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Nitric oxide modulates the effect of RHIGF-I on the accumulation of collagen and kidney growthAli, Abdulgader Saleh January 1997 (has links)
No description available.
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The prevalence and nutritional causes of hypoglycaemia in patients with end-stage renal failure (ESRF) on maintenance haemodialysis (MHD) at Kenyatta National Hospital Nairobi, Kenya /Kariuki, Anastacia Wanjiku. January 2008 (has links)
Thesis (MNutr)--University of Stellenbosch, 2008. / Bibliography. Also available via the Internet.
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Hemodialysis patients and end-of-life medical treatment decisions : a theory of personal preservation /Calvin, Amy Olivier, January 2000 (has links)
Thesis (Ph. D.)--University of Texas at Austin, 2000. / Vita. Includes bibliographical references (leaves 261-290). Available also in a digital version from Dissertation Abstracts.
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Protein and carbohydrate metabolism in metabolic acidosisReaich, David January 1995 (has links)
Chronic renal failure (CRF) is associated with loss of lean body mass, a high incidence of malnutrition, and with insulin resistance. CRF is often complicated by metabolic acidosis. Metabolic acidosis is known to alter both protein and carbohydrate metabolism. A series of studies have been undertaken to investigate the effect of metabolic acidosis on protein metabolism in both normal and CRF human subjects, and to study whether metabolic acidosis in CRF affects insulin sensitivity. Protein turnover was studied using the technique of primed constant infusions of L-[1-<sup>13</sup>C]leucine. Normal subjects were studied before and after ammonium chloride induced metabolic acidosis. Acidosis was associated with increased protein turnover and amino acid oxidation. In CRF subjects, correction of acidosis with sodium bicarbonate decreased protein turnover and amino acid oxidation. The effect of acidosis in CRF on insulin mediated carbohydrate metabolism was studied using the technique of the hyperinsulinaemic euglycaemic clamp. Insulin sensitivity increased with correction of acidosis. By combining L-[1-<sup>13</sup>C]leucine infusions with hyperinsulinaemic euglycaemic clamps, the response of protein metabolism to hyperinsulinaemia was measured before and after correction of acidosis. The presence of acidosis did not impair the ability of insulin to modulate protein metabolism. There is therefore, dissociation between the effects of acidosis in CRF on insulin mediated carbohydrate metabolism and insulin mediated protein metabolism. In summary, metabolic acidosis increases protein catabolism in both normal and CRF man and may contribute to the loss of lean body mass characteristic of CRF. Insulin resistance in CRF improves with correction of acidosis. However the effects of acidosis on protein metabolism are not mediated via alterations in insulin sensitivity.
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Studies on carnitine and related metabolism in end stage renal diseaseBartel, Lavon L. January 1979 (has links)
Thesis--University of Wisconsin--Madison. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 146-168).
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The economic burden of end-stage renal disease in Canada: present and future /Zelmer, Jennifer. January 2005 (has links)
Thesis (Ph.D.)--McMaster University, 2005. / Includes bibliographical references (p. 155-185). Also available online.
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Treatment preferences and regimen compliance of hemodialysis patients : assessment and correlates /Goldman, Daryl January 1984 (has links)
No description available.
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The biology of kidney malformationsWinyard, Paul Julian Douglas January 1998 (has links)
No description available.
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A comprehensive cellular and transcriptomic analysis of end-stage renal failure and transplantationJolly, Elaine Christina January 2014 (has links)
No description available.
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Mexican-American women's perspectives on end-stage renal disease and the hemodialysis regimen : pychosocial influences on compliance with treatment recommendations /Tijerina, Mary Sylvia, January 2000 (has links)
Thesis (Ph. D.)--University of Texas at Austin, 2000. / Vita. Includes bibliographical references (leaves 242-253). Available also in a digital version from Dissertation Abstracts.
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