Global ETD Search

71	Epidemiologic study of risk factors for Ewing's sarcoma family of tumors in Australia / Valery, Patricia Casarolli. January 2002 (has links) (PDF) Thesis (Ph. D.)--University of Queensland, 2002. / Includes bibliographical references. Ewing's sarcoma. Tumors.
72	Maternal prenatal consumption of bioflavonoids and phenolic acids and risk of childhood brain cancer Lal, Priya Kumari, January 2004 (has links) Thesis (Ph. D.)--Ohio State University, 2004. / Title from first page of PDF file. Document formatted into pages; contains xvii, 274 p.; also includes graphics (some col.). Includes abstract and vita. Advisor: J. Schwartzbaum, School of Public Health. Includes bibliographical references (p. 171-203).
73	Differential gene expression in gestational trophoblastic disease / Fong, Pui-yee. January 2001 (has links) Thesis (M. Phil.)--University of Hong Kong, 2001. / Includes bibliographical references (leaves 150-165).
74	Molecular mechanisms in the development of odontogenic keratocysts / Nawshad, Ali Irteza. January 2000 (has links) (PDF) Thesis (Ph. D.)--University of Queensland, 2001. / Includes bibliographical references. Odontongenic cysts. Odontongenic tumors.
75	Studies on the fibrinolytic activity in human tumour tissues : isolation and characterization of a plasminogen activator and an inhibitor in human tumour tissues / Cheung, Wai-kin, Alfred Benjamin. January 1984 (has links) Thesis (Ph. D.)--University of Hong Kong, 1985. Tumors. Antifibrinolytic agents.
76	Molecular and cellular biology of the multiple endocrine neoplasia type 1 tumour suppressor gene / Bergman, Lee Melissa. January 2001 (has links) (PDF) Thesis (Ph. D.)--University of Queensland, 2002. / Includes bibliographical references. Molecular biology. Cytology. Tumors
77	Significance of latency change, amplitude change in intra-operative motor evoked potential by transcranial electrical stimulation duringsupratentorial craniotomy in predicting surgical outcome Chan, Ping-hon, 陳秉漢 January 2006 (has links) published_or_final_version / Medical Sciences / Master / Master of Medical Sciences Electromyography. Brain - Tumors - Surgery.
78	Molecular cytogenetic, epigenetic and tissue dynamic study of gestational trophoblastic disease Xue, Weicheng., 薛衛成. January 2004 (has links) published_or_final_version / Pathology / Doctoral / Doctor of Philosophy Trophoblastic tumors - Genetic aspects.
79	Role of kinesin superfamily 7 in gestational trophoblastic disease Ho, Wing-kiu, Joanna., 何泳翹. January 2011 (has links) published_or_final_version / Pathology / Master / Master of Research in Medicine Kinesin. Trophoblastic tumors.
80	The role of FoxD3 in gestational trophoblastic disease Chiu, Ka-yue., 招家裕. January 2012 (has links) Gestational trophoblastic disease (GTD) is arised from the neoplastic trophoblasts in placenta. Trophoblasts have the characteristic of proliferation and invasion. GTD is classified as partial hydatidiform mole (PHM), complete hydatidiform form mole (CHM), invasive hydatidiform mole (IHM), choriocarcinoma (CCA), placental site trophoblastic tumour (PSTT), epithelioid trophoblastic tumour (ETT), exaggerated placental site trophoblastic reaction (EPSR) and placental site nodule (PSN). HM has the potential to develop into malignant trophoblastic disease, and metastasis to other parts of body. FoxD3 gene belongs to Forkhead family. Its protein acts as embryonic stem cell transcription factor and plays an important role in neural crest and placenta development. Previous studies from our team have reported that other embryonic stem cell transcription factors, such as Nanog, Sox2 Oct4 and Stat3, are related with pathogenesis of GTD. This study aim is to investigate the protein expression profile of FoxD3 in different types of GTD using immunohistochemistry method. In this study, 70 formalin fixed paraffin embedded tissue blocks from 16 normal first trimester placenta, 38 CHM, 9 CCA, 5 PSTT and 2 ETT were retrieved. Paraffin sections were prepared and stained with FoxD3 antibody by using immunohistochemistry method. Compared with normal placentas, there was significantly increased expression of FoxD3 in trophoblasts of CM and PSTT (p<0.05). In CCA, there was high expression of FoxD3 in syncytiotrophoblasts and intermediate trophoblasts (p<0.05). In ETT, the immunoreactivity of FoxD3 is not significantly increased when compared with intermediate trophoblasts (p=0.07). To conclude, FoxD3 was found to be over-expressed in GTD. FoxD3 may contribute to pathogenesis of GTD. Further investigations are needed to discover the relationship with other embryonic transcription factors and genes to improve the diagnosis, prognosis and treatment of GTD. / published_or_final_version / Pathology / Master / Master of Medical Sciences Trophoblastic tumors. Transcription factors.

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