Thesis (Ph.D.)--Murdoch University, 2003. / Appendix 1 attached as CD-ROM. Thesis submitted to the Division of Science and Engineering. Bibliography: leaves [156]-173.

Thesis (M.S.)--University of North Texas, August, 2008. / Title from title page display. Includes bibliographical references.

Thesis (M. Ed.)--Kutztown University of Pennsylvania, 1990. / Source: Masters Abstracts International, Volume: 45-06, page: 3051. Typescript. Includes bibliographical references (leaves 42-46).

Thesis (Ph. D.)--University of Missouri-Columbia, 2002. / Typescript. Vita. Includes bibliographical references (leaves 177-191). Also available on the Internet.

Thesis (Ph. D.)--University of Missouri-Columbia, 2002. / Typescript. Vita. Includes bibliographical references (leaves 177-191). Also available on the Internet.

System requirements, Adobe Acrobat reader, Microsoft Power Point. / Includes bibliographical references (Leaves: [289]-291)

(M.Arch.)--University of Hong Kong, 1999. / Includes special report entitled : Building systems and materials for assembly construction. Includes bibliographical references. Also available in print.

Thesis (Ph.D. (Zoology and Entomology)) - Rhodes University, 2007.

<p> The domestication of tuna species has proven to be one of the most challenging endeavours in aquaculture. Among the issues yet unresolved is the occurrence of &lsquo;puffy snout syndrome,&rsquo; a condition in which tumour-like growths form on the head. Ultimately, vision and feeding are impaired and long-term afflictions typically result in mortality. Though few mentions of puffy snout exist in the literature, evidence suggests that it is not uncommon among facilities that hold tunas in captivity. The specific aims of this study were to: a) describe pathological features of puffy snout, and b) investigate its etiology through the evaluation of conditions and protocols at facilities rearing/holding tuna. </p><p> To describe pathological features of puffy snout, clinical signs were detailed by observing captive tunas in a land-based holding system, and examination of evidence of infection by parasites, bacteria, and viral agents was conducted on tissues collected from fish with and without puffy snout. Histological examination of tissue from normal and affected fish was also conducted. To investigate etiology, a survey was developed and sent electronically to 28 tuna-holding facilities globally. The survey inquired about the prevalence of puffy snout and the husbandry conditions and protocols employed (e.g., biological characteristics, capture and transport procedures, holding system design and water quality, feeding regime). These data were compared across facilities in an attempt to couple puffy snout prevalence with holding conditions and/or protocols. </p><p> Clinical signs of puffy snout included occlusion of the eyes and mouth, followed by changes to swimming and feeding behaviours. Parasitology, bacteriology, and virology examination all indicated no commonly-found pathological agents were responsible for inducing the condition. Histology showed that puffy snout is largely characterised by the apparent degeneration of muscle tissue with the replacement of a loose collagenous fibrosis and an undetermined fluid filling the interstitial space in tissues anterior to the eye. In dorsal musculature, collagenous growth may occur in the epidermal or hypodermal regions. </p><p> Based on survey data, the capture and transport process and feeding regime were unrelated to development of puffy snout. However, certain biological (e.g., fish size) and holding system (e.g., tank/pen size) parameters showed weak but non-dismissible coupling with puffy snout prevalence. Survey data and additional personal communication with field researchers confirmed that puffy snout in tuna is solely a captivity-related condition.</p>

<p> The introduction of lake trout <i>Salvelinus namaycush</i> into Yellowstone Lake preceded the collapse of the native Yellowstone cutthroat trout <i>Oncorhynchus clarkii bouvieri</i> population. As a system with a simple fish assemblage and several long-term data sets, Yellowstone Lake provided a unique opportunity to evaluate the ecology of a native salmonid in the presence of a non-native salmonid population undergoing suppression in a large natural lake. Diet data for Yellowstone cutthroat trout and lake trout were evaluated at varying densities to determine the effects of density on diet composition. Temporal diet shifts from 1996-1999 to 2011-2013 were likely caused by limitation of prey fish for lake trout. Diets, stable isotopes, and depth-related patterns in CPUE indicated lake trout > 300 mm consumed primarily amphipods, making them trophically similar to Yellowstone cutthroat trout from during 2011-2013. A lake trout removal program was initiated during 1995 to reduce predation on Yellowstone cutthroat trout. Abundance and fishing mortality were estimated for lake trout from 1998 through 2013 and Yellowstone cutthroat trout from 1986 through 2013. Density-dependence was evaluated by examining individual growth, weight, maturity, and pre-recruit survival as a function of abundance. In addition, a simulation model was developed for the lake trout-Yellowstone cutthroat trout system to determine the probability of Yellowstone cutthroat trout abundance persisting at performance metrics given potential reductions in lake trout abundance. Estimates of Yellowstone cutthroat trout abundance varied 5-fold and lake trout abundance varied 6-fold. Yellowstone cutthroat trout weight and pre-recruit survival decreased with increasing Yellowstone cutthroat trout abundance; however, individual growth and maturity were not related to abundance. Lake trout population metrics did not vary with lake trout abundance. Simulation model results were variable because of uncertainty in lake trout pre-recruit survival. Conservative estimates for required lake trout reductions were > 97% of 2013 abundance for a > 70% probability of Yellowstone cutthroat trout persistence at the performance metrics outlined in the Native Fish Conservation Plan. Lake trout removal will likely reduce lake trout abundance and result in Yellowstone cutthroat trout recovery if the amount of fishing effort exerted in 2013 is maintained for at least 15 years.</p>