vs 57%). Spermatozoa treated with lyso-platelet activating factor, the biologically inactive form of platelet activating factor, were noted to have fertilization rates similar to those of the untreated (noncapacitated) group. These data show that synthetic platelet activating factor treatment of uncapacitatedspermatozoa induces fertilization of rabbit oocytes in vitro in a manner similar to that for spermatozoa capacitated by high-ionic-strength media and significantly higher than that for untreated spermatozoa or after treatment with the biologically inactive form of platelet activating factor (lyso-platelet activating factor).
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The role of neutrophils in systemic anaphylaxis in the rabbitDunn, Anita Marie, 1956- January 1989 (has links)
The objective of this study was to determine whether neutrophils play a significant role in anaphylaxis or in the response to the anaphylactic mediator platelet activating factor (PAF) in the rabbit. Vinblastine and anti-neutrophil antibodies were compared as neutrophil depleting agents, and 0.35 mg/kg vinblastine was selected as optimal for efficiency and specificity of depletion. Anaphylaxis was induced in sensitized rabbits by intravenous antigen challenge. Neutrophil depletion to 399 ± 101 cells/mm³ blood (14 ± 3%) did not significantly inhibit the physiologic and hematologic events associated with anaphylaxis except tachycardia. However, vinblastine pretreatment significantly reduced tachycardia and the right ventricular pressure increase and abolished the increase in pulmonary resistance caused by intravenous PAF. We conclude that although neutrophils do not play a significant role in IgE-anaphylaxis, they are important in the PAF-induced increases in right ventricular pressure and pulmonary resistance. PAF may not be a major mediator of these two physiologic alterations in IgE-anaphylaxis.
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