Thesis (B.Sc)--University of Hong Kong, 2001. / "A dissertation submitted in partial fulfilment of the requirements for the Bachelor of Science (Speech and Hearing Sciences), The University of Hong Kong, May 4, 2001." Also available in print.

Thesis (B.Sc)--University of Hong Kong, 2002. / "A dissertation submitted in partial fulfilment of the requirements for the Bachelor of Science (Speech and Hearing Sciences), The University of Hong Kong, May 10, 2002." Also available in print.

Thesis (B.Sc)--University of Hong Kong, 1994. / "A dissertation submitted in partial fulfilment of the requirements for the Bachelor of Science (Speech and Hearing Sciences), The University of Hong Kong, April 29, 1994." Also available in print.

Thesis (B.Sc)--University of Hong Kong, 1994. / "A dissertation submitted in partial fulfilment of the requirements for the Bachelor of Science (Speech and Hearing Sciences), The University of Hong Kong, 29th April, 1994." Also available in print.

Thesis (D.M.A.)--University of North Carolina at Greensboro, 2007. / Title from PDF title page screen. Advisor: Kelly J. Burke; submitted to the School of Music. Includes bibliographical references (p. 31-37).

Thesis (Ph.D.)--University of Nebraska-Lincoln, 2006. / Title from title screen (site viewed May 23, 2007). PDF text: vi, 79 p. : ill. ; 0.35Mb UMI publication number: AAT 3236907. Includes bibliographical references. Also available in microfilm and microfiche formats.

Thesis (Honors)--College of William and Mary, 2009. / Includes bibliographical references (leaves 30-33). Also available via the World Wide Web.

Thesis (Ed.M.)--Boston University / The inability to hear similarities and differences between words causes difficulty for many children in learning to read. Since children with this failing should be trained in auditory discrimination, it is imperative that the teacher recognize those who require additional ear training. Many attempts have been made to develop a practical measure which discriminates between the mediocre and poor achievers in auditory perception of initial consonants. Boston University has developed such a test which includes explicit directions geared to the nonreader, is quick-scoring, and does not waste the precious quota of reading instruction time. The purpose of this study is to analyze 233 scores of the abovementioned test in terms of reliability, measures of dispersion, and the effects of teaching.

Thesis (Ph.D.)--Boston University. / Rats were trained on three schedules of reinforcement, a sound aversive schedule, a light aversive schedule and a sound and light discrimination schedule. On the aversive schedules a press response terminated the aversive stimulus. On the discrimination schedule a response in the presence of either stimulus produced food. An auditory threshold was measured on this schedule. Following training the animals received electrolytic lesions in the ventral auditory system of the medulla. Following this they were tested on the behavioral schedules. The brain of each animal was then removed and impregnated with protargol for microscopic examination. Six rats received unilateral lesions, seven received bilateral lesions and six received sham operations. The results were as follows. Of the six animals who received unilateral lesions, one showed a substantial loss of the sound aversive behavior. Of the seven who received bilateral lesions five suffered a loss of the aversive behavior. Of these five, two had a substantial increase in the discriminative threshold. The behavior of the animals who received sham operations was essentially unaffected. There was a consistent relation between extensive damage to the large fiber pathway, the superior olivary pathway and the small fiber pathway and loss of the auditory aversive behavior. Destruction of the superior olivary pathway was not sufficient to produce a loss of the aversive behavior. The suggestion in the literature that the large fiber pathway is responsible for the maintenance of the aversive behavior was confirmed. Destruction of the superior olivary pathway either alone or in combination with destruction of the large fiber pathway did not materially change the auditory discriminative threshold. Destruction of all ventral acoustic pathways caused a loss of both aversive and discriminative auditory behaviors. Dorsal auditory pathways did not by themselves support either behavior. It has not been possible to determine if destruction of the small fiber pathway by itself can cause a loss of discriminative behavior. It was not possible to determine if return of the release response was due to the lesion or due to the loss of the press response.

Humans report a strong pitch percept in response to a complex tone – the sum of a series of harmonics – presented to either a single ear (‘monaurally’) or both ears (‘diotically’). Interspike interval histograms of responses of neurons in the auditory system to monaural complex tones show a peak at the period of the pitch reported by humans – a ‘neural correlate of pitch’. However, the same pitch percept can be generated by presenting complexes with harmonics distributed across both ears (‘dichotically’). This requires combination of the neural signals underlying pitch from both sides of the auditory system, termed ‘binaural fusion’. Temporal coding generally deteriorates along the auditory pathway; binaural fusion should occur at a relatively early stage. One of the prime candidates is in the superior olivary complex (SOC). Although the guinea pig auditory system has been extensively studied, this work is the first in vivo investigation of the guinea pig SOC. Cells of the lateral superior olive (LSO) show sensitivity to interaural level differences; medial superior olive (MSO) cells show sensitivity to interaural time differences. Additionally, cells with responses similar to the medial nucleus of the trapezoid body (MNTB) and superior paraolivary nucleus (SPN) of other species were found in the guinea pig SOC. Presumed MNTB cells showed a three-component spike waveform shape; presumed SPN cells responded at the offset of contralaterally-presented stimuli. MSO and LSO cells respond to the overall pitch of complex tones, even if the monaural waveforms presented to each ear differ; this is consistent with the perception of humans. In contrast, cells of the ventral cochlear nucleus, which provide the main input to MSO and LSO cells, do not show evidence of a binaural pitch response. In conclusion, SOC cells are able to encode the pitch of binaural complex tones in their spike timing patterns.