Fetal alcohol syndrome : changes in transcriptional activation in the cerebellum caused by ethanol exposure during neurodevelopment /Acquaah-Mensah, George Kwamina, January 2001 (has links)
Thesis (Ph. D.)--University of Texas at Austin, 2001. / Vita. Includes bibliographical references (leaves 102-114). Available also in a digital version from Dissertation Abstracts.
Bayly, Ronald Cecil.
Thesis (M. Sc.)--University of Melbourne, 1960. / Typescript. Includes bibliographical references (leaves 118-125).
Alcohol withdrawal syndrome : characterisation, predictors of severity, and relationship to relapse /Humeniuk, Rachel. January 1999 (has links) (PDF)
Thesis (Ph.D.) -- University of Adelaide, Dept. of Clinical and Experimental Pharmacology, 2000. / Bibliography: leaves 246-263.
Cleaveland, Bonnie L.,
Thesis (M.S.)--Virginia Polytechnic Institute and State University, 1992. / Vita. Abstract. Includes bibliographical references (leaves 50-53). Also available via the Internet.
Influence of ethanol on furfural yield in the coproduction of ethanol and furfural from wood hydrolyzateTabata, Haruro. January 1961 (has links)
Thesis (M.S.)--University of Wisconsin--Madison, 1961. / Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 47-50).
Burnell, Joe C.
This document only includes an excerpt of the corresponding thesis or dissertation. To request a digital scan of the full text, please contact the Ruth Lilly Medical Library's Interlibrary Loan Department (firstname.lastname@example.org).
Effects of ethanol on glutethimide absorption, distribution and metabolism in relationship to a mechanism for toxicity enhancement /Hetland, Lynn B. January 1973 (has links)
No description available.
To giveth and taketh away determination of taurine's protective role during ethanol withdrawal through supplementation and depletion paradigms /Zalud, André W. Diaz-Granados, Jamie L. January 2008 (has links)
Thesis (Ph.D.)--Baylor University, 2008. / Includes bibliographical references (p. 113-134).
Osteopontin-mediated neutrophilic infiltration and higher liver injury in a female rodent alcoholic steatohepatitis modelBanerjee, Atrayee 15 May 2009 (has links)
Females are known to be more susceptible to alcoholic liver disease (ALD), but the precise mechanism behind this increased susceptibility is not well understood. The objective of this study was to identify the molecular mechanism behind the increased susceptibility of females to alcoholic steatohepatitis (ASH). Female rats in ASH model were found to have significantly higher neutrophilic infiltration in the liver as compared to the males. Osteopontin (OPN), a member of the SIBLING family of proteins was also found to be induced in females. Neutralizing OPN antibody experiments provided further evidence that OPN acts as a chemokine in attracting neutrophils into the liver making females more susceptible to ASH. Since neutrophil transmigration in the liver is mediated by intergins, the mechanism for OPN-mediated neutrophil infiltration was tested. Females in ASH had significantly higher expression of α4β1 and α9β1 integrins, and animals treated with neutralizing OPN antibody had significantly lower expression of these integrins, wherein hepatic neutrophilic infiltration was also decreased by 50% compared to the untreated group. Immunoprecipitation experiments suggested the binding of OPN to α4β1 and α9β1 integrins. OPN-mediated neutrophilic infiltration was further confirmed using Boyden chamber assays. Antibodies directed against α4, β1 integrins and SLAYGLR sequence was also found to significantly inhibit neutrophilic migration in vitro, suggesting that higher hepatic neutrophil chemokinesis in the female ASH appears to be mediated through both α4β1 and α9β1 integrin signaling. In addition, higher liver injury and higher expression of OPN in females were also found to be regulated by estrogen in a biphasic pattern; ovariectomy resulted in significantly increased liver injury compared to intact rats. Depending on dose, estradiol supplementation in the ovariectomized rats fed ethanol resulted in both a protective and damaging effect on liver. Besides OPN, several other oxidative stress related proteins like Ferritin H Chain, ER60, HSP60, Peroxiredoxin 6 were identified by proteomics approach. Females in ASH were found to have differentially regulated levels of these proteins as compared to their male counterparts, highlighting the potential novel mechanisms associated with higher liver injury noted in our female rat ASH model.
The preparation and properties of the chloromethyl ethers and formals of high molecular weight alcoholsMetcalf, Joe Shelby 05 1900 (has links)
No description available.
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