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The Global ETD Search service is a free service for researchers
to find electronic theses and dissertations. This service is
provided by the
Networked Digital Library of Theses and Dissertations.
Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
Search results
Showing 1 to 10 of 403 (0.025 seconds)| 1 |
Flüssigchromatographische Bestimmung aliphatischer und aromatischer Amine mit 4-Chloro-7-nitrobenzo-2-oxa1,3-diazolJachmann, Nicole. January 2001 (has links)
Münster (Westfalen), Universiẗat, Diss., 2002. / Dateien im PDF-Format.
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Beef liver mitochondrial amine oxidase: purification and studies on some physical and chemical propertiesGomes, Benedict January 1968 (has links)
Typescript. / Thesis (Ph. D.)--University of Hawaii, 1968. / Bibliography: leaves [153]-162. / xiii, 162 l illus., tables
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Mechanism-based Inhibitors for Copper Amine Oxidases: Synthesis, Mechanism, and EnzymologyZhong, Bo January 2010 (has links)
No description available.
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Syntheses and mechanistic studies of some octahedral ruthenium (III) amine complexesIsabirye, David Awubwa. January 1977 (has links)
published_or_final_version / Chemistry / Doctoral / Doctor of Philosophy
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Synthetic studies of N-diphenylphosphinyl aziridinesCantrill, Alexander A. January 1996 (has links)
No description available.
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Metalloporphyrin-catalysed model systems for the cytochrome P450-dependent mono-oxygenasesMortimer, D. N. January 1986 (has links)
No description available.
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Diastereoselective additions to iminium ions in the synthesis of enantiopure α amino acids : stereocontrol using the (S) 5 phenylmorpholin 2 one templateVickers, Richard John January 2000 (has links)
No description available.
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Novel approaches to polycyclic heterocyclesBillington, Helen January 2002 (has links)
No description available.
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Accelerated Cytotoxicity Mechanism Screening of 4-Aminobiphenyl in an in vitro Hepatocyte Inflammation ModelDelaney, Sarah 23 August 2011 (has links)
4-Aminobiphenyl is an aromatic amine compound that is present in cigarette smoke, diesel exhaust, cooking oil fumes and dye intermediates. It is a well-known human bladder carcinogen and liver carcinogen in experimental animals that is metabolically activated by liver CYP1A1/2. We have used the “Accelerated Cytotoxicity Mechanism Screening” (ACMS) techniques to analyze the molecular cytotoxic mechanisms of 4-aminobiphenyl. Hepatocyte exposure to an inflammatory system significantly increased hepatocyte susceptibility to 4-aminobiphenyl. 4-Aminobiphenyl- induced cytotoxicity and lipid peroxidation were both prevented by altering cellular redox status and with the addition of antioxidants. Toxicity was increased with the depletion of hepatocyte GSH levels and by inhibiting N-acetyltransferase. These results will provide more insight into the cytotoxic and genotoxic mechanisms of 4-aminobiphenyl and also suggest that inflammation may be responsible for an increase in arylamine carcinogenesis.
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Accelerated Cytotoxicity Mechanism Screening of 4-Aminobiphenyl in an in vitro Hepatocyte Inflammation ModelDelaney, Sarah 23 August 2011 (has links)
4-Aminobiphenyl is an aromatic amine compound that is present in cigarette smoke, diesel exhaust, cooking oil fumes and dye intermediates. It is a well-known human bladder carcinogen and liver carcinogen in experimental animals that is metabolically activated by liver CYP1A1/2. We have used the “Accelerated Cytotoxicity Mechanism Screening” (ACMS) techniques to analyze the molecular cytotoxic mechanisms of 4-aminobiphenyl. Hepatocyte exposure to an inflammatory system significantly increased hepatocyte susceptibility to 4-aminobiphenyl. 4-Aminobiphenyl- induced cytotoxicity and lipid peroxidation were both prevented by altering cellular redox status and with the addition of antioxidants. Toxicity was increased with the depletion of hepatocyte GSH levels and by inhibiting N-acetyltransferase. These results will provide more insight into the cytotoxic and genotoxic mechanisms of 4-aminobiphenyl and also suggest that inflammation may be responsible for an increase in arylamine carcinogenesis.
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