Spelling suggestions: "subject:"papillomavirus.""
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Information amount and patient empowerment participation in the HPV vaccination decision-making process /Eisenberg, Dana J. January 2009 (has links)
Thesis (M.A.)--Ohio State University, 2009. / Title from first page of PDF file. Includes vita. Includes bibliographical references (p. 39-43).
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HPV genotyping and integration in cervical cancer and precursor lesionsTsang, Chi-kit, Percy., 曾誌傑. January 2005 (has links)
published_or_final_version / Medical Sciences / Master / Master of Medical Sciences
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Evaluation and comparison of molecular diagnostic methods for detection of human papillomavirus (HPV) in relation to cervicalneoplasiaSze, S. M., Candy., 施少妹. January 2006 (has links)
published_or_final_version / Medical Sciences / Master / Master of Medical Sciences
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Clinical and pathological significance of HPV infection and p53 mutation in human esophageal cancer何丹, He, Dan. January 1997 (has links)
published_or_final_version / Anatomy / Doctoral / Doctor of Philosophy
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Analysis of co-infection of human immunodeficiency virus with human papillomavirus.Maregere, Bothwell. 17 May 2014 (has links)
We formulate a deterministic mathematical model for the co-infection of HPV with HIV without
treatment. Mathematical techniques were used to analyze the stability of the models in terms of basic
reproduction numbers for disease-free equilibrium point and fixed point theory used for analysis of the
endemic equilibrium point. The model incorporating HIV and HPV co-infection sought to investigate
the impact of HIV infection in the natural history of HPV infection, and the impact of HPV infection
in the natural history of HIV infection, over a period of time. Numerical simulations were carried out
to illustrate the trends of progression of HIV and HPV in the case of co-infection. The results from
our study showed that when both HIV and HPV infected individuals are active in the system then
co-infection grows faster compared to one infection which is active in the system. Our study also
showed that when we started with HPV infection in the community and introduces HIV infection
after sometime has more impact in the growth of co-infection population compared to start with HIV
infection and introduces HPV infection after sometime in the community. / Thesis (M.Sc.)-University of KwaZulu-Natal, Pietermaritzburg, 2014.
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Human Papillomavirus in human breast cancer and cellular immortalisationKan, Chin Yi, Biotechnology & Biomolecular Sciences, Faculty of Science, UNSW January 2007 (has links)
Human Papillomavirus (HPV) is a small, double stranded DNA tumour virus. Infection with HPV normally results in formation of warts. Certain types of HPV, such as type -16 and -18, are shown to have a causal role in the development of uterine cervical cancer, and are so called high risk type HPV. Recently, a role of HPV in breast cancer has been suggested, although a causal role for HPVs in human breast cancer is yet to be demonstrated. The first part of this study investigates the association of HPV with human breast cancer. The results demonstrate that 48% of breast cancers that occurred in Australian women are HPV positive and they are mainly variants of HPV-18. Further analysis shows that HPV positive breast cancer patients are significantly younger than HPV negative patients, suggesting infection with HPV increases the risk of breast cancer development. This is coincidental with increased risk of HPV infection in sexually active young women and provides evidence that HPV has a role in breast cancer development. The second part of this project investigates the mechanisms by which high risk type HPV oncogenic protein E6, transforms primary human foreskin keratinocytes (natural host cells of HPV). HPV E6 is always expressed in HPV positive cervical carcinoma and results in the degradation of the cellular tumour suppressor protein p53. It is generally believed that HPV E6 contributes to HPV transformation by degradation of p53 protein which leads to cellular immortalisation ? an early step in tumorigenic transformation. Subsequent studies, however, indicate that HPV E6 possesses other functions (such as induction of telomerase activity) which may also be involved in cellular immortalisation. The results of my investigations demonstrate: 1) that degradation of p53 protein is required but is insufficient to immortalise primary cells; 2) that HPV E6 induced telomerase activity is coincidental with an increase in cell culture passage number; 3) that multiple functions of high risk type HPV E6 protein are required for cellular immortalisation. This finding suggests HPV infection is associated with early onset of breast cancer and that multiple functions of high risk type HPV E6 protein are involved in cellular immortalisation. Further study in both of these areas should provide alternative diagnostic markers, leading to prevention and treatment strategies for HPV positive breast cancer and other cancers.
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Therapeutic reactivation of the p53 tumor suppressor protein in HPV-positive cervical cancer cells by the creosote bush lignan 3'-O-methyl-nordihydroguaiaretic acidAllen, Kristi Lynne. January 2007 (has links)
Thesis (Ph.D.)--Kent State University, 2007. / Title from PDF t.p. (viewed Mar. 11, 2009). Advisor: Angelo L. DeLucia. Keywords: human papillomavirus, E6 oncogene, lignan, p53, apoptosis. Includes bibliographical references (p. 132-144).
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Using the health belief model to enhance understanding of female college students' intention to receive the human papillomavirus vaccinationRidgeway, Kristi, January 2009 (has links) (PDF)
Thesis (M.Nurs.)--Washington State University, August 2009. / Title from PDF title page (viewed on July 21, 2009). "College of Nursing." Includes bibliographical references (p. 40-44).
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Human papillomavirus and oral cancerLiu, Xuan. January 1998 (has links)
Thesis (Ph. D.)--University of California, Los Angeles, 1998. / eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.
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HPV vaccination and adolescent femalesEadens, Melanie. January 2009 (has links)
Thesis (M.A.)--Northern Kentucky University, 2009. / Made available through ProQuest. Publication number: AAT 1467416. ProQuest document ID: 1851395831. Includes bibliographical references (p. 29-32)
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