Introduction: Nursing clinicians are primarily responsible for the monitoring and treatment of increased body temperature. The body temperature of patients during their acute care hospital stay is measured at regular repeated intervals. In the event a patient is assessed with an elevated temperature, a multitude of decisions are required. The action of instigating temperature reducing strategies is based upon the assumption that elevated temperature is harmful and that the strategy employed will have some beneficial effect. Background and Significance: The potential harmful effects of increased body temperature (fever, hyperthermia) following neurological insult are well recognised. Although few studies have investigated this phenomenon in the diagnostic population of non-traumatic subarachnoid haemorrhage, it has been demonstrated that increased body temperature occurs in 41 to 72% of patients with poor clinical outcome. However, in the Australian context the frequency, or other characteristics of increased body temperature, as well as the association between increased body temperature with poor clinical outcome has not been established. Design: This study used a correlational study design to: describe the frequency, duration and timing of increased body temperature; determine the association between increased body temperature and clinical outcome; and describe the clinical interventions used to manage increased body temperature in patients with non-traumatic subarachnoid haemorrhage. A retrospective clinical chart audit was conducted on 43 patients who met the inclusion criteria. Findings: The major findings of this study were: increased body temperature occurred frequently; persisted for a long time; and onset did not occur until 20 hours after primary insult; increased body temperature was associated with death or dependent outcome; and no intervention was recorded in many instances. Conclusion: This study has quantified in a non-traumatic subarachnoid haemorrhage patient population the characteristics of increased body temperature, established an association between increased body temperature with death or dependent outcome and described the current management of elevated temperatures in the Australian context to improve nursing practice, education and research.

Despite high prevalence rates of insomnia among the elderly, very few treatment options adapted to the characteristics and needs of this population group exist. This study aims to investigate the efficacy of a thermal biofeedback based technique in treating sleep onset insomnia in an elderly sample. Eight healthy elderly good sleepers and eight healthy elderly sleep onset insomniacs participated in the study. The effect of the thermal biofeedback on participants' forehead temperature, clavicular temperature, right hand temperature, left hand temperature, stomach temperature, thigh temperature, right foot temperature, core body temperature, and sleep latency was assessed. Furthermore, the impact of the thermal biofeedback technique on participants' subjective ratings of biofeedback proficiency and sleepiness was investigated. The thermal biofeedback protocol failed to generate adequate and consistent changes in participants' body temperature, indicating that it is of limited practical used as a treatment of sleep onset insomnia in its present form. Several interesting trends observed during the current study are explored and areas for further research identified. / Thesis (MPsy(Clinical))--University of South Australia, 2006.

This thesis examines the following theory in several parts: across a wide range of experimental situations, changes in core body temperature (Tc) have been consistantly associated with inverse changes in sleep propensity. Previous research has focussed on the possible role that a declining Tc and distal vasodilation may play in normal nocturnal sleep onset. This research has lead to the suggestion that these changes in thermoregulation may mediate, or be functionally involved in, the regulation of sleep propensity in sedative-hypnotic agents.

Thesis (Ph.D.) -- McMaster University, 1997. / Includes bibliographical references (leaves 67-70). Also available via World Wide Web.

Thesis (Ph.D.) -- University of Adelaide, Dept. of Obstetrics and Gynaecology, 1998? / Bibliography: leaves 162-197.

Thesis (M.S.)--Virginia Polytechnic Institute and State University, 1990. / Vita. Abstract. Includes bibliographies. Also available via the Internet.

Thesis (Ph. D.)--Ohio State University, 2004. / Title from first page of PDF file. Document formatted into pages; contains xviii, 257 p.; also includes graphics (some col.) Includes bibliographical references (p. 245-257).

Thesis (Ph. D.)--University of Wisconsin--Madison, 1963. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves [131]-145).

Thesis (Ph. D.)--University of Wisconsin--Madison, 1962. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 97-100).

Thesis (M.S.)--University of Wisconsin-Madison. / Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 37-40).