The goals of present study were: (a) to examine associations between adverse childhood experiences (ACEs) and telomere length during childhood using ACE composite scores both with and without “new” adversities (i.e., parental death and poverty), and (b) to determine if ACEs indirectly affect telomere length through children’s self-regulatory abilities (i.e., effortful control and self-control). The analytic sample consisted of national data from teachers, biological parents, and their children (N = 2,527; Mage = 9.35, SD = .36 years; 52% male; 45% Black). Results from linear regression analyses revealed a statistically significant main effect of updated (but not traditional) ACEs on child telomere length, controlling for hypothesized covariates, although the additional amount of variance explained by ACEs was negligible. Results from mediation analyses revealed an indirect effect of ACEs on child telomere length through self-control, assessed via a teacher-reported Social Skills Rating System, but not effortful control. While longitudinal studies are needed to strengthen claims of causation, the present study clarifies the association between ACEs and telomere length during middle childhood, and identifies a pathway from ACEs to changes in telomere length that should be explored further.

Background: For decades, researchers have been striving to better understand the complex mix of factors underlying post-combat mental illness, in the hope of enabling better outcomes for military personnel. This study focuses on investigating the behavior and possible interaction of two risk factors for poor mental health post-deployment to a combat mission: adverse childhood experience (ACE) and deployment-related stressful experience (DRSE). Methods: A longitudinal dataset linking data from 3302 military personnel at recruitment to data collected post-deployment to Canada’s mission in Afghanistan was analysed, using novel scoring systems for exposure classification. Results: Significant ACE-DRSE interaction terms were found in relation to SF-36 MCS, depression and suicidal ideation: the negative effect DRSE had on mental health outcomes was amplified as ACE levels increased. Conclusion: Individuals with a history of childhood adversity are more susceptible to the negative mental health impacts of stressful experiences during deployment to a combat zone.

Social health is an important predictor of overall health. Yet, it is an often neglected area of research. Strikingly, social connectedness is associated with a 50% reduction in risk of early death. While a plethora of research evidence supports the beneficial impact of nature exposure on physical and mental health, literature regarding the beneficial impact of nature exposure on social health is scant. In fact, no research to date has investigated the causal influence of nature exposure on social motivation, a construct comprised here of three measures (State Motivation to Foster Social Connections, State Positive Affect, and State Anxiety). The purpose of this study was twofold: 1) to examine the effects of virtual nature exposure on state social motivation, and 2) to investigate adverse childhood experiences as a moderator of those effects. In this online study, adult participants (N = 444) aged 18 to 58 were randomly assigned to one of the three experimental video conditions (wilderness nature exposure, urban non-nature exposure, indoor non-nature exposure). After watching a 15-minute video, participants completed measures related to state social motivation. Results revealed a significant main effect of nature exposure on state social motivation. However, the effects of nature exposure on state social motivation were not significantly moderated by adverse childhood experiences. Results suggest that nature exposure may have a positive impact on the development and maintenance of social connections and should be explored further as a social health intervention aimed at improving overall health.

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Social health is an important predictor of overall health. Yet, it is an often neglected area of research. Strikingly, social connectedness is associated with a 50% reduction in risk of early death. While a plethora of research evidence supports the beneficial impact of nature exposure on physical and mental health, literature regarding the beneficial impact of nature exposure on social health is scant. In fact, no research to date has investigated the causal influence of nature exposure on social motivation, a construct comprised here of three measures (State Motivation to Foster Social Connections, State Positive Affect, and State Anxiety). The purpose of this study was twofold: 1) to examine the effects of virtual nature exposure on state social motivation, and 2) to investigate adverse childhood experiences as a moderator of those effects. In this online study, adult participants (N = 444) aged 18 to 58 were randomly assigned to one of the three experimental video conditions (wilderness nature exposure, urban non-nature exposure, indoor non-nature exposure). After watching a 15-minute video, participants completed measures related to state social motivation. Results revealed a significant main effect of nature exposure on state social motivation. However, the effects of nature exposure on state social motivation were not significantly moderated by adverse childhood experiences. Results suggest that nature exposure may have a positive impact on the development and maintenance of social connections and should be explored further as a social health intervention aimed at improving overall health.

An abundance of research has implicated socioeconomic disadvantage as a major risk factor for increased rates of morbidity and mortality worldwide. While advances in science, medicine, and technology have enabled a positive trend in health outcomes over the previous two decades, individuals of lower socioeconomic status have experienced negligible improvements in health and longevity. Furthermore, individuals of lower socioeconomic status face higher risks of mental health disorders than their higher socioeconomic status counterparts. In order to improve methods of intervention, it is important to understand how the roots of these issues are cultivated during childhood. Socioeconomic status is operationalized in multiple ways, including objective measures at both the household and neighborhood levels such as income, education, occupation, employment status, and single-parent status, as well as subjective measures such as perceived social status. This work explores the relationships between exposure to childhood socioeconomic disadvantage and the development of psychopathology. It reviews the literature for impacts of lower socioeconomic status during childhood on both internalizing (mood and anxiety) and externalizing (behavioral and substance use) disorders. Overall, mental health disorders constitute a significant proportion of the worldwide health burden, affecting one in four adults across the global population, including one in five adults and one in two adolescents in the United States. These staggering prevalence rates illuminate the importance of better understanding the mechanisms by which mental health illnesses emerge. Childhood exposure to socioeconomic deprivation has been identified as a robust contributing factor to the increased risk of psychopathology development. Exposure to socioeconomic disadvantage increases the risk for mood disorders such as major depressive disorder, dysthymic disorder, bipolar I and II disorder, cyclothymic disorder; anxiety disorders such as generalized anxiety disorder, panic disorder, social anxiety disorder, and specific phobias; behavioral disorders such as oppositional defiant disorder, conduct disorder, antisocial personality disorder, and attention-deficit/hyperactivity disorder; and substance use disorders such as alcohol and tobacco use. These conditions can disrupt normal growth and development; cause significant impairment in social, academic, and occupational environments; and create tremendous distress in important areas of daily functioning. An ecobiodevelopmental model reveals how the interplay of biological factors, such as genetic inheritance and physiological adaptations/disruptions, with ecological factors, such as the social and physical environment, occurs continuously across the entire life span from the prenatal period through infancy, childhood, adolescence, and adulthood to drive development and the evolution of individual health and disease. Application of this approach helps to explain how genetic predispositions interact with exposure to poverty to cultivate an environment more prone to adverse childhood experiences. Adverse childhood experiences are stressors occurring prior to the age of 18 that can be threatening or harmful emotionally or physically, which can include traumatic or potentially traumatic experiences such as neglect and abuse. Examples include socioeconomic hardship, racial/ethnic discrimination, parental death, separation from parents, divorce, neighborhood violence, parental mental illness, abuse, neglect, parental substance abuse, violence and criminality in the home, and life-threatening physical illness. Adverse childhood experiences promote toxic stress, which occurs from distressing situations high in magnitude, duration, or frequency without protective, buffering adult relationships to help the child cope. Toxic stress is characterized by an overloading of the body’s normal physiologic response mechanisms, which can have adverse long-term consequences through brain circuitry alterations and physiologic disruptions of the hypothalamic-pituitary-adrenocortical axis occurring during sensitive, critical periods of development. Toxic stress-induced alterations can occur in brain regions such as the hippocampus, amygdala, and prefrontal cortex, which are important in mood control, anxiety, stress coping, decision-making, and self-regulation. Self-regulation is a critical mediator in the link between childhood deprivation and subsequent psychopathology, as deficits in self-regulation increases the risk of both internalizing and externalizing disorders. In summation, the ecobiodevelopmental model is a multi-disciplinary approach that integrates developmental science constructs of toxic stress and self-regulation with ecology, neuroscience, and life course sciences to supply promising explanations for the underlying mechanisms linking childhood poverty to mental illnesses.

The therapeutic alliance is a core element of successful treatment in therapy. Recent literature has explored variables that predict the alliance at various time points during therapy, but few studies have explored how the alliance develops over time and the factors that influence its rate of change. The current study addresses these questions by estimating latent growth-curve models to analyze how male and female partners' alliance scores develop over time and how adverse childhood experiences (ACEs) impact the development of the alliance during the first six sessions of therapy. Results from these analyses show that neither men nor women's ACEs had a significant effect on the rate of change in the alliance. A discussion of the attachment implications of these findings, as well as the limitations of this study and potential directions for future research are then presented.

Introduction: A culturally competent survey currently does not exist to characterize the burden of Adverse Childhood Experiences (ACEs) among a HIV/AIDS population receiving care at a local Center of Excellence (COE). Methods: A qualitative study was conducted including 11 interviews involving opinions on national surveillance questions to develop a culturally competent survey. Purposive sampling, reactive probing, and analysis of transcribed interviews were completed using structured coding to determine which questions were kept, modified, or removed in the final survey. Results: The final 55-question survey contained more generalized ACE questions, topics pertaining to barriers to HIV care, and a list that patients could select from to indicate what they need to improve their HIV care. Conclusion: The final survey provided the opportunity to characterize the burden of ACEs at a COE. Future directions involve piloting the survey as a quality improvement tool with the goal of increasing retention rates through more individualized HIV care.

El texto completo de este trabajo no está disponible en el Repositorio Académico UPC por restricciones de la casa editorial donde ha sido publicado. / Objective: The objective of this study was to evaluate the association between (1) different types of ACEs and migraine, and (2) the number of ACEs and migraine among adolescent mothers in Lima, Peru. Methods: Our cross-sectional study included 787 adolescent mothers (14- to 18-years of age) in Peru. In-person interviews were conducted postpartum, in hospital, within 2-days of delivery. Nine types of ACEs were assessed, including exposure to three categories of abuse, two categories of neglect, and four categories of household dysfunction. Multivariable logistic regression procedures were used to estimate odds ratios (ORs) and 95% confidence intervals (95% CI) for the association between ACEs and migraine while adjusting for putative confounders. Results: Approximately 75% of adolescent mothers reported having experienced at least one type of ACE. Adolescent mothers who reported any childhood abuse had 1.49-fold increased odds of migraine (aOR = 1.49; 95% CI 1.03–2.18) compared to those with no history of childhood abuse. Adolescent mothers who reported experiencing household dysfunction had 1.56-fold increase odds of migraine (aOR = 1.56; 95% CI 1.09–2.24). Compared to participants who reported no ACE, those who experienced four or more ACEs had 3.09-fold (aOR = 3.09; 95% CI 1.80–5.40) increased odds of migraine (ptrend < 0.001). Conclusion: Exposure to ACEs is highly prevalent in adolescent-aged mothers postpartum and is associated with increased odds of migraine. These findings support the importance of screening for ACEs and migraine among adolescent mothers; and the need for providing culturally appropriate, trauma-informed headache care. / National Institutes of Health / Revisión por pares

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