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Rekonstitution der gekoppelten Fumarat-Atmung von Wolinella succinogenesBiel, Simone. January 1900 (has links) (PDF)
Frankfurt (Main), Univ.,Diss., 2003. / Erscheinungsjahr an der Haupttitelstelle: 2002. Computerdatei im Fernzugriff.
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Penjelidikan Vibrio el tor jang diasingkan di Djakarta, dan tjara baru untuk membedakan antara Vibrio el tor dan Vibrio choleraeTjia, Soe Khie. January 1900 (has links)
Thesis--Universitas Indonesia. / In Indonesia. Summary in English. "Dalil-dalil" (1 p.) inserted. Bibliography: p. 81-94.
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Structural biology of Vibrio cholerae pathogenicity factors /Sheikh, Md. Arif. January 2009 (has links)
Thesis (Ph.D.) - University of St Andrews, May 2009.
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Rekonstitution der gekoppelten Fumarat-Atmung von Wolinella succinogenesBiel, Simone. January 1900 (has links) (PDF)
Frankfurt (Main), Universiẗat, Diss., 2003. / Erscheinungsjahr an der Haupttitelstelle: 2002.
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Biosynthesis and Regulation of Poly-3-Hydroxybutyrate in Vibrio HarveyiSun, Weiqun January 1994 (has links)
Note:
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Study in the Propagation Conditions for Vibriophages £pVP and £pVAChiu, Chi-Wen 26 July 2010 (has links)
Seventeen strains of multi drug-resistant strains were isolated from aquaculture farms in previous study, and 82% (14/17) of these strains are belong to Vibrio species, which is one of the major pathogens in the aquaculture farms. Vibrio disease (vibriosis) which caused by the Vibrio will cause the fish skin ulcer with bleeding and gastrointestinal symptoms such as swelling. The highest mortality rate of Vibrio infected marine fish is up to 100%. Two vibriophages, Vibrio parahaemolyticus vibriophage £pVP2 and Vibrio alginolyticus vibriophage £pVA2, were isolated from aquaculture environments in this study. The morphology of vibriophage was examined by electron microscopy. The £pVP2 phage has an oval head with a tail and six tail fibers. As £pVA2, an icosarhedral head with an short tail is observed. In the DNA electrophoresis analysis, a fragment at 17.8k bp was observed in both £pVA2 and £pVP2 sample. Restriction endonuclease digestion pattern revealed that the £pVA2 vibriophage DNA can be cut into various small size of DNA fragments by EcoR I and Xba I endonuclease. In the host-range analysis, £pVP2 can infect Vibrio parahaemolyticus and Vibrio alginolyticus, while £pVA2 and £pVP2 cannot infect other Vibrio spp., Pasteurella, E. Coli, and antibiotic-resistant bacteria isolated from aquaculture farms. £pVA2 and £pVP2 can survive at the temperature below 65¢XC and 63¢XC, respectively. To obtain the maximum proliferation of vibriophage, different conditions were tested, including infection time point, host bacteria age, MOI (multiplicity of infection) and various mediums. Vibriophage £pVA2 was proliferated under following conditions: (1) phage was add to the medium with host seed together; (2) the overnight cultured host bacteria was used as seed; (3) the host cells were infected at MOI= 10-6; (4) MSWYE media was used as culture media. Vibriophage £pVP2 was proliferated following £pVA2 conditions but MOI values was 10-4.
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Studies on Venezuelan fish and shrimp associated bacteriaAlvarez, Julia D. January 1998 (has links)
No description available.
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In vitro and in vivo cold shock response in Vibrio vulnificusLimthammahisorn, Suttinee, Brady, Yolanda Juanita, Arias, Covadonga R. January 2007 (has links) (PDF)
Dissertation (Ph.D.)--Auburn University, 2007. / Abstract. Vita. Includes bibliographic references.
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Analysis of the vlpA genes and their roles in the pathogenesis of Vibrio cholerae O139 / Pranom Kaewrakon.Pranom Kaewrakon January 2000 (has links)
Includes bibliographical references (leaves 196-229). / 229 leaves : ill. (some col.) ; 30 cm. / Title page, contents and abstract only. The complete thesis in print form is available from the University Library. / A mutant lacking Vibrio cholerae lipoprotein A activity has been constructed in V. cholerae O139 which contains two copies of vlpA, by allelic exchange after inactivation via the insertion of kanamycin and tetracycline resistance gene cartridges. The vlpA-negative mutant has been assessed for virulence in the infant mouse cholera model. / Thesis (Ph.D.)--Adelaide University, Dept. of Molecular Biosciences, 2001
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Analysis of the vlpA genes and their roles in the pathogenesis of Vibrio cholerae O139 / Pranom Kaewrakon.Pranom Kaewrakon January 2000 (has links)
Includes bibliographical references (leaves 196-229). / 229 leaves : ill. (some col.) ; 30 cm. / Title page, contents and abstract only. The complete thesis in print form is available from the University Library. / A mutant lacking Vibrio cholerae lipoprotein A activity has been constructed in V. cholerae O139 which contains two copies of vlpA, by allelic exchange after inactivation via the insertion of kanamycin and tetracycline resistance gene cartridges. The vlpA-negative mutant has been assessed for virulence in the infant mouse cholera model. / Thesis (Ph.D.)--Adelaide University, Dept. of Molecular Biosciences, 2001
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