Thesis (Ph. D.)--University of Wisconsin--Madison, 1981. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 194-209).

vii, 228 leaves : ill. ; 28 cm. / Title page, contents and abstract only. The complete thesis in print form is available from the University Library. / Thesis (Ph.D.)--University of Adelaide, Dept. of Surgery, 1970

Thesis (Ph.D.)-- University of Adelaide, Dept. of Surgery, 1970.

Thesis (Ph. D.)--University of Wisconsin--Madison, 1970. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.

No description available.

Thesis (Ph.D.)--Ohio University, November, 2004. / Includes bibliographical references (p. 137-159)

Thesis (Ph.D.)--Ohio University, November, 2004. / Title from PDF t.p. Includes bibliographical references (p. 137-159)

The present research explored the role of RES manipulation on ongoing Sidman avoidance behavior. Results of the first phase revealed that both experimental drugs significantly altered RES levels in predicted directions after the first measure; however, only stimulated subjects maintained significant differences after 5 days. No activity-level differences were noted in any subjects due to drugs across time. Sidman avoidance data indicated that RES-stimulated subjects showed significant deterioration in avoidance performance as compared to other groups for the first session. Stimulated subjects were also poorer on the second and third sessions, but statistical significance was not obtained because some saline subjects also showed poorer performance. A rank order correlation revealed that a significant negative correlation existed between RES stimulation and avoidance performance, based on changes in RES levels from baseline to the end of the shock program. These data suggest that increased stress resistance due to RES stimulation may reduce the aversive properties of the shock program, thus decreasing motivation for responding. It was concluded that artificial methods of inducing stress resistance by RES stimulation may be a useful therapeutic technique for patients experiencing psychological stress.

Although research has linked the central nervous system with changes in immunoresponsivity, research on the possible role of the central nervous system in altering reticuloendothelial activity is lacking. This study investigated the possible relationship between hypothalamic structures and changes in responsivity of the reticuloendothelial system. Eight male albino rats received bilateral electrode implants in the ventromedial area of the hypothalamus and, following brain stimulation, reticuloendothelial activity was assessed 3, 6, 12, 24, and 96 hours after stimulation. Brain stimulation decreased phagocytic activity of the reticuloendothelial system. These findings may increase our understanding of a possible neural mechanism underlying relationships between stress and resistance to disease states.