Spelling suggestions: "subject:"5ystemic lupus erythematosus"" "subject:"asystemic lupus erythematosus""
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Exploration of Pro- and Anti-inflammatory Effector Functions of Plasmacytoid Dendritic Cells in Systemic Lupus ErythematosusDavison, Laura Marie 03 September 2015 (has links)
No description available.
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Identifying Novel Disease-associated Variants and Understanding the Role of the STAT1-STAT4 Locus in SLEPatel, Zubin 15 December 2017 (has links)
No description available.
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K<sup>+</sup> Channel Trafficking in the Immunological Synapse of Human T Cells in Health and AutoimmunityNicolaou, Stella A. January 2007 (has links)
No description available.
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Identifying Common Genes from Rheumatoid Arthritis, Systemic Lupus, Multiple Sclerosis and Sjogrens Syndrome by Pooling Existing Microarray Data.Haynes, Eric E. 26 August 2013 (has links)
No description available.
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IL10 mRNA stability defects as a mechanism contributing to the development of lupusLi, Yuan 11 September 2015 (has links)
No description available.
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X Chromosome Gene Dosage in Autoimmune Disease Susceptibility and B Cell DevelopmentLiu, Ke (Coco) 24 October 2016 (has links)
No description available.
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RELATIONSHIP ADJUSTMENT, PARTNER SUPPORT, AND PSYCHOSOCIAL OUTCOMES FOR WOMEN WITH SYSTEMIC LUPUS ERYTHEMATOSUSLewis, Traci Lyn 20 December 2002 (has links)
No description available.
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The genetic complexity and protein polymorphism of complement c4 in health and diseaseYang, Yan 21 July 2004 (has links)
No description available.
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Validation and verification of the Japanese version of the systemic lupus erythematosus symptom checklist for patient quality of life / 日本語版systemic lupus erythematosus symptom checklistの信頼性及び妥当性の検証Doi, Hiroshi 23 March 2022 (has links)
京都大学 / 新制・課程博士 / 博士(医学) / 甲第23790号 / 医博第4836号 / 新制||医||1057(附属図書館) / 京都大学大学院医学研究科医学専攻 / (主査)教授 山本 洋介, 教授 佐藤 俊哉, 教授 柳田 素子 / 学位規則第4条第1項該当 / Doctor of Medical Science / Kyoto University / DFAM
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The Biology of Dendritic Cells in the Context of AutoimmunityQiu, Connie Claire January 2019 (has links)
Systemic lupus erythematosus (SLE) is a complex autoimmune disease that affects at least five million people worldwide. An increased expression of type I interferon (IFN) regulated genes is a hallmark of SLE, but the precise etiology of SLE initiation and flares is poorly understood. Because plasmacytoid dendritic cells (pDCs) are the primary type I IFN producers, their role in SLE has long been suspected, with murine pDC depletion models successfully delaying the progression of murine lupus-like disease. However, the mechanism behind how exactly how pDCs contribute to lupus autoimmunity is unknown, contributing to the current dearth lack of disease modifying treatments; current treatments only succeed in suppressing symptoms, and do not halt disease progression. In this study, we take a multifactorial approach to understanding the biology of pDCs in the context of lupus autoimmunity. Although the exact etiology of lupus is unknown, infections are an important environmental trigger for / Infectious Disease & Immunity
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