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Explanatory models of rheumatoid arthritisSamford Fair, Betty Susan, Kahn, David L. January 2002 (has links)
Thesis (Ph. D.)--University of Texas at Austin, 2002. / Supervisor: David L. Kahn. Vita. Includes bibliographical references. Also available from UMI.
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A dosimetric study of dysprosium-165-macroaggregates used in treating rheumatoid arthritisHannelin, Martti. January 1988 (has links)
Thesis--Åbo Akademi.
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The latex fixation test; a diagnostic test for rheumatoid arthritis (with a review of the results of 100 positive cases).Urist, Arnold A. January 1961 (has links)
Inaug.-Diss.--Basel. / At head of title: Aus der Medizinischen Universitäts-Poliklinik Basel. Includes bibliographical references.
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The latex fixation test; a diagnostic test for rheumatoid arthritis (with a review of the results of 100 positive cases).Urist, Arnold A. January 1961 (has links)
Inaug.-Diss.--Basel. / At head of title: Aus der Medizinischen Universitäts-Poliklinik Basel. Includes bibliographical references.
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A combination method of treatment of rheumatoid arthritis by means of physical therapyMashburn, Mary Ellen. January 1942 (has links)
Thesis (M.S.)--University of Wisconsin--Madison, 1942. / Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaf 74).
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Parent families of rheumatoid arthritis patients.Phillips, George. E. January 1953 (has links)
No description available.
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Chronic autoimmune arthritis in rats pathogenesis and genetic factors /Vingsbo Lundberg, Carina. January 1997 (has links)
Thesis (doctoral)--Lund University, 1997. / Added t.p. with thesis statement inserted. Includes bibliographical references.
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Chronic autoimmune arthritis in rats pathogenesis and genetic factors /Vingsbo Lundberg, Carina. January 1997 (has links)
Thesis (doctoral)--Lund University, 1997. / Added t.p. with thesis statement inserted. Includes bibliographical references.
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Effective use of TNF antagonistsYocum, David January 2004 (has links)
Tumor necrosis factor (TNF) antagonists are biologic response modifiers that have significantly improved functional outcomes in patients with rheumatoid arthritis (RA). RA is a progressive disease in which structural joint damage can continue to develop even in the face of symptomatic relief. Before the introduction of biologic agents, the management of RA involved the use of disease-modifying antirheumatic drugs (DMARDs) early in the course of disease. This focus on early treatment, combined with the availability of the anti-TNF agents, has contributed to a shift in treatment paradigms favoring the early and timely use of DMARDs with biologic therapies. Improvement in symptom control does not always equate to a reduction in disease progression or disability. With the emergence of structure-related outcome measures as the primary means for assessing the effectiveness of antirheumatic agents, the regular use of X-rays is recommended for the continued monitoring and evaluation of patients. In addition to the control of symptoms and improvement in physical function, a reduction in erosions and joint-space narrowing should be considered among the goals of therapy, leading to a better quality of life. Adherence to therapy is an important element in optimizing outcomes. Durability of therapy with anti-TNF agents as reported from clinical trials can also be achieved in the clinical setting. Concomitant methotrexate therapy might be important in maintaining TNF antagonist therapy in the long term. Overall, the TNF antagonists have led to improvements in clinical and radiographic outcomes in patients with RA, especially those who have failed to show a complete response to methotrexate.
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Characteristics of uveitis in juvenile idiopathic arthritis patients in a screening program in Hong Kong劉韋形, Lau, Wai-ying, Winnie. January 2008 (has links)
published_or_final_version / Community Medicine / Master / Master of Public Health
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