Based on existing theories of personality and socio-emotional functioning (e.g. Clark, 2005; Lynch, Hempel & Clark, in press) a new model is proposed and tested. The model hypothesises that (i) temperament (reward and threat sensitivity) and childhood invalidation predict problems with interpersonal functioning, (ii) this effect is mediated by self-regulatory capacity; where self-regulatory capacity comprises self-control (ranging from emotional over-control to emotional under-control) and flexible control and (iii) self-regulatory capacity itself has a quadratic relationship with interpersonal functioning. A UK community sample (n= 512) completed a self-report survey, measuring each of the aforementioned latent variables. Structural equation modelling (SEM) was used to determine the goodness-of-fit of this and variations of this model. SEM identified that a non-mediation model provided the best fit (χ²=49.403, p< 0.001; CFI=0.98; RMSEA=0.056). Good-fit was obtained for a model including flexible control as a partial mediator (χ²=269.06, p< 0.001; CFI=0.956; RMSEA=0.081) and adequate-fit for a model including over-control as a partial mediator (χ²= 91.744, p < 0.001, CFI=0.932; RMSEA= 0.096). Correlation analyses suggested that over-control and under-control correlated positively with interpersonal problems. Results from SEM provided promising initial evidence for the mediating role of self-regulatory capacity, particularly for the flexible control component. Correlation analyses provided support for the non-linear relationship between self-regulatory capacity and interpersonal functioning, whereby extreme over-control or extreme under-control is associated with interpersonal problems. Findings have implications for identifying mechanisms of change for therapeutic approaches to emotion dysregulation and for understanding the over-controlled population, which has previously been overlooked.

The reasons why people may periodically resort to binge eating behavior have long been a focus of study, and the reasons are elusive and varied. For people troubled by poor sleep and living with chronic stress, binge eating may be an attempt by the brain's glucose-depleted executive processing center to both regulate (i.e., increase) glucose levels and induce restorative sleep. Recovery resulting from restorative sleep may lead to a reduction in perceived stress, improved mood, and increased willpower, reducing the likelihood of another binge episode in close temporal proximity to the sleep-induced recovery. A repetitive cycle may ensue when stress inevitably again disturbs sleep, lowering mood, reducing willpower, and heightening sensitivity to stigma and stress. The purpose of the research described here is to synthesize recent findings from three diverse fields of scientific inquiry to predict factors that influence episodes of binge eating. Combining studies of sleep and sleep disorders, stress and stigma research, and recent work on self-regulatory capacity, I attempt to show how poor sleep ultimately leads to binge eating. A seven-day study consisted of three parts: an initial set of baseline questionnaire and physiological measures; collection of objective sleep quality data using an electronic motion logger; and an online daily diary in which participants completed measures of self-regulatory capacity and reported details about their sleep, stress levels, experiences with stigma, mood, and eating events. The data partially supported a path model where sleep quality, stress, mood, and self-regulation affected binge eating behavior.

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