61 |
Functional neurobiology in normal aging, mild cognitive impairment and Alzheimer's disease : focus on visuospatial processing using functional magnetic resonance imaging /Vannini, Patrizia, January 2007 (has links)
Diss. (sammanfattning) Stockholm : Karolinska institutet, 2007. / Härtill 5 uppsatser.
|
62 |
Neural correlates of irrelevant speech /Gisselgård, Jens, January 2006 (has links)
Diss. (sammanfattning) Stockholm : Karolinska institutet, 2006. / Härtill 4 uppsatser.
|
63 |
Caregiving for children who have had a traumatic brain injury structuring for security : a thesis submitted to Auckland University of Technology in partial fulfilment of the degree of Master of Health Science, December 2003.Jones, Margaret A. January 2003 (has links) (PDF)
Thesis (MHSc--Health Science) -- Auckland University of Technology, 2003. / Also held in print (251 leaves, 30 cm.) in North Shore Theses Collection (T 617.4810443083 JON).
|
64 |
Algorithms to detect high frequency oscillations in human intracerebral EEGChander, Rahul. January 1900 (has links)
Thesis (M.Eng.). / Written for the Dept. of Biomedical Engineering. Title from title page of PDF (viewed 2008/05/13). Includes bibliographical references.
|
65 |
Signal Processing for Time Series of Functional Magnetic Resonance ImagingZhu, Quan January 2008 (has links)
Thesis (Ph. D.)--Duke University, 2008. / Includes bibliographical references.
|
66 |
Quantitative assessment of glioma therapy effiicacy using magnetic resonance diffusion tensor imagingGoel, Priya. January 2008 (has links) (PDF)
Thesis (M.S.)--University of Alabama at Birmingham, 2008. / Additional advisors: Louis B. Nabors, James M. Markert, Donald B. Twieg. Description based on contents viewed July 8, 2009; title from PDF t.p. Includes bibliographical references (p. 73-79).
|
67 |
Automatic segmentation of brain structures for radiotherapy planningJoshi, Pallavi V. January 2005 (has links)
Thesis (M.S. in Electrical Engineering)--Vanderbilt University, May 2005. / Title from title screen. Includes bibliographical references.
|
68 |
Quantitative MRI analysis of human brain development following pre and perinatal brain injury /Moses, Pamela. January 1999 (has links)
Thesis (Ph. D.)--University of California, San Diego, 1999. / Vita. Includes bibliographical references.
|
69 |
The effects of atomoxetine on cognitive performace [sic] and neuroplasticity after traumatic brain injuryReid, Wendy Murdock, January 1900 (has links)
Thesis (Ph.D.)--Virginia Commonwealth University, 2008. / Prepared for: Dept. of Anatomy & Neurobiology. Title from title-page of electronic thesis. Bibliography: leaves 155-194.
|
70 |
Long-term effects of prenatal and early postnatal environment on brain remodelling : focus on hippocampal volume and astrogliaShende, Vishvesh H. January 2013 (has links)
The main aim of this thesis was to assess if early deprivation (ED) and glucocorticoid (GC) treatment exert long-term effects on the volume of the brain regions implicated in responses to stress, and if it associates with alterations in the distribution and structure of astroglia, which are known to support brain plasticity. This study also investigated the effects of prenatal dexamethasone (Dex) treatment on selected brain receptors, namely the oxytocin and 5-HT1A receptors, as they are implicated in the regulation of responses to stress. In addition, in vitro effects of Dex on neural stem cells were studied, in order to explore the drug effects on cell proliferation and differentiation, and on glial cell markers. Unbiased stereological estimation was employed to determine the regional brain volume, astroglial morphology and total cell count. Peripheral quantitative computed tomography (pQCT) technique was used to quantify total brain volume. Autoradiography technique was employed to visualise and analyse oxytocin and 5HT-1A serotonin receptor binding using selective radioactive ligands. The results of the present study demonstrate that both ED and prenatal Dex exposure leads to long-term effects on hippocampal remodelling with volume losses and impoverished astroglial morphology in the form of reduced primary process length. The observed deterioration in astroglial morphology adds further evidence that astrocytic changes contribute to hippocampal volume losses, a phenomenon that deserves more research in the context of effects of corticosteroid overload and stress-related pathologies. The present results also demonstrate that prenatal Dex induces long-term effects at the level of central neuroregulatory processes. Thus significant region- and sex-dependent reductions or increases in the oxytocin and 5-HT1A receptor binding were observed. The in vitro study has shown that Dex affects both proliferation and differentiation of GFAP positive NSCs with no toxic effects as such. Overall, both early postnatal or prenatal manipulations that increase levels of stress and/or glucocorticoids as the chemical mediators of stress, lead to a long-term maladaptive brain remodelling with losses in the hippocampal volume, impoverishment of hippocampal astroglial morphology and changes in the properties of central regulatory receptors in the brain areas involved in the reaction to stress.
|
Page generated in 0.0409 seconds